Nutritional Profiles to Recipes

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It’s Q&A Day at 10almonds!

Have a question or a request? We love to hear from you!

In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!

As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!

So, no question/request too big or small

❝I like the recipes. Most don’t seem to include nutritional profile. would lilke to see that. Macro/micro world…. Thank you❞

We’re glad you’re enjoying them! There are a couple of reasons why we don’t, but the reasons can be aggregated into one (admittedly rare) concept: honesty

To even try to give you these figures, we’d first need to use the metric system (or at least, a strictly mass-based system) which would likely not go well with our largely American readership, because “half a bulb of garlic, or more if you like”, and “1 cucumber” or “1 cup chopped carrot” could easily way half or twice as much, depending on the sizes of the vegetables or the chopping involved, and in the case of chopped vegetables measured by the cup, even the shape of the cup (because of geometry and the spaces left; it’s like Tetris in there). We can say “4 cups low-sodium broth” but we can’t say how much sodium is in your broth. And so on.

And that’s without getting into the flexibility we offer with substitutions, often at a rate of several per recipe.

We’d also need to strictly regulate your portion sizes for you, because we (with few exceptions, such as when they are a given number of burger patties, or a dessert-in-a-glass, etc) give you a recipe for a meal and leave it to you how you divide it and whether there’s leftovers.

Same goes for things like “Extra virgin olive oil for frying”; a recipe could say to use “2 tbsp” but let’s face it, you’re going to use what you need to use, and that’s going to change based on the size of your pan, how quickly it’s absorbed into the specific ingredients that you got, which will change depending on how fresh they are, and things like that.

By the time we’ve factored in your different kitchen equipment, how big your vegetables are, the many factors effecting how much oil you need, substitutions per recipe per making something dairy-free, or gluten-free, or nut-free, etc, how big your portion size is (we all know that “serves 4” is meaningless in reality)… Even an estimated average would be wildly misleading.

So, in a sea of recipes saying “500 kcal per serving” from the same authors who say you can caramelize onions in 4–5 minutes “or until caramelized” and then use the 4–5 minutes figure for calculating the overall recipe time… We prefer to stay honest.

PS: for any wondering, caramelizing onions takes closer to 45 minutes than 4–5 minutes, and again will depend on many factors, including the onions, how finely you chopped them, the size and surface of your pan, the fat you’re using, whether you add sugar, what kind, how much you stir them, the mood of your hob, and the phase of the moon. Under very favorable circumstances, it could conceivably be rushed in 20 minutes or so, but it could also take 60. Slow-cooking them (i.e. in a crock pot) over 3–4 hours is a surprisingly viable “cheat” option, by the way. It’ll take longer, obviously, but provided you plan in advance, they’ll be ready when you need them, and perfectly done (the same claim cannot be made if you budgeted 4–5 minutes because you trusted a wicked and deceitful author who wants to poop your party).

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  • Fast Diet, Fast Exercise, Fast Improvements

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    Diet & Exercise, Optimized

    This is Dr. Michael Mosley. He originally trained in medicine with the intention of becoming a psychiatrist, but he grew disillusioned with psychiatry as it was practised, and ended up pivoting completely into being a health educator, in which field he won the British Medical Association’s Medical Journalist of the Year Award.

    He also died under tragic circumstances very recently (he and his wife were vacationing in Greece, he went missing while out for a short walk on the 5th of June, appears to have got lost, and his body was found 100 yards from a restaurant on the 9th). All strength and comfort to his family; we offer our small tribute here today in his honor.

    The “weekend warrior” of fasting

    Dr. Mosley was an enjoyer (and proponent) of intermittent fasting, which we’ve written about before:

    Fasting Without Crashing? We Sort The Science From The Hype

    However, while most attention is generally given to the 16:8 method of intermittent fasting (fast for 16 hours, eat during an 8 hour window, repeat), Dr. Mosley preferred the 5:2 method (which generally means: eat at will for 5 days, then eat a reduced calorie diet for the other 2 days).

    Specifically, he advocated putting that cap at 800 kcal for each of the weekend days (doesn’t have to be specifically the weekend).

    He also tweaked the “eat at will for 5 days” part, to “eat as much as you like of a low-carb Mediterranean diet for 5 days”:

    ❝The “New 5:2” approach involves restricting calories to 800 on fasting days, then eating a healthy lower carb, Mediterranean-style diet for the rest of the week.

