
The Medicinal Chef – by Dale Pinnock
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The philosophy here is very much like our own—to borrow from Hippocrates: “let food be thy medicine”. Obviously please do also let medicine be thy medicine if you need it, but the point is that food is a very good starting place for combatting a lot of disease.
To this end, instead of labelling the recipes with such things as “V”, “Ve”, “GF” and suchlike, it assumes we can tell those things from the ingredients lists, and instead labels things per what they are especially good for:
- S: skin
- J: joints & bones
- R: respiratory system
- I: immune system
- M: metabolic health
- N: nervous system and mental health
- H: heart and circulation
- D: digestive system
- U: reproductive & urinary systems
As for the recipes themselves… They’re a lot like the recipes we share here at 10almonds in their healthiness, skill level, and balance of easy-to-find ingredients with the occasional “order it online” items that punch above their weight. In fact, we’ll probably modify some of the recipes for sharing here.
Bottom line: if you’re looking for genuinely healthy recipes that are neither too basic nor too arcane, this book has about 80 of them.
Click here to check out The Medicinal Chef: Healthy Every Day, and be healthy every day!
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Fix Your Upper Back With These Three Steps
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When it comes to back pain, the lower back gets a lot of attention, but what about when it’s nearer the neck and shoulders?
Reaching for better health
In this short video, Liv describes and shows three exercises:
Exercise 1: Thoracic Pullover (Dumbbell Pullover)
Purpose: Improves overhead reach and shoulder mobility.
Equipment: light weight, yoga block, or foam roller.
Steps:- Lie on the floor with the foam roller/block beneath the upper back.
- Hold the weight in both hands, arms extended upward.
- Inhale deeply and reach the weight toward the ceiling.
- Exhale and arc your spine over the block, moving the weight backward.
- Keep core tension to maintain a neutral lower back position.
- Perform 10 repetitions.
Exercise 2: Rotational Mobility Stretch
Purpose: enhances torso rotation, core strength, and hip mobility.
Equipment: none (or a mat)
Steps:- Lie on your side with knees stacked at 90° and arms extended in front.
- Hold a weight in the top hand.
- Inhale and lift the top arm toward the ceiling, extending the shoulder blade.
- Exhale and twist your torso, allowing the arm to move toward the floor.
- Modify by extending the bottom leg for a deeper twist if needed.
- Perform 6 reps per side, switching legs and repeating on the other side.
Exercise 3: Doorway/Pole Side Stretch
Purpose: targets multiple areas for a deep, satisfying stretch.
Equipment: door frame, pole, or wall.
Steps:- Stand at arm’s length from the wall or frame.
- Cross the outer leg (furthest from the wall) behind the inner leg.
- Place the closest hand on the wall and reach the other arm overhead.
- Grip the wall or frame with the top hand, pressing away with the bottom hand.
- Lean into a banana-shaped curve and rotate your chest upward for a deeper stretch.
- Hold for 20–30 seconds per side and repeat 2–3 times.
For more on all of these, plus visual demonstrations, enjoy:
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People on Ozempic may have fewer heart attacks, strokes and addictions – but more nausea, vomiting and stomach pain
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Ozempic and Wegovy are increasingly available in Australia and worldwide to treat type 2 diabetes and obesity.
The dramatic effects of these drugs, known as GLP-1s, on weight loss have sparked huge public interest in this new treatment option.
However, the risks and benefits are still being actively studied.
In a new study in Nature Medicine, researchers from the United States reviewed health data from about 2.4 million people who have type 2 diabetes, including around 216,000 people who used a GLP-1 drug, between 2017 and 2023.
The researchers compared a range of health outcomes when GLP-1s were added to a person’s treatment plan, versus managing their diabetes in other ways, often using glucose-lowering medications.
Overall, they found people who used GLP-1s were less likely to experience 42 health conditions or adverse health events – but more likely to face 19 others.
myskin/Shutterstock What conditions were less common?
