My shins hurt after running. Could it be shin splints?

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If you’ve started running for the first time, started again after a break, or your workout is more intense, you might have felt it. A dull, nagging ache down your shins after you exercise.

Should you push through? Or could it be the sign of something more serious?

Shin splints are one of the most common and preventable injuries among runners, whether new or seasoned.

The good news is they can usually be treated effectively in a few weeks. But it’s important to recognise when to take a break. Knowing the simple ways to treat and prevent shin splints can prevent a more serious injury, and get you back on track faster.

What are shin splints?

Shin splints, medically known as medial tibial stress syndrome, are a common overuse injury.

They cause pain along the inner border of the tibia (shinbone), usually triggered by repetitive stress on the lower leg. Your leg may also feel tender or swell.

Shin splints are a type of periostitis, which means inflammation of the tissue lining the bone. The pain often fades with rest but quickly returns once activity resumes.

This kind of injury is especially common in sports such as football, rugby, and track and field, affecting between 4% and 35% of athletes, and up to 20% of runners. It can also affect dancers and military recruits.

What puts you at risk?

Shin splints can appear soon after sudden changes to your physical activity or exercise routine.

For example, you may have started exercising more often or for longer, or more intensely (such as running uphill or for longer distances).

A variety of factors can add fuel to the fire. They generally fall into two types:

  • activity-related (what you do with your body)
  • biomechanical (how your body moves or is built).

Aside from sudden spikes in training, activity-related risks include playing sport or running on hard surfaces or exercising in poorly designed shoes. For example, studies of soldiers have shown exercising in unsuitable or worn-out boots increases their risk of overuse injuries in the lower legs, including shin splints.

Diet may make a difference, too. There is evidence not eating enough calcium can make you more susceptible to shin splints. A vitamin D deficiency may also contribute, since it’s vital for calcium absorption.

Biomechanical risks can include a higher body mass index (BMI), having one leg longer than the other, tight calf muscles or flat feet (low or unusually inflexible arches).

If your feet roll in too much when you walk or run – often called flat feet or fallen arches – you’re also more susceptible.

While some studies suggest female athletes may experience shin splints more often than males, we need more research to fully understand why.

In short, shin splints aren’t just a bone issue. They reflect a complex mismatch between how much or hard you train and how your body tries (and sometimes fails) to adapt.

How can I tell if it’s something worse?

Shin splints are typically less severe than a stress fracture. This is a small crack in the bone caused by repeated impact or overuse, and usually requires a longer recovery period.

A stress fracture often causes sharp, localised pain that worsens with activity and may even hurt at rest or when touched.

A simple test can help you decide whether to seek additional advice: if you are unable to hop on one leg about ten times without sharp pain, it’s time to talk to a physio, sports doctor or podiatrist.

They can assess your symptoms and suggest treatment options. Imaging such as an x-ray or MRI may be used to rule out more serious conditions.

Treatment: rest, rehab, and return

The first and most important treatment is rest. Usually, shin splints resolve over three to four weeks. Continuing to train during the healing process will only prolong recovery and increase the risk of more serious injury.

Other effective strategies include:

You’ll want to be pain-free for at least three weeks before gradually resuming your exercise routine.

When returning, go slow and build up the amount and intensity of exercise gradually.

Prevention is the best treatment

Preventing shin splints is all about balance and preparation. Here are some evidence-based tips:

https://www.youtube.com/embed/EMiRjzMs7Zw?wmode=transparent&start=0 Strengthening your lower leg muscles can prevent further injury.

Krissy Kendall, Senior Lecturer in Exercise and Sports Science, Edith Cowan University and Caitlin Fox-Harding, Senior Lecturer/Researcher in Exercise and Sports Science, Edith Cowan University

This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • What is frozen shoulder? And will I need surgery?

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    Frozen shoulder can make simple tasks – such as lifting your arm, sleeping on your side, getting out of bed, putting on a bra, driving or playing with your kids – painful and challenging.

    This condition usually starts with pain suddenly developing in the shoulder and stiffness. Over time, the pain and stiffness get worse. It can drag on for months or even years.

    So, what causes frozen shoulder? And can it be treated?

    Mikolette/Getty

    What is frozen shoulder?

    This shoulder condition, also known as “adhesive capsulitis”, affects around 8% of men and 10% of women aged 25–64. But it’s more common over 40, especially for people in their 60s.

    We don’t fully understand what causes frozen shoulder.

    The tissues around the joint become tight, swollen and stiff. But we don’t know exactly why these changes occur and lead to pain and limited movement.