    The beauty of intermittent fasting means that as your insulin sensitivity returns, you will feel fuller for longer on smaller portions. This is why, on non-fasting days, you do not have to count calories, just eat sensible portions. By maintaining a Mediterranean-style diet, you will consume all of the healthy fats, protein, fibre and fresh plant-based food that your body needs.❞

    ~ Dr. Michael Mosley

    Read more: The Fast 800 | The New 5:2

    And about that tweaked Mediterranean Diet? You might also want to check out:

    Four Ways To Upgrade The Mediterranean Diet

    Knowledge is power

    Dr. Mosley encouraged the use of genotyping tests for personal health, not just to know about risk factors, but also to know about things such as, for example, whether you have the gene that makes you unable to gain significant improvements in aerobic fitness by following endurance training programs:

    The Real Benefit Of Genetic Testing

    On which note, he himself was not a fan of exercise, but recognised its importance, and instead sought to minimize the amount of exercise he needed to do, by practising High Intensity Interval Training. We reviewed a book of his (teamed up with a sports scientist) not long back; here it is:

    Fast Exercise: The Simple Secret of High Intensity Training – by Dr. Michael Mosley & Peta Bee

    You can also read our own article on the topic, here:

    How To Do HIIT (Without Wrecking Your Body)

    Just One Thing…

    As well as his many educational TV shows, Dr. Mosley was also known for his radio show, “Just One Thing”, and a little while ago we reviewed his book, effectively a compilation of these:

    Just One Thing: How Simple Changes Can Transform Your Life – by Dr. Michael Mosley

    Enjoy!

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  • Seed Saving Secrets – by Alice Mirren

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    We all know that home-grown is best, and yet many of us are not exactly farmers (this reviewer tries with mixed results—hardy crops survive; others, not so much). While it’s easy to blame the acidic soil, the harsh climate, or not having enough time and money (this reviewer blames all of the above), the fact remains that a skilled gardener can produce a good crop in any conditions.

    That’s where this book helps; right from the beginning, from the seeds. Have you ever bought a pack of seeds, excitedly sown them, and then had a germination rate of zero or something close to that (this reviewer has)?

    Alice Mirren takes us on a tour of how to save seeds from plants you know are regionally viable (not the product of some vast globalized industry that doesn’t know you live in an ancient bog with a cold south-east wind blowing in from Siberia), and then how to care for and curate them, how to store them for future years, how to keep a self-perpetuating seed bank.

    She goes beyond that, though. Regular 10almonds readers might remember about the supercentenarian “Blue Zones”, and how big factors in healthy longevity include community and purpose; Mirren advocates for organizing community seed banks, which will also mean that everyone (including you) has access to much more diverse seeds, and when it comes to the perils of natural selection, diversity means survival. Otherwise, if you have just one seed type, a single blight can wipe out everything pretty much overnight.

    Bottom line: if you grow your own food or would like to, this is a “bible of…” level book that you absolutely should have to hand.

    Click here to check out Seed Saving Secrets, and see the results in your kitchen and on your plate!

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  • New study suggests weight loss drugs like Ozempic could help with knee pain. Here’s why there may be a link

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    The drug semaglutide, commonly known by the brand names Ozempic or Wegovy, was originally developed to help people with type 2 diabetes manage their blood sugar levels.

    However, researchers have discovered it may help with other health issues, too. Clinical trials show semaglutide can be effective for weight loss, and hundreds of thousands of people around the world are using it for this purpose.

    Evidence has also shown the drug can help manage heart failure and chronic kidney disease in people with obesity and type 2 diabetes.

    Now, a study published in the New England Journal of Medicine has suggested semaglutide can improve knee pain in people with obesity and osteoarthritis. So what did this study find, and how could semaglutide and osteoarthritis pain be linked?

    Pormezz/Shutterstock

    Osteoarthritis and obesity

    Osteoarthritis is a common joint disease, affecting 2.1 million Australians. Most people with osteoarthritis have pain and find it difficult to perform common daily activities such as walking. The knee is the joint most commonly affected by osteoarthritis.

    Being overweight or obese is a major risk factor for osteoarthritis in the knee. The link between the two conditions is complex. It involves a combination of increased load on the knee, metabolic factors such as high cholesterol and high blood sugar, and inflammation.