Cardiometabolic conditions
GLP-1 use was associated with fewer serious cardiovascular and coagulation disorders. This includes deep vein thrombosis, pulmonary embolism, stroke, cardiac arrest, heart failure and myocardial infarction.
Neurological and psychiatric conditions
GLP-1 use was associated with fewer reported substance use disorders or addictions, psychotic disorders and seizures.
Infectious conditions
GLP-1 use was associated with fewer bacterial infections and pneumonia.
What conditions were more common?
Gastrointestinal conditions
Consistent with prior studies, GLP-1 use was associated with gastrointestinal conditions such as nausea, vomiting, gastritis, diverticulitis and abdominal pain.
Other adverse effects
Increased risks were seen for conditions such as low blood pressure, syncope (fainting) and arthritis.
People who took Ozempic were more likely to experience stomach upsets than those who used other type 2 diabetes treatments. Douglas Cliff/Shutterstock How robust is this study?
The study used a large and reputable dataset from the US Department of Veterans Affairs. It’s an observational study, meaning the researchers tracked health outcomes over time without changing anyone’s treatment plan.
A strength of the study is it captures data from more than 2.4 million people across more than six years. This is much longer than what is typically feasible in an intervention study.
Observational studies like this are also thought to be more reflective of the “real world”, because participants aren’t asked to follow instructions to change their behaviour in unnatural or forced ways, as they are in intervention studies.
However, this study cannot say for sure that GLP-1 use was the cause of the change in risk of different health outcomes. Such conclusions can only be confidently made from tightly controlled intervention studies, where researchers actively change or control the treatment or behaviour.
The authors note the data used in this study comes from predominantly older, white men so the findings may not apply to other groups.
Also, the large number of participants means that even very small effects can be detected, but they might not actually make a real difference in overall population health.
Observational studies track outcomes over time, but can’t say what caused the changes. Jacob Lund/Shutterstock Other possible reasons for these links
Beyond the effect of GLP-1 in the body, other factors may explain some of the findings in this study. For example, it’s possible that:
- people who used GLP-1 could be more informed about treatment options and more motivated to manage their own health
- people who used GLP-1 may have received it because their health-care team were motivated to offer the latest treatment options, which could lead to better care in other areas that impact the risk of various health outcomes
- people who used GLP-1 may have been able to do so because they lived in metropolitan centres and could afford the medication, as well as other health-promoting services and products, such as gyms, mental health care, or healthy food delivery services.
Did the authors have any conflicts of interest?
Two of the study’s authors declared they were “uncompensated consultants” for Pfizer, a global pharmaceutical company known for developing a wide range of medicines and vaccines. While Pfizer does not currently make readily available GLP-1s such as Ozempic or Wegovy, they are attempting to develop their own GLP-1s, so may benefit from greater demand for these drugs.
This research was funded by the US Department of Veterans Affairs, a government agency that provides a wide range of services to military veterans.
No other competing interests were reported.
Diabetes vs weight-loss treatments
Overall, this study shows people with type 2 diabetes using GLP-1 medication generally have more positive health outcomes than negative health outcomes.
However, the study didn’t include people without type 2 diabetes. More research is needed to understand the effects of these medications in people without diabetes who are using them for other reasons, including weight loss.
While the findings highlight the therapeutic benefits of GLP-1 medications, they also raise important questions about how to manage the potential risks for those who choose to use this medication.
The findings of this study can help many people, including:
- policymakers looking at ways to make GLP-1 medications more widely available for people with various health conditions
- health professionals who have regular discussions with patients considering GLP-1 use
- individuals considering whether a GLP-1 medication is right for them.
Lauren Ball, Professor of Community Health and Wellbeing, The University of Queensland and Emily Burch, Accredited Practising Dietitian and Lecturer, Southern Cross University
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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How to donate your poo to science or medicine
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When most people think about donating body parts to science or medicine, they might think of life-saving donations of organs, tissues or blood. But you can also donate your poo.