    There are usually three stages:

    • freezing – pain gradually gets worse and the shoulder becomes stiff, limiting the range of movement
    • frozen – stiffness and pain usually peak, but may begin to ease
    • thawing – pain and stiffness slowly improve, and movement begins to return.

    While health professionals commonly accept it, this staged description suggests frozen shoulder will follow a predictable pattern and always get better on its own. But research suggests this is not always the case.

    For example, the “freezing” stage is usually expected to last at least ten weeks. But some people will start to notice improved movement sooner.

    Recovery stages will vary from person to person and can take months to years. Some people may not fully recover, even with treatment.

    One 2020 study followed up with 215 patients with frozen shoulder. While over 70% of participants said they were happy with improvements in their symptoms, around 40% still had some movement restriction two years after their symptoms began.

    Another study from 2008 found over a third of people they surveyed (41%) had ongoing symptoms two to seven years later, including pain and difficulty sleeping.

    Who is most at risk?

    Certain groups are more likely to develop frozen shoulder:

    There is some evidence genetics also plays a role, as a family history increases your risk.

    But we need more high-quality research to understand what’s behind these risk factors.

    For example, people with diabetes are around five times more likely to develop frozen shoulder than those without diabetes – and also have worse pain. This may be linked to diabetes-related changes in the body, such as reduced blood flow to tissues and chemical changes from high blood sugar. But the exact mechanisms are unclear, and research is yet to determine whether controlling blood sugar better could help prevent or slow frozen shoulder.

    Similarly, women are 40% more likely to develop frozen shoulder than men, with one theory suggesting hormone fluctuations during menopause are responsible. But there is no clear evidence yet to support this.

    How is frozen shoulder treated?

    There is mixed evidence about which treatments are effective, including whether over-the-counter pain medication such as Voltaren helps.

    Oral steroids

    A review of the evidence suggests oral steroids, such as prednisolone, can provide some short-term pain relief and improve shoulder movement, compared to doing nothing or a placebo. But these benefits don’t seem to last beyond six weeks, and the evidence comes from a few small studies. These require a prescription.

    Injections

    High-quality evidence shows corticosteroid injections can provide short-term relief, compared to doing nothing.

    There is also some limited evidence that corticosteroid injections and platelet rich plasma injections can provide better short-term pain relief, compared with over-the-counter pain relief and physiotherapy. However, the studies are small or poorly designed and the effects are small, so the evidence needs to be interpreted with caution.

    Physiotherapy

    Moderate-quality evidence suggests physiotherapy can help improve shoulder movement. Benefits of physio are greater when combined with a steroid injection, and followed up by doing the exercises at home. More research is needed to understand how well these treatments work in the long term.

    What about surgery?

    There are two main procedures for frozen shoulder, both done while the patient is unconscious under anaesthetic.

    1. Manipulation under anaesthetic

    This is a less invasive procedure where the surgeon stretches the shoulder, without cutting into the joint, to help loosen tight tissue that may be causing stiffness.

    2. Arthroscopic capsular release

    In this type of keyhole surgery, the surgeon cuts tight tissues inside the shoulder joint to try to free up shoulder movement.

    Improvements from these procedures are typically small, and evidence suggests the results are not better than non-surgical treatments. For example, one study showed that after one year, patients who’d had surgery had similar improvements to those who’d had physiotherapy and a steroid injection, but no surgery.

    These procedures also have several downsides. It’s more expensive than other treatments, carries additional risks, and typically requires weeks (and up to three months) of rehabilitation.

    The bottom line

    Being physically active and doing exercises can help if you’re experiencing pain and limited movement. But you don’t have to work this out alone. It’s a good idea to get advice on managing pain and how to stay active.

    If you suspect you have frozen shoulder, it’s important to see a doctor or physiotherapist so they can rule out other conditions, such as fracture and arthritis.

    A health professional can also discuss management – the potential benefits, harms, costs, and how easy it is to access each treatment option.

    Fernando Sousa, Research Fellow in Physiotherapy, Monash University; Joshua Zadro, NHMRC Emerging Leader Research Fellow, Sydney Musculoskeletal Health, University of Sydney, and Peter Malliaras, Professor in Physiotherapy, Monash University

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • Bromelain vs Inflammation & Much More

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    Let’s Get Fruity

    Bromelain is an enzyme* found in pineapple (and only in pineapple), that has many very healthful properties, some of them unique to bromelain.

    *actually a combination of enzymes, but most often referred to collectively in the singular. But when you do see it referred to as “they”, that’s what that means.