    For example, elevated blood sugar levels increase the production of inflammatory molecules in the body, which can damage the cartilage in the knee, and lead to the development of osteoarthritis.

    Weight loss is strongly recommended to reduce the pain of knee osteoarthritis in people who are overweight or obese. International and Australian guidelines suggest losing as little as 5% of body weight can help.

    But losing weight with just diet and exercise can be difficult for many people. One study from the United Kingdom found the annual probability of people with obesity losing 5% or more of their body weight was less than one in ten.

    Semaglutide has recently entered the market as a potential alternative route to weight loss. It comes from a class of drugs known as GLP-1 receptor agonists and works by increasing a person’s sense of fullness.

    Semaglutide for osteoarthritis?

    The rationale for the recent study was that while we know weight loss alleviates symptoms of knee osteoarthritis, the effect of GLP-1 receptor agonists was yet to be explored. So the researchers set out to understand what effect semaglutide might have on knee osteoarthritis pain, alongside body weight.

    They randomly allocated 407 people with obesity and moderate osteoarthritis into one of two groups. One group received semaglutide once a week, while the other group received a placebo. Both groups were treated for 68 weeks and received counselling on diet and physical activity. At the end of the treatment phase, researchers measured changes in knee pain, function, and body weight.

    As expected, those taking semaglutide lost more weight than those in the placebo group. People on semaglutide lost around 13% of their body weight on average, while those taking the placebo lost around 3% on average. More than 70% of people in the semaglutide group lost at least 10% of their body weight compared to just over 9% of people in the placebo group.

    A man outdoors holding his knee.
    Osteoarthritis of the knee is the most common type of osteoarthritis. SKT Studio/Shutterstock

    The study found semaglutide reduced knee pain significantly more than the placebo. Participants who took semaglutide reported an additional 14-point reduction in pain on a 0–100 scale compared to the placebo group.

    This is much greater than the pain reduction in another recent study among people with obesity and knee osteoarthritis. This study investigated the effects of a diet and exercise program compared to an attention control (where participants are provided with information about nutrition and physical activity). The results here saw only a 3-point difference between the intervention group and the control group on the same scale.

    The amount of pain relief reported in the semaglutide trial is also larger than that reported with commonly used pain medicines such as anti-inflammatories, opioids and antidepressants.

    Semaglutide also improved knee function compared to the placebo. For example, people who took semaglutide could walk about 42 meters further than those on the placebo in a six-minute walking test.

    How could semaglutide reduce knee pain?

    It’s not fully clear how semaglutide helps with knee pain from osteoarthritis. One explanation may be that when a person loses weight, there’s less stress on the joints, which reduces pain.

    But recent studies have also suggested semaglutide and other GLP-1 receptor agonists might have anti-inflammatory properties, and could even protect against cartilage wear and tear.

    While the results of this new study are promising, it’s too soon to regard semaglutide as a “miracle drug” for knee osteoarthritis. And as this study was funded by the drug company that makes semaglutide, it will be important to have independent studies in the future, to confirm the findings, or not.

    The study also had strict criteria, excluding some groups, such as those taking opioids for knee pain. One in seven Australians seeing a GP for their knee osteoarthritis are prescribed opioids. Most participants in the trial were white (61%) and women (82%). This means the study may not fully represent the average person with knee osteoarthritis and obesity.

    It’s also important to consider semaglutide can have a range of side effects, including gastrointestinal symptoms and fatigue.

    There are some concerns that semaglutide could reduce muscle mass and bone density, though we’re still learning more about this.

    Further, it can be difficult to access.

    I have knee osteoarthritis, what should I do?

    Osteoarthritis is a disease caused by multiple factors, and it’s important to take a multifaceted approach to managing it. Weight loss is an important component for those who are overweight or obese, but so are other aspects of self-management. This might include physical activity, pacing strategies, and other positive lifestyle changes such as improving sleep, healthy eating, and so on.

    Giovanni E. Ferreira, NHMRC Emerging Leader Research Fellow, Institute of Musculoskeletal Health, University of Sydney and Christina Abdel Shaheed, Associate Professor, School of Public Health, University of Sydney

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • Measles cases are surging globally. Should children be vaccinated earlier?

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Measles has been rising globally in recent years. There were an estimated 10.3 million cases worldwide in 2023, a 20% increase from 2022.

    Outbreaks are being reported all over the world including in the United States, Europe and the Western Pacific region (which includes Australia). For example, Vietnam has reportedly seen thousands of cases in 2024 and 2025.