The idea is to use it for poo transplants, otherwise known as faecal microbiota transplantation. That’s when poo products made from healthy donor poo are transplanted into another person to improve their health.
Scientists like myself rely on poo donations to run clinical trials into this type of research. Some clinics rely on poo donations to treat patients.
To put it bluntly, we rely on people having a shit for science or medicine.
Here’s how to get involved and become a poo donor.
DBenitostock/Getty Why would you want to?
Think of a poo donation as donating a different type of “organ”, your gut microbiome. This is the community of microbes in your gut responsible for critical functions in the body, including shaping your immune system and how you metabolise food.
We’re learning more about the gut microbiome all the time. This includes identifying functions important to our health and discovering potential new antimicrobial products derived from poo.
To get involved with this type of science, you’d need to donate your poo or make a series of donations at set times that fit into a study’s design. Your microbiome would be profiled and the data used to answer questions relating to that study.
For instance, it was this type of science that led to researchers learning more about how we share our gut microbiome with our social networks, the people we interact with in person, day to day.
Poo donations can be used to treat people. This is now accepted as an option for recurrent infection with the bacterium Clostridioides difficile that hasn’t responded to conventional treatment.
Poo donations have also been explored to treat inflammatory bowel diseases, irritable bowel syndrome, liver diseases, long-term urinary tract infections, mental health issues, improving cancer immunotherapy, and more.
There’s a hierarchy of poo
Not all poo is created equal. All donations from individuals that pass study inclusion and exclusion criteria are welcome for research. But poo donations for treatment need to pass an exceptional threshold of safety and quality.
These poo donors undergo extensive medical screening before selection because of the many unknowns in poo. When we transplant poo, we want to make sure the donor is free from blood-borne viruses (such as HIV or hepatitis). We also want to make sure their poo is free from parasites, and disease-causing viruses and bacteria (such as C. difficile) and certain antibiotic-resistant bacteria.
To complicate matters, a commitment to donate consistently is expected. With that comes ongoing medical screening, which can be time-consuming.
Donors also have to avoid activities that increase their chances of acquiring a blood-borne infection, such as injecting drugs or having unprotected sex. They also have to avoid visiting countries where traveller’s diarrhoea is common.
Poo stability is also an issue as it doesn’t last long without proper storage. This means poo donation only works if you live or work near one of these sites.
All these restrictions quickly reduce the pool of donors we can recruit.
A decade ago we conducted our own clinical trial and quickly became aware of the difficulties of obtaining and maintaining a source of therapeutic poo.
Out of 116 potential donors we screened, an expensive and time-consuming process, only 12 individuals passed. That’s roughly 10%. Many decided not to participate due to the frequency of donations required. Some had medical conditions, parasites or detectable blood in their poo. Others had risk factors for variant Creutzfeldt-Jakob disease, a rare brain disease associated with bovine spongiform encephalopathy (BSE, or “mad cow” disease).
You could improve someone’s life
Is there an upside? You could be saving someone’s life, or at least improving their quality of life significantly.
It is likely your donation will treat someone with recurrent C. difficile infection. Otherwise, it would be used in a clinical trial or study to treat another important medical condition.
As a poo donor, you’d also get a free, extensive and ongoing health check. Depending on where you donate, you might get paid.
However, the more of such health checks you have, the more chance of finding a medical condition (an incidental finding) that may need to be investigated, prompting a cascade of further tests.
Where can I sign up?
There are a number of organisations in Australia that recruit poo donors, including:
- Australian Red Cross Lifeblood recruits unpaid donors in Perth. You can check online if you’re eligible
- BiomeBank is a company that recruits paid donors in Adelaide based on certain criteria and assessments
- the Centre for Digestive Diseases is a private Sydney clinic and research centre that screens donors before selection. Paid donors are asked to follow dietary recommendations to maximise the quality of their donation.