    What does it do?

    It does a lot of things, for starters:

    ❝Various in vivo and in vitro studies have shown that they are anti-edematous, anti-inflammatory, anti-cancerous, anti-thrombotic, fibrinolytic, and facilitate the death of apoptotic cells. The pharmacological properties of bromelain are, in part, related to its arachidonate cascade modulation, inhibition of platelet aggregation, such as interference with malignant cell growth; anti-inflammatory action; fibrinolytic activity; skin debridement properties, and reduction of the severe effects of SARS-Cov-2

    ~ Dr. Carolina Varilla et al.

    Some quick notes:

    • “facilitate the death of apoptotic cells” may sound alarming, but it’s actually good; those cells need to be killed quickly; see for example: Fisetin: The Anti-Aging Assassin
    • If you’re wondering what arachidonate cascade modulation means, that’s the modulation of the cascade reaction of arachidonic acid, which plays a part in providing energy for body functions, and has a role in cell structure formation, and is the precursor of assorted inflammatory mediators and cell-signalling chemicals.
    • Its skin debridement properties (getting rid of dead skin) are most clearly seen when using bromelain topically (one can literally just make a pineapple poultice), but do occur from ingestion also (because of what it can do from the inside).
    • As for being anti-thrombotic and fibrinolytic, let’s touch on that before we get to the main item, its anti-inflammatory properties.

    If you want to read more of the above before moving on, though, here’s the full text:

    Bromelain, a Group of Pineapple Proteolytic Complex Enzymes (Ananas comosus) and Their Possible Therapeutic and Clinical Effects. A Summary

    Anti-thrombotic and fibrinolytic

    While it does have anti-thrombotic effects, largely by its fibrinolytic action (i.e., it dissolves the fibrin mesh holding clots together), it can have a paradoxically beneficial effect on wound healing, too:

    Stem Bromelain Proteolytic Machinery: Study of the Effects of its Components on Fibrin (ogen) and Blood Coagulation

    For more specifically on its wound-healing benefits:

    In Vitro Effect of Bromelain on the Regenerative Properties of Mesenchymal Stem Cells

    Anti-inflammatory

    Bromelain is perhaps most well-known for its anti-inflammatory powers, which are so diverse that it can be a challenge to pin them all down, as it has many mechanisms of action, and there’s a large heterogeneity of studies because it’s often studied in the context of specific diseases. But, for example:

    ❝Bromelain reduced IL-1β, IL-6 and TNF-α secretion when immune cells were already stimulated in an overproduction condition by proinflammatory cytokines, generating a modulation in the inflammatory response through prostaglandins reduction and activation of cascade reactions that trigger neutrophils and macrophages, in addition to accelerating the healing process

    ~ Dr. Taline Alves Nobre et al.

    Read in full:

    Bromelain as a natural anti-inflammatory drug: a systematic review

    Or if you want a more specific example, here’s how it stacks up against arthritis:

    ❝The results demonstrated the chondroprotective effects of bromelain on cartilage degradation and the downregulation of inflammatory cytokine (tumor necrosis factor (TNF)-α, IL-1β, IL-6, IL-8) expression in TNF-α–induced synovial fibroblasts by suppressing NF-κB and MAPK signaling❞

    ~ Dr. Perephan Pothacharoen et al.

    Read in full:

    Bromelain Extract Exerts Antiarthritic Effects via Chondroprotection and the Suppression of TNF-α–Induced NF-κB and MAPK Signaling

    More?

    Yes more! You’ll remember from the first paper we quoted today, that it has a long laundry list of benefits. However, there’s only so much we can cover in one edition, so that’s it for today

    Is it safe?

    It is generally recognized as safe. However, its blood-thinning effect means it should be avoided if you’re already on blood-thinners, have some sort of bleeding disorder, or are about to have a surgery.

    Additionally, if you have a pineapple allergy, this one may not be for you.

    Aside from that, anything can have drug interactions, so do check with your doctor/pharmacist to be sure (with the pharmacist usually being the more knowledgeable of the two, when it comes to drug interactions).

    Want to try some?

    You can just eat pineapples, but if you don’t enjoy that and/or wouldn’t want it every day, bromelain is available in supplement form too.

    We don’t sell it, but here for your convenience is an example product on Amazon

    Enjoy!

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  • Rewired – by Erica Spiegelman

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    The subtitle promises “a bold new approach to addiction and recovery”, so first we must ask: does it deliver?

    The answer is subjective and relative to one’s experience, but we would say: it’s bold to call the approach “new”, per se.