    In Australia, 77 cases of measles have been recorded in the first five months of 2025, compared with 57 cases in all of 2024.

    Measles cases in Australia are almost all related to international travel. They occur in travellers returning from overseas, or are contracted locally after mixing with an infected traveller or their contacts.

    Measles most commonly affects children and is preventable with vaccination, given in Australia in two doses at 12 and 18 months old. But in light of current outbreaks globally, is there a case for reviewing the timing of measles vaccinations?

    EyeEm Mobile GmbH/Getty Images

    Some measles basics

    Measles is caused by a virus belonging to the genus Morbillivirus. Symptoms include a fever, cough, runny nose and a rash. While it presents as a mild illness in most cases, measles can lead to severe disease requiring hospitalisation, and even death. Large outbreaks can overwhelm health systems.

    Measles can have serious health consequences, such as in the brain and the immune system, years after the infection.

    Measles spreads from person to person via small respiratory droplets that can remain suspended in the air for two hours. It’s highly contagious – one person with measles can spread the infection to 12–18 people who aren’t immune.

    Because measles is so infectious, the World Health Organization (WHO) recommends two-dose vaccination coverage above 95% to stop the spread and achieve “herd immunity”.

    Low and declining vaccine coverage, especially since the COVID pandemic, is driving global outbreaks.

    When are children vaccinated against measles?

    Newborn babies are generally protected against measles thanks to maternal antibodies. Maternal antibodies get passed from the mother to the baby via the placenta and in breast milk, and provide protection against infections including measles.

    The WHO advises everyone should receive two doses of measles vaccination. In places where there’s a lot of measles circulating, children are generally recommended to have the first dose at around nine months old. This is because it’s expected maternal antibodies would have declined significantly in most infants by that age, leaving them vulnerable to infection.

    If maternal measles antibodies are still present, the vaccine is less likely to produce an immune response.

    Research has also shown a measles vaccine given at less than 8.5 months of age can result in an antibody response which declines more quickly. This might be due to interference with maternal antibodies, but researchers are still trying to understand the reasons for this.

    A second dose of the vaccine is usually given 6–9 months later. A second dose is important because about 10–15% of children don’t develop antibodies after the first vaccine.

    In settings where measles transmission is under better control, a first dose is recommended at 12 months of age. Vaccination at 12 months compared with nine months is considered to generate a stronger, longer-lasting immune response.

    In Australia, children are routinely given the measles-mumps- rubella (MMR) vaccine at 12 months and the measles-mumps-rubella-varicella (MMRV, with “varicella” being chickenpox) vaccine at 18 months.

    Babies at higher risk of catching the disease can also be given an additional early dose. In Australia, this is recommended for infants as young as six months when there’s an outbreak or if they’re travelling overseas to a high-risk setting.

    A new study looking at measles antibodies in babies

    A recent review looked at measles antibody data from babies under nine months old living in low- and middle-income countries. The review combined the results from 20 studies, including more than 8,000 babies. The researchers found that while 81% of newborns had maternal antibodies to measles, only 30% of babies aged four months had maternal antibodies.

    This study suggests maternal antibodies to measles decline much earlier than previously thought. It raises the question of whether the first dose of measles vaccine is given too late to maximise infants’ protection, especially when there’s a lot of measles around.

    Should we bring the measles vaccine forward in Australia?

    All of the data in this study comes from low- and middle-income countries, and might not reflect the situation in Australia where we have much higher vaccine coverage for measles, and very few cases.

    Australia’s coverage for two doses of the MMR vaccine at age two is above 92%.

    Although this is lower than the optimal 95%, the overall risk of measles surging in Australia is relatively low.

    Nonetheless, there may be a case for broadening the age at which an early extra dose of the measles vaccine can be given to children at higher risk. In New Zealand, infants as young as four months can receive a measles vaccine before travelling to an endemic country.

    But the current routine immunisation schedule in Australia is unlikely to change.

    Adding an extra dose to the schedule would be costly and logistically difficult. Lowering the age for the first dose may have some advantages in certain settings, and doesn’t pose any safety concerns, but further evidence would be required to support this change. In particular, research is needed to ensure it wouldn’t negatively affect the longer-term protection that vaccination offers from measles.

    Making sure you’re protected

    In the meantime, ensuring high levels of measles vaccine coverage with two doses is a global priority.