What’s the take-home message?
We’re a long way from replicating the entire gut microbial community in the lab. So we have to rely on live microbial products made from donated poo as research moves from the laboratory bench to the clinic.
As with all health products, the benefits and evidence need to be weighed with caution.
Yet, if we unlock the potential of the gut microbiome via donated poo, this opens exciting avenues to develop probiotics and more therapeutics.
Nadeem O. Kaakoush, Scientia Associate Professor, Host-Microbiome Interactions Group, UNSW Sydney
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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Blueberries vs Redcurrants – Which is Healthier?
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Our Verdict
When comparing blueberries to redcurrants, we picked the redcurrants.
Why?
Both are great! But…
In terms of macros, blueberries have more carbs while redcurrants have nearly 2x more fiber as well as more protein; most of the numbers are small, but by virtue of the greater fiber, redcurrants win this round.
In the category of vitamins, blueberries have more of vitamins A, B3, B5, E, and K, while redcurrants have more of vitamins B1, B2, B6, B7, B9, and C, for a marginal win in this round.
Looking at minerals, blueberries have a little more manganese, while redcurrants have a lot more calcium, copper, iron, magnesium, phosphorus, potassium, selenium, and zinc, winning this round easily.
In other considerations, both are excellent sources of polyphenols, and the numbers (and variety) are close enough that this one will be decided by individual variation from one crop to the next. So in the interest of fairness, we’ll call this round a tie.
Adding up the sections makes for a clear overall win for redcurrants, but by all means do enjoy either or both, as diversity is best!
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21 Most Beneficial Polyphenols & What Foods Have Them
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How can I improve my running? 5 top tips for every runner, from a biomechanics expert
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Humans and our ancestors have been running for millions of years. Back then, it helped us capture – or avoid becoming – prey. Now, we do it to keep fit, boost mental health, unwind in nature, or play our favourite sport.
But while many of us were taught how to ride a bike, throw and catch a ball, or kick a footy, it seems very few people are ever taught how to run. You might’ve wondered: am I running wrong?
Well, the truth is there’s no one right way to run. Your ideal technique depends on factors such as leg and foot length, muscle mass, and even how springy your tendons are.
It also depends on whether you’re out for your Sunday run or running full pelt in a sprint.
That said, thinking a little more about how to run can make it feel easier and faster, and reduce injury risk.
Here are five basics to keep in mind.
Thinking a little more about how to run can make it feel easier and faster. Rocksweeper/Shutterstock 1. Feet: how you land matters
Some of us land on our heels, others on the balls of our feet. If you grew up running barefoot, you’ll more often land towards the forefoot.
Debate rages on which is best. The truth is heel-first striking stresses the knees a bit more while forefoot landing places more impact on the calves and Achilles tendon.
So, if you’re injury prone in one of those areas, it might be worth adjusting your style.
But for healthy runners, there’s no strong evidence one technique is better for injury.
If you’re considering a change, do it slowly over several months, ideally with expert help.
As you run faster, you’ll bounce more in each step. You’ll naturally land more on your forefoot, especially when sprinting.
Majdanski/Shutterstock 2. Legs: softer landings and smoother strides
Three things are worth focusing on:
- minimise the twisting of the legs under your body as you land, to reduce strain on knees and ankles
- keep your pelvis level during landings (dropping or rotating it increases injury risk)
- don’t bounce too high; a smooth, low trajectory uses less energy and keeps impacts manageable.
These principles are perfectly demonstrated by Ethiopian former long-distance runner Haile Gebrselassie:
Just keep relaxed, and allow the knees and ankles to flex normally.
If you find your landing style causes stress or pain, consider running with slightly shorter strides.
Then there’s the “leg recovery phase” – when your leg swings forward after push-off. During jogging, we pull the leg forward briefly with our hip muscles, but otherwise it’s a pretty passive task.
