
Foods For & Against Hiatus Hernia
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It’s Q&A Day at 10almonds!
Have a question or a request? We love to hear from you!
In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!
As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!
So, no question/request too big or small 😎
❝How does diet impact hiatus hernias?❞
Short answer: indirectly
More useful answer:
- Diet that favors obesity is more likely to result in getting a hiatus hernia, because obesity is one of the risk factors for it.
- Once you have a hiatus hernia, one of the more likely consequences is gastroesophageal reflux disease (GERD).
- Diet that is high in acids, fats, and/or spices will tend to worsen the GERD symptoms, as will alcohol.
We’ll cover the relevant dietary decisions involved in a moment, but first, for anyone wondering:
What actually is a hiatus hernia?
- A hernia (in general) is when an organ, or more often just part of an organ, “escapes” from where it is supposed to be kept in place, and thus finds itself somewhere it shouldn’t. The result is not usually very dangerous (although some can be, depending on what and where it is), and/but it’s often painful.
- A hiatus hernia is a hernia in which part of the stomach finds itself above the diaphragm, sneaking up where the esophagus makes its way through. This is usually periodic in nature, i.e. it doesn’t go there and get stuck and stay there; it sometimes slips back down, but easily makes its way back up again in response to certain conditions. On which note…
Calming it down (and keeping it down)
Since obesity is a risk factor, losing weight is indicated if you’re carrying excess fat. We’ll keep it simple here for the sake of space, but the biggest dietary risk factor for obesity is excessive quick-release carbs without sufficient fiber to accompany them.
So, to reverse that, getting plenty of fiber is good, as is getting plenty of protein to increase satiety signals, and getting at least enough good quality carbs and fats to give you enough energy that your body doesn’t think starvation is at hand (which perceived threat it would respond to by slowing down your metabolism and storing fat wherever/however possible).
For a more comprehensive approach that’s easy* to apply, see: How To Lose Weight (Healthily!)
*Unless there are other factors, e.g. food poverty and/or comorbidities that make healthy eating more difficult. But even in those cases, it’s good to know what to aim for, to be able to make the best choices where choices are available.
As a quick aside, we’re focusing on the food-related side of things because the question was about diet, but please do understand there are other risk factors for hernia that are more important than diet, including:
- genetic risk factors that you can’t control at all, and can only really be aware of as an extra cause for caution (either by health genomics services or by knowing about a family history of hernia)
- aging which you technically can control somewhat because the pace of biological aging is not set in stone (but as it stands, old age is coming for us all sooner or later if something else doesn’t get us first)
- frequent/hard coughing, sneezing, and/or vomiting, which are not usually optional activities, and this means that other maladies can lead to an increased risk of hernia through no fault of our own
Now, let us assume you already have a hiatus hernia and would like it to kindly stop herniating.
One thing to do is the same as we ideally would have done to avoid it, which is (again) weight loss, if applicable.
Another thing to do is to tailor one’s diet to reduce the symptoms of GERD, which as we mentioned up top, is one of the common consequences of hiatus hernia.
GERD has no known cure once established, but its symptoms can be managed by:
- Healthy eating (Mediterranean diet is, as usual, great)
- Weight loss (if and only if obese)
- Avoiding trigger foods (acidic, spicy, fatty*)
- Eating smaller meals
- Practicing mindful eating
- Staying upright for 3–4 hours after eating
And of course, don’t smoke, and ideally don’t drink alcohol.
*about avoiding fatty foods when we told you above to get at least enough good quality fats: the trick here is to enjoy high-quality fats little and often**, and avoid unduly oily cooking. And certainly, deep-frying anything is not what you want here.
**about “little and often”: this is very important, because part of the problem that causes GERD, and this is exacerbated in hiatus hernia, is physical in nature. Your stomach is somewhat stretchy but still limited in size. How much it can expand does depend on some other factors, for example, if you have more abdominal fat, it will have less room to expand—because the fat is packing it inwards and the stomach contents must push against that, meaning that by the laws of motion and fluid mechanics, the weight of fat from the outside exerts a force that can squeeze the stomach contents (per GERD) and/or the stomach itself (per hiatus hernia) up in the direction of your esophagus. With this in mind, “little and often” means there is, at any given time, less in your stomach and thus less chance of having your stomach contents (or indeed the stomach itself) pushed so far up that it ends up making its way out.