    However! Where this claim of newness may come from is that—notwithstanding the blurb’s claim that it can be used in conjunction with or in place of 12-step programs—in fact it is quite opposed to some of the 12 steps principles, insofar as it places much greater importance on personal agency, responsibility, and empowerment.

    So, for a reader whose understanding of addiction and recovery has been largely informed by the ideas championed by 12-step programs, this approach will certainly be new, and yes, bold.

    The goal of this book is help the reader to practise self-actualization, which as a standalone term may sound like a lot of woo, but what it means in plain words is “to have a clear idea of the kind of person one wants to be, and then become that person”.

    Indeed, while some principles this book espouses may be in line with 12-step programs (such as: complete honesty), others stand intentionally apart, such as solitude—making the argument that recovery can never be complete if we cannot be alone with ourselves and our abstinence (from whatever it may be for any given reader) would otherwise be dependent on the strength of those around us.

    But nor does the book preach any rejection of society either; attention is also given to integration and relationships with others, which is important too. In short, that we can stand alone whenever we need to, and/but that we still need not be isolated in general.

    The style is quite soft self-help, while nonetheless getting straight to the point and not getting tangled up in platitudes or such. It’s a clear and instructive book, that explains its ideas well as it goes.

    Bottom line: if you or a loved one are struggling with an addiction (or have done so recently enough that recovery is still a case of being not yet “out of the woods” entirely), then this book can help bring a lot of strength and sense of direction, ultimately making things easier and at the same time more likely to go well for you/them.

    Click here to check out Rewired, and rewire your way into a much better life!

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  • Age & Aging: What Can (And Can’t) We Do About It?

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    How old do you want to be?

    We asked you how old you are, and got an interesting spread of answers. This wasn’t too surprising; of course we have a general idea of who our readership is and we write accordingly.

    What’s interesting is the gap for “40s”.

    And, this wasn’t the case of a broken poll button, it’s something that crops up a lot in health-related sociological research. People who are most interested in taking extra care of their body are often:

    • Younger people full of optimism about maintaining this perfectly healthy body forever
    • Older people realizing “if I don’t want to suffer avoidable parts of age-related decline, now is the time to address these things”

    In between, we often have a gap whereby people no longer have the optimism of youth, but do not yet feel the pressure of older age.

    Which is not to say there aren’t 40-somethings who do care! Indeed, we know for a fact we have some subscribers in their 40s (and some in their 90s, too), just, they evidently didn’t vote in this poll.

    Anyway, let’s bust some myths…

    Aging is inevitable: True or False?

    False, probably. That seems like a bold (and fortune-telling) claim, so let’s flip it to deconstruct it more logically:

    Aging is, and always will be, unstoppable: True or False?

    That has to be “False, probably”. To say “true” now sounds like an even bolder claim. Just like “the moon will always be out of reach”.

    • When CPR was first developed, first-aiders were arrested for “interfering with a corpse”.
    • Many diseases used to be death sentences that are now “take one of these in the morning”
      • If you think this is an appeal to distant history, HIV+ status was a death sentence in the 90s. Now it’s “take one of these in the morning”.

    But, this is an appeal to the past, and that’s not always a guarantee of the future. Where does the science stand currently? How is the research and development doing on slowing, halting, reversing aging?

    We can slow aging: True or False?

    True! There’s a difference between chronological age (i.e., how much time has passed while we’ve been alive) and biological age (i.e., what our diverse markers of aging look like).

    Biological age often gets talked about as a simplified number, but it’s more complex than that, as we can age in different ways at different rates, for example:

    • Visual markers of aging (e.g. wrinkles, graying hair)
    • Performative markers of aging (e.g. mobility tests)
    • Internal functional markers of aging (e.g. tests for cognitive decline, eyesight, hearing, etc)
    • Cellular markers or aging (e.g. telomere length)
    • …and more, but we only have so much room here

    There are things we can do to slow most of those, including:

    In the case of cognitive decline particularly, check out our previous article:

    How To Reduce Your Alzheimer’s Risk

    It’s too early to worry about… / It’s too late to do anything about… True or False?

    False and False!

    Many things that affect our health later in life are based on early-life choices and events. So it’s important for young people to take advantage of that. The earlier one adopts a healthy lifestyle, the better, because, and hold onto your hats for the shocker here: aging is cumulative.

    However, that doesn’t mean that taking up healthy practices (or dropping unhealthy ones) is pointless later in life, even in one’s 70s and beyond!