In sprinting, however, the faster leg recovery powered by your hip can contribute about 25% of your forward propulsion in each step. So make sure you flex at the hip while you push back into the ground, so your legs act like scissors as they swing.
Also, the faster you run, the more your knee should flex, and the more the foot should rise under you. This helps the leg swing forwards faster.
In other words: pick your feet up more as you pick up the pace.
3. Arms: built-in shock absorbers
During jogging, your arms help with balance, absorbing bumps or stumbles, especially on uneven ground, as seen here: https://www.youtube.com/embed/ifctluuNkXE?wmode=transparent&start=0
They swing mostly passively and act as shock absorbers during jogging; they can’t do their job when they’re stiff. Relaxation is key.
To keep energy cost low, try bending your elbows to keep their mass closer to your shoulder and keep your shoulders relaxed.
When sprinting, your arms become more active. They help stabilise your whole body in the short time your feet are on the ground.
Top sprint coaches often insist the “drive arm” (the arm swinging backwards) contributes to forward propulsion, thanks to physics.
But the limited studies to date suggest the effect on propulsion is moderate; future studies might shed more light.
That said, the fastest sprinters, like Usain Bolt, are renowned for their aggressive backwards arm drive: https://www.youtube.com/embed/D09QkQ8Cyow?wmode=transparent&start=0
See how his drive arm whips backwards with rapid extension of the shoulder and elbow? Meanwhile, the recovery arm – swinging forwards – is more flexed and moves much slower.
4. Torso: lean just a little
When we run, the torso naturally rotates left and right. That’s fine, although when we run faster there should be less rotation. A more aggressive arm swing helps balance out these rotations.
Our pelvis then rotates in the opposite direction to the torso. The twisting helps us balance, but also contributes a little to forward force.
But as we run faster, these rotations should become smaller as we use our arms to balance better. As your speed increases, swing your arms a bit harder and your body, legs and other arm will follow.
Finally, it’s generally accepted that we keep our torso upright when we run relaxed, with only a very slight forward lean.
But if we want to speed up, leaning forward is a great way to accelerate quickly without doing too much tiring muscle work.
And for those with knee troubles, leaning forward a bit might help reduce impact on the knees.
If you’re not sure how you run, try asking a friend to take a quick video of you running. Demkat/Shutterstock 5. Head: a balancing act
You might be tempted to tilt your head down when you run, to watch your feet or in an effort to accelerate forwards.
But during upright (non-sprinting) running, try to keep it in normal position. Rest your head quietly on the top of your shoulders, just as as evolution intended.
During sprinting, try looking about 20 metres in front of you (a slight chin tuck is fine). When jogging, try looking ahead toward the horizon.
Not sure what your own technique looks like? Try asking a friend to take a quick video of you running. Compare it to an experienced runner running at the same speed.
You might be surprised what you notice.
Anthony Blazevich, Professor of Biomechanics, Edith Cowan University
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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We’re the ‘allergy capital of the world’. But we don’t know why food allergies are so common in Australian children
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Australia has often been called the “allergy capital of the world”.
An estimated one in ten Australian children develop a food allergy in their first 12 months of life. Research has previously suggested food allergies are more common in infants in Australia than infants living in Europe, the United States or Asia.
So why are food allergies so common in Australia? We don’t know exactly – but local researchers are making progress in understanding childhood allergies all the time.
Miljan Zivkovic/Shutterstock What causes food allergies?
There are many different types of reactions to foods. When we refer to food allergies in this article, we’re talking about something called IgE-mediated food allergy. This type of allergy is caused by an immune response to a particular food.
Reactions can occur within minutes of eating the food and may include swelling of the face, lips or eyes, “hives” or welts on the skin, and vomiting. Signs of a severe allergic reaction (anaphylaxis) include difficulty breathing, swelling of the tongue, swelling in the throat, wheeze or persistent cough, difficulty talking or a hoarse voice, and persistent dizziness or collapse.