    People born after 1966 are recommended to have two doses of measles vaccine. This is because those born before the mid-1960s likely caught measles as children (when the vaccine was not yet available) and would therefore have natural immunity.

    If you’re unsure about your vaccination status, you can check this through the Australian Immunisation Register. If you don’t have a documented record, ask your doctor for advice.

    Catch-up vaccination is available under the National Immunisation Program.

    Meru Sheel, Associate Professor, Infectious Diseases, Immunisation and Emergencies (IDIE) Group, Sydney School of Public Health, University of Sydney and Anita Heywood, Associate Professor, School of Population Health, UNSW Sydney

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • Food Expiration Dates Don’t Mean What Most People Think They Mean

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    Have you ever wondered why rock salt that formed during the Precambrian era has a label on it saying that it expires next month? To take something more delicate, how about eggs that expire next Thursday; isn’t that oddly specific for something that is surely affected by many variables? What matters, and what doesn’t?

    Covering their assets

    The US in particular wastes huge amounts of food, with 37% of food waste coming from households. Confusion over date labels is a major contributor, accounting for 20% of household food waste. Many people misinterpret these labels, often discarding food that is still safe to eat—which is good for the companies selling the food, because then they get to sell you more.

    Date labels were introduced in the 70s with the “open dating” system to indicate optimal freshness, not safety. These dates are often conservative, set by manufacturers to ensure food is consumed at its best quality and encourage repeat purchases. However, many foods remain safe well past their labeled dates, including shelf-stable items like pasta, rice, and canned goods, as well as frozen foods stored properly.

    Some foods do pose safety risks, especially meat and dairy products, as well as many grain-based foods, all of which which can harbor harmful bacteria. Infant formula labels are strictly regulated for safety. However, most date labels are not linked to health risks, leading to unnecessary waste.

    When it comes down to it, our senses of sight, smell, and taste are more reliable than dates on packaging. Some quick pointers and caveats:

    • If it has changed color in some way that’s not associated with a healthily ripening fruit or vegetable, that’s probably bad
    • If it is moldy, that’s probably bad (but the degree of badness varies from food to food; see the link beneath today’s video for more on that)
    • If a container has developed droplets of water on the inside when it didn’t have those before, that’s probably bad (it means something is respiring, and is thus alive, that probably shouldn’t be)
    • If it smells bad, that’s probably bad—however this is not a good safety test, because a bad smell may often mean you are inhaling mold spores, which are not good for your lungs.
    • If it tastes different than that food usually does, that’s probably bad (especially if it became bitter, pungent, tangy, sour, or cheesy, and does nor normally taste that way).

    Some places have trialled clearer labelling, for example a distinction between “expires” and merely “best before”, but public awareness about the distinction is low. Some places have trialled removing dates entirely, to oblige the consumer to use their own senses instead. This is good for the seller in a different way than household food waste is, because it means the seller will have less in-store waste (because they can still sell something that might previously have been labelled as expired).

    For more on all of this, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like to read:

    Mythbusting Moldy Food

    Take care!

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  • Tuna vs Catfish – Which is Healthier?

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    Our Verdict

    When comparing tuna to catfish, we picked the tuna.

    Why?

    Today in “that which is more expensive and/or harder to get is not necessarily healthier”…

    Looking at their macros, tuna has more protein and less fat (and overall, less saturated fat, and also less cholesterol).

    In the category of vitamins, both are good but tuna distinguishes itself: tuna has more of vitamins A, B1, B2, B3, B6, and D, while catfish has more of vitamins B5, B9, B12, E, and K. They are both approximately equal in choline, and as an extra note in tuna’s favor (already winning 6:5), tuna is a very good source of vitamin D, while catfish barely contains any. All in all: a moderate, but convincing, win for tuna.

    When it comes to minerals, things are clearer still: tuna has more copper, iron, magnesium, phosphorus, potassium, and selenium, while catfish has more calcium, manganese, and zinc. Oh, and catfish is also higher in one other mineral: sodium, which most people in industrialized countries need less of, on average. So, a 6:3 win for tuna, before we even take into account the sodium content (which makes the win for tuna even stronger).

    In short: tuna wins the day in every category!

    Want to learn more?

    You might like to read:

    Farmed Fish vs Wild Caught (It Makes Quite A Difference)

    Take care!

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