You can read more about GERD (and the different ways it can go from there), here:
NICE | Gastro-oesophageal reflux disease
Note: this above page refers to it as “GORD”, because of the British English spelling of “oesophagus” rather than “esophagus”. It’s the exact same organ and condition, just a different spelling.
Take care!
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Raw vs Cooked Oats: Two Very Different Health Impacts
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Which is better, raw or cooked? Can you guess?
Oat so simple
In fact, both are great, and/but both are great in different ways!
It comes down to β-glucan, the soluble fiber in oats that affects cholesterol, blood sugar, and gut health in different ways depending on how it’s processed:
- Raw oats (e.g. overnight oats) yield more intact β-glucan: research shows about 26% digestion vs 9% for cooked oats, meaning more of the fiber stays structurally intact, and thus reaches your large intestine, where gut bacteria ferment it into short-chain fatty acids like butyrate, which supports gut health, reduces inflammation, beneficially influences metabolism, and can even beneficially affect brain function, via the gut–brain axis.
- Cooked oats yield greater solubility: heating breaks down cell walls, making β-glucan more soluble and viscous in your gut. This too is important, it helps reduce cholesterol absorption and slows glucose spikes, which improvements have positive effects on many aspects of health, including the heart, liver, and brain.
So, in fewer words:
- Raw/overnight oats: better for gut health and butyrate production
- Cooked oats: better for cholesterol and glucose control
For more on all of this, enjoy:
Click Here If The Embedded Video Doesn’t Load Automatically!
Want to learn more?
You might also like:
- The Best Kind Of Fiber For Overall Health? ← yes, it’s β-glucan; you can read more about the science of it here
- What Two Days Of Oats Will Do To Your Lipids ← it’s pretty impressive!
- Nutrivore – by Dr. Sarah Ballantyne ← this is the Dr. Ballantyne whose work was cited in the video 🙂
- Spiced Pear & Pecan Polyphenol Porridge ← a recipe we enjoy and you might too
- Anti-Cholesterol Cardamom & Pistachio Porridge ← another recipe!
Take care!
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Semaglutide’s Surprisingly Unexamined Effects
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Semaglutide’s Surprisingly Big Research Gap
GLP-1 receptor agonists like Ozempic, Wegovy, and other semaglutide drugs. are fast becoming a health industry standard go-to tool in the weight loss toolbox. When it comes to recommending that patients lose weight, “Have you considered Ozempic?” is the common refrain.
Sometimes, this may be a mere case of kicking the can down the road with regard to some other treatment that it can be argued (sometimes even truthfully) would go better after some weight loss:
How weight bias in health care can harm patients with obesity: Research
…which we also covered in fewer words in the second-to-last item here:
But GLP-1 agonists work, right?
Yes, albeit there’s a litany of caveats, top of which are usually:
- there are often adverse gastrointestinal side effects
- if you stop taking them, weight regain generally ensues promptly
For more details on these and more, see:
…but now there’s another thing that’s come to light:
The dark side of semaglutide’s weight loss
In academia, “dark” is often used to describe “stuff we don’t have much (or in some cases, any) direct empirical evidence of, but for reasons of surrounding things, we know it’s there”.
Well-known examples include “dark matter” in physics and the Dark Ages in (European) history.
In the case of semaglutide and weight loss, a review by a team of researchers (Drs. Sandra Christenen, Katie Robinson, Sara Thomas, and Dominique Williams) has discovered how little research has been done into a certain aspect of GLP-1 agonist’s weight loss effects, namely…
Dietary changes!
There’s been a lot of popular talk about “people taking semaglutide eat less”, but it’s mostly anecdotal and/or presumed based on parts of the mechanism of action (increasing insulin production, reducing glucagon secretions, modulating dietary cravings).
Where studies have looked at dietary changes, it’s almost exclusively been a matter of looking at caloric intake (which has been found to be a 16–39% reduction), and observations-in-passing that patients reported reduction in cravings for fatty and sweet foods.
This reduction in caloric intake, by the way, is not significantly different to the reduction brought about by counselling alone (head-to-head studies have been done; these are also discussed in the research review).