Recent results from Australia’s large, long-running food allergy study, HealthNuts, show one in ten one-year-olds have a food allergy, while around six in 100 children have a food allergy at age ten.
A food allergy can present with skin reactions. comzeal images/Shutterstock In Australia, the most common allergy-causing foods include eggs, peanuts, cow’s milk, shellfish (for example, prawn and lobster), fish, tree nuts (for example, walnuts and cashews), soybeans and wheat.
Allergies to foods like eggs, peanuts and cow’s milk often present for the first time in infancy, while allergies to fish and shellfish may be more common later in life. While most children will outgrow their allergies to eggs and milk, allergy to peanuts is more likely to be lifelong.
Findings from HealthNuts showed around three in ten children grew out of their peanut allergy by age six, compared to nine in ten children with an allergy to egg.
Are food allergies becoming more common?
Food allergies seem to have become more common in many countries around the world over recent decades. The exact timing of this increase is not clear, because in most countries food allergies were not well measured 40 or 50 years ago.
We don’t know exactly why food allergies are so common in Australia, or why we’re seeing a rise around the world, despite extensive research.
But possible reasons for rising allergies around the world include changes in the diets of mothers and infants and increasing sanitisation, leading to fewer infections as well as less exposure to “good” bacteria. In Australia, factors such as increasing vitamin D deficiency among infants and high levels of migration to the country could play a role.
In several Australian studies, children born in Australia to parents who were born in Asia have higher rates of food allergies compared to non-Asian children. On the other hand, children who were born in Asia and later migrated to Australia appear to have a lower risk of nut allergies.
Meanwhile, studies have shown that having pet dogs and siblings as a young child may reduce the risk of food allergies. This might be because having pet dogs and siblings increases contact with a range of bacteria and other organisms.
This evidence suggests that both genetics and environment play a role in the development of food allergies.
We also know that infants with eczema are more likely to develop a food allergy, and trials are underway to see whether this link can be broken.
Can I do anything to prevent food allergies in my kids?
One of the questions we are asked most often by parents is “can we do anything to prevent food allergies?”.
We now know introducing peanuts and eggs from around six months of age makes it less likely that an infant will develop an allergy to these foods. The Australasian Society of Clinical Immunology and Allergy introduced guidelines recommending giving common allergy-causing foods including peanut and egg in the first year of life in 2016.
Our research has shown this advice had excellent uptake and may have slowed the rise in food allergies in Australia. There was no increase in peanut allergies between 2007–11 to 2018–19.
Introducing other common allergy-causing foods in the first year of life may also be helpful, although the evidence for this is not as strong compared with peanuts and eggs.
Giving kids peanuts early can reduce the risk of a peanut allergy. Madame-Moustache/Shutterstock What next?
Unfortunately, some infants will develop food allergies even when the relevant foods are introduced in the first year of life. Managing food allergies can be a significant burden for children and families.
Several Australian trials are currently underway testing new strategies to prevent food allergies. A large trial, soon to be completed, is testing whether vitamin D supplements in infants reduce the risk of food allergies.
Another trial is testing whether the amount of eggs and peanuts a mother eats during pregnancy and breastfeeding has an influence on whether or not her baby will develop food allergies.
For most people with food allergies, avoidance of their known allergens remains the standard of care. Oral immunotherapy, which involves gradually increasing amounts of food allergen given under medical supervision, is beginning to be offered in some facilities around Australia. However, current oral immunotherapy methods have potential side effects (including allergic reactions), can involve high time commitment and cost, and don’t cure food allergies.
There is hope on the horizon for new food allergy treatments. Multiple clinical trials are underway around Australia aiming to develop safer and more effective treatments for people with food allergies.
Jennifer Koplin, Group Leader, Childhood Allergy & Epidemiology, The University of Queensland and Desalegn Markos Shifti, Postdoctoral Research Fellow, Child Health Research Centre, Faculty of Medicine, The University of Queensland
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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