    Read about this and more from the National Institute of Aging:

    What Do We Know About Healthy Aging?

    We can halt aging: True or False?

    False, for now at least. Our bodies are not statues; they are living organisms, constantly rebuilding themselves, constantly changing, every second of every day, for better or for worse. Every healthy or unhealthy choice you make, every beneficial or adverse experience you encounter, affects your body on a cellular level.

    Your body never, ever, stops changing for as long as you live.

    But…

    We can reverse aging: True or False?

    True! Contingently and with limitations, for now at least.

    Remember what we said about your body constantly rebuilding itself? That goes for making itself better as well as making itself worse.

    But those aren’t really being younger, we’ll still die when our time is up: True or False?

    False and True, respectively.

    Those kinds of things are really being younger, biologically. What else do you think being biologically younger is?

    We may indeed die when our time is up, but (unless we suffer fatal accident or incident first) “when our time is up” is something that is decided mostly by the above factors.

    Genetics—the closest thing we have to biological “fate”—accounts for only about 25% of our longevity-related health*.

    Genes predispose, but they don’t predetermine.

    *Read more: Human longevity: Genetics or Lifestyle? It takes two to tango

    (from the Journal of Immunity and Ageing)

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  • The Science of Self-Learning – by Peter Hollins

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    Teaching oneself new things is often the most difficult kind of bootstrapping, especially when one is unsure of such critical things as:

    • Where to begin? How, for that matter, do we find where to begin?
    • What can/should a learning journey look like?
    • What challenges should we expect, and how will we overcome them?

    Hollins answers all of these questions and more. The greatest value of this book is perhaps in its clear presentation of concrete step-by-step instructions. Hollins gives illustrated examples too, but most importantly, he gives models that can be applied to any given type of learning.

    The book also covers the most difficult problems most people face when trying to learn something by themselves, including:

    • Keeping oneself on-task (maintaining discipline)
    • Measuring progress (self-testing beyond memorization)
    • Keeping a fair pace of progress (avoiding plateaus)
    • How to know when one’s knowledge is sufficient or not (avoiding Dunning-Kruger Club)

    All in all, if you’re looking to learn a new subject or skill, this could be a first step that saves you a lot of time later!

    Get your copy of the Science of Self-Learning on Amazon today!

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  • Green Tea Allergies and Capsules

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    It’s Q&A Day at 10almonds!

    Have a question or a request? You can always hit “reply” to any of our emails, or use the feedback widget at the bottom!

    In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!

    As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!

    So, no question/request too big or small

    ❝Hey Sheila – As always, your articles are superb !! So, I have a topic that I’d love you guys to discuss: green tea. I used to try + drink it years ago but I always got an allergic reaction to it. So the question I’d like answered is: Will I still get the same allergic reaction if I take the capsules ? Also, because it’s caffeinated, will taking it interfere with iron pills, other vitamins + meds ? I read that the health benefits of the decaffeinated tea/capsules are not as great as the caffeinated. Any info would be greatly appreciated !! Thanks much !!❞

    Hi! I’m not Sheila, but I’ll answer this one in the first person as I’ve had a similar issue:

    I found long ago that taking any kind of tea (not herbal infusions, but true teas, e.g. green tea, black tea, red tea, etc) on an empty stomach made me want to throw up. The feeling would subside within about half an hour, but I learned it was far better to circumvent it by just not taking tea on an empty stomach.

    However! I take an l-theanine supplement when I wake up, to complement my morning coffee, and have never had a problem with that. In all likelihood, the issue is neither caffeine (or else it’d happen for coffee or other sources of caffeine) nor theanine (or it’d happen for theanine supplements), but rather, the tannins in tea.

    Of course, my physiology is not your physiology, and this “shouldn’t” be happening to either of us in the first place, so it’s not something there’s a lot of scientific literature about, and we just have to figure out what works for us.

    This last Monday I wrote (inspired in part by your query) about l-theanine supplementation, and how it doesn’t require caffeine to unlock its benefits after all, by the way. So that’s that part in order.

    I can’t speak for interactions with your other supplements or medications without knowing what they are, but I’m not aware of any known issue, beyond that l-theanine will tend to give a gentler curve to the expression of some neurotransmitters. So, if for example you’re talking anything that affects that (e.g. antidepressants, antipsychotics, ADHD meds, sleepy/wakefulness meds, etc) then checking with your doctor is best.

    Don’t Forget…

    Did you arrive here from our newsletter? Don’t forget to return to the email to continue learning!

    Learn to Age Gracefully

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