However! It gets worse. Very few studies of good quality have been done, even fewer (two studies) actually had a registered dietitian nutritionist on the team, and only one of them used the “gold standard” of nutritional research, the 24-hour dietary recall test. Which, in case you’re curious, you can read about what that is here:
Dietary Assessment Methods: What Is A 24-Hour Recall?
Of the four studies that actually looked at the macros (unlike most studies), they found that on average, protein intake decreased by 17.1%. Which is a big deal!
It’s an especially big deal, because while protein’s obviously important for everyone, it’s especially important for anyone trying to lose weight, because muscle mass is a major factor in metabolic base rate—which in turn is much important for fat loss/maintenance than exercise, when it comes to how many calories we burn by simply existing.
A reasonable hypothesis, therefore, is that one of the numerous reasons people who quit GLP-1 agonists immediately put fat back on, is because they probably lost muscle mass in amongst their weight loss, meaning that their metabolic base rate will have decreased, meaning that they end up more disposed to put on fat than before.
And, that’s just a hypothesis and it’s a hypothesis based on very few studies, so it’s not something to necessarily take as any kind of definitive proof of anything, but it is to say—as the researchers of this review do loudly say—more research needs to be done into this, because this has been a major gap in research so far!
Any other bad news?
While we’re talking research gaps, guess how many studies looked into micronutrient intake changes in people taking GLP-1 agonists?
If you guessed zero, you guessed correctly.
You can find the paper itself here:
What’s the main take-away here?
On a broad, scoping level: we need more research!
On a “what this means for individuals who want to lose weight” level: maybe we should be more wary of this still relatively new (less than 10 years old) “wonder drug”. And for most of those 10 years it’s only been for diabetics, with weight loss use really being in just the past few years (2021 onwards).
In other words: not necessarily any need to panic, but caution is probably not a bad idea, and natural weight loss methods remain very reasonable options for most people.
See also: How To Lose Weight (Healthily!)
Take care!
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Bell Pepper vs Zucchini – Which is Healthier?
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Our Verdict
When comparing bell pepper to zucchini, we picked the bell pepper.
Why?
In terms of macros, bell peppers have nearly 2x the fiber for slightly more carbs and comparable (negligible) protein, winning this category.
In the category of vitamins, amounts of vitamins A and C do vary by bell pepper color (more on that in the “learn more” section below), but even using the most conservative numbers for each, bell peppers have more of vitamins A, B1, B3, B6, C, E, and K, while zucchini has more of vitamins B2, B5, and B9, giving bell peppers a 7:3 win here.
Looking at minerals, bell peppers have more copper, while zucchini have more calcium, iron, magnesium, manganese, phosphorus, potassium, selenium, and zinc, giving zucchini a compelling win in this round.
In other considerations, bell peppers have more polyphenols (especially quercetin and luteolin), as well as some good carotenoids not otherwise covered, such as lutein, so this round’s another win for bell peppers.
Adding up the sections makes for a clear overall win for bell peppers, but by all means enjoy either or both, as diversity is good (and those minerals are great)!
Want to learn more?
You might like:
- Brain Food? The Eyes Have It! ← this is mostly about lutein
- Which Bell Peppers To Pick? A Spectrum Of Specialties ← for the differences between the different colors
Enjoy!
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How Acetaminophen (Paracetamol) Suppresses Endocannabinoids
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…contrary to all expectations (and how this can lead to more effective acetaminophen-like pain relief, without the toxicity!
It’s well known that acetaminophen (international name: paracetamol, most well-known brand name in the US: Tylenol) is a mild painkiller and anti-fever agent; what’s not well understood is how it actually works.
Recently, a team of researchers (Dr. Michaela Dvorakova et al.) looked into this, and found something that was quite surprising, to understate it considerably.
That sounds like a clickbait headline (“and what they discovered will shock you!”) but well, we don’t do clickbait and our own headline covers it: acetaminophen suppresses endocannabinoids, when generally for painkilling purposes we want more endocannabinoids, not less.
Before we get into that, let’s take a moment to note some of the bad parts of acetaminophen’s safety profile, which is to say: it’s really not very safe.
Rather than repeat ourselves though, we’ll link to where we did a whole main feature about that, here: Before You Reach For That Tylenol…
Now, about acetaminophen and endocannabinoids
In the lead researcher’s words:
❝There are hypotheses, but we still don’t know precisely how it works. Up until now we thought that elevated endocannabinoids in our body meant less pain, but our study shows that in the case of 2-AG, it might be the opposite. Actually, reduced levels of 2-AG leads to decreased pain.❞
~ Dr. Michaela Dvorakova
You may be wondering: what’s 2-AG?
And the answer is that it’s 2-arachidonoyl glycerol, which is a cannabinoid naturally made by the human body (thus, endogenous cannabinoid, or usually written: endocannabinoid).
What the researchers found is that acetaminophen inhibits an enzyme that makes that endocannabinoid, namely, diacylglycerol lipase α (DAGLα) ← as in, this is the name of the enzyme that makes it.
Thus, inhibiting the enzyme means inhibiting endocannabinoid production. So, what gives, and why does this work as a painkiller, when it looks like it’s doing the opposite?
The researchers propose… Well, we’ll quote them:
❝This gives rise to the counterintuitive hypothesis that decreasing endocannabinoid production by DAGLα inhibition may be antinociceptive in certain settings.
Supporting this hypothesis, we find that diacylglycerol lipase (DAGL) inhibition by RHC80267 is antinociceptive in wild-type but not CB1 knockout mice in the hot-plate test.
We propose (1) that activation of DAGLα may exacerbate some forms of nociception and (2) a mechanism for the antinociceptive actions of acetaminophen, whereby acetaminophen inhibits a DAGLα/CB1-based circuit that plays a permissive role in at least one form of nociception.❞
Translating from sciencese: by stopping the endocannabinoid production, the body is triggered to engage a different, more effective method of killing pain.
You can read the paper in full here: Acetaminophen inhibits diacylglycerol lipase synthesis of 2-arachidonoyl glycerol: Implications for nociception
As for what the implications are? Per the researchers, it means that if we understand that inhibiting the enzyme DAGLα triggers a painkilling response, then new drugs can be designed to target DAGLα without the toxicity of acetaminophen.
So, keep an eye out for that, and you heard it hear first!
Want to learn more?
We’ve written quite a bit about pain management, including:
- How To Stop Pain Spreading
- How To Dial Down Your Pain
- Managing Chronic Pain (Realistically!)
- Get The Right Help For Your Pain
- The 7 Approaches To Pain Management
- Science-Based Alternative Pain Relief (When Painkillers Aren’t Helping, These Things Might)
Take care!
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Pumpkin vs Tomatoes – Which is Healthier?
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
Our Verdict
When comparing pumpkin to tomatoes, we picked the pumpkin.
Why?
Both have their merits, but there was a clear winner:
In terms of macros, pumpkin has nearly 3x the fiber, 2x the carbs, and slightly more protein (which latter isn’t much), meaning a win for pumpkin in this round, mostly on account of the fiber.
In the category of vitamins, pumpkin has a lot more of vitamins A, B2, B5, B7, E, and K, while tomato has more of vitamins B1, B3, B6, B9, and C, yielding a marginal 6:5 win to pumpkin.
Looking at minerals, it’s less close; pumpkin has more calcium, copper, iron, magnesium, manganese, phosphorus, and selenium, while tomato has (slightly) more potassium, giving pumpkin an easy 7:1 victory here.
In other considerations, it’s worth noting that both of these plants are good sources of carotenoids including lutein and lycopene, so we’ll call this round a tie.
Adding up the sections makes for an overall win for pumpkin, but by all means enjoy either or both, as diversity is good!
Want to learn more?
You might like:
- Brain Food? The Eyes Have It! ← this is mostly about lutein
- Lycopene’s Benefits For The Gut, Heart, Brain, & More
Enjoy!
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I Will Make You Passionate About Exercise – by Bevan Eyles
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
What this isn’t: a “just do it!” motivational pep-talk.
What this is:a compassionate and thoughtful approach to help non-exercisers become regular exercisers, by looking at the real life factors of what holds people back (learning from his own early failures as a coach, by paying attention now to things he inadvertently neglected back then), both in the material/practical and in the psychological/emotional.
Further, he gives a 10-step method, for those who would like to be walked through it by the hand, making the transition to exercising regularly (and as a leisure habit, rather than as a chore) as frictionless as possible.
The style is friendly and energetic, and very easy-reading throughout.
Bottom line: if you are someone who finds exercising to be a chore, this book can definitely help you “get from here to there” in terms of finding joy in it, and finding exercise even easier than not exercising. Yes, really.
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