
A Better Second Half – by Liz Earle
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Here we have a book that’s aimed at women from midlife onwards. The author is not a doctor or scientist, but has been in the wellness industry in one form or another for pretty much her entire career (and she’s now in her 60s), so you can imagine she’s accumulated a lot of knowledge and experience.
As a result, what she delivers is a guide to aging female health that is comprehensive in its recommendations, while being quite light on the underlying science (which she herself may or may not understand—she neither shows scientific expertise nor scientific ignorance; she simply doesn’t discuss the science beyond the level of mentioning that it is there).
To this end, we get practical advice on supplements, HRT, nutrition, exercise, brain health specifically, sleep, stress-management, social considerations, and even “beauty bio-hacks”.
The style is polished, including in her self-presentation when discussing her own practices and life history. The book is written in British English, by the way, which means not only spellings such as “oestrogen” but also things like her mentioning when she was at her heaviest, post-pregnancy, she weighed 12 stone. This European reviewer had to look up what that is (apparently it’s 76 kg, or 168 lbs, for Americans).
Bottom line: the advice here is good, albeit by no means groundbreaking or deep. If you’re a regular 10almonds reader, you’ll doubtlessly know the main content already, though you’ll probably also pick up at least some new information, since she peppers a great miscellany of facts throughout her work. Which, given the very minimal referencing, may then require fact-checking by the reader, but either way, learning occurs. Which is what we’re all about!
Click here to check out A Better Second Half, and enjoy a better second half!
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What is a blood cholesterol ratio? And what should yours be?
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Have you had a blood test to check your cholesterol level? These check the different blood fat components:
- total cholesterol
- LDL (low-density lipoprotein), which is sometimes called “bad cholesterol”
- HDL (high-density lipoprotein), which is sometimes called “good cholesterol”
- triglycerides.
Your clinician then compares your test results to normal ranges – and may use ratios to compare different types of cholesterol.
High blood cholesterol is a major risk factor for cardiovascular disease. This is a broad term that includes disease of blood vessels throughout the body, arteries in the heart (known as coronary heart disease), heart failure, heart valve conditions, arrhythmia and stroke.
So what does cholesterol do? And what does it mean to have a healthy cholesterol ratio?
Shutterstock What are blood fats?
Cholesterol is a waxy type of fat made in the liver and gut, with a small amount of pre-formed cholesterol coming from food.
Cholesterol is found in all cell membranes, contributing to their structure and function. Your body uses cholesterol to make vitamin D, bile acid, and hormones, including oestrogen, testosterone, cortisol and aldosterone.
When there is too much cholesterol in your blood, it gets deposited into artery walls, making them hard and narrow. This process is called atherosclerosis.
High blood cholesterol is a major risk factor for cardiovascular disease. Halfpoint/Shutterstock Cholesterol is packaged with triglycerides (the most common type of fat in the body) and specific “apo” proteins into “lipo-proteins” as a package called “very-low-density” lipoproteins (VLDLs).
These are transported via the blood to body tissue in a form called low-density lipoprotein (LDL) cholesterol.
Excess cholesterol can be transported back to the liver by high-density lipoprotein, the HDL, for removal from circulation.
Another less talked about blood fat is Lipoprotein-a, or Lp(a). This is determined by your genetics and not influenced by lifestyle factors. About one in five (20%) of Australians are carriers.
Having a high Lp(a) level is an independent cardiovascular disease risk factor.
Knowing your numbers
Your blood fat levels are affected by both modifiable factors:
- dietary intake
- physical activity
- alcohol
- smoking
- weight status.
And non-modifiable factors:
- age
- sex
- family history.
What are cholesterol ratios?
Cholesterol ratios are sometimes used to provide more detail on the balance between different types of blood fats and to evaluate risk of developing heart disease.
Commonly used ratios include:
1. Total cholesterol to HDL ratio
This ratio is used in Australia to assess risk of heart disease. It’s calculated by dividing your total cholesterol number by your HDL (good) cholesterol number.
A higher ratio (greater than 5) is associated with a higher risk of heart disease, whereas a lower ratio is associated with a lower risk of heart disease.
A study of 32,000 Americans over eight years found adults who had either very high, or very low, total cholesterol/HDL ratios were at 26% and 18% greater risk of death from any cause during the study period.
Those with a ratio of greater than 4.2 had a 13% higher risk of death from heart disease than those with a ratio lower than 4.2.
2. Non-HDL cholesterol to HDL cholesterol ratio (NHHR)
Non-HDL cholesterol is the total cholesterol minus HDL. Non-HDL cholesterol includes all blood fats such as LDL, triglycerides, Lp(a) and others. This ratio is abbreviated as NHHR.
This ratio has been used more recently because it compares the ratio of “bad” blood fats that can contribute to atherosclerosis (hardening and narrowing of the arteries) to “good” or anti-atherogenic blood fats (HDL).
Non-HDL cholesterol is a stronger predictor of cardiovascular disease risk than LDL alone, while HDL is associated with lower cardiovascular disease risk.
Because this ratio removes the “good” cholesterol from the non-HDL part of the ratio, it is not penalising those people who have really high amounts of “good” HDL that make up their total cholesterol, which the first ratio does.
Research has suggested this ratio may be a stronger predictor of atherosclerosis in women than men, however more research is needed.
Another study followed more than 10,000 adults with type 2 diabetes from the United States and Canada for about five years. The researchers found that for each unit increase in the ratio, there was around a 12% increased risk of having a heart attack, stroke or death.
They identified a risk threshold of 6.28 or above, after adjusting for other risk factors. Anyone with a ratio greater than this is at very high risk and would require management to lower their risk of heart disease.
The greater this ratio, the greater the chance of having a heart attack or stroke. Alex Yeung/Shutterstock 3. LDL-to-HDL cholesterol ratio
LDL/HDL is calculated by dividing your LDL cholesterol number by the HDL number. This gives a ratio of “bad” to “good” cholesterol.
A lower ratio (ideal is less than 2.0) is associated with a lower risk of heart disease.
While there is lesser focus on LDL/HDL, these ratios have been shown to be predictors of occurrence and severity of heart attacks in patients presenting with chest pain.
If you’re worried about your cholesterol levels or cardiovascular disease risk factors and are aged 45 and over (or over 30 for First Nations people), consider seeing your GP for a Medicare-rebated Heart Health Check.
Clare Collins, Laureate Professor in Nutrition and Dietetics, University of Newcastle and Erin Clarke, Postdoctoral Researcher, Nutrition and Dietetics, University of Newcastle
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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I have a bit of a cold. Am I sick enough to take a day off work?
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Whether it’s your first or fourth cold of the season, many Australians are waking up at the moment with a sniffle, a sore throat or feeling more tired than usual.
June to August is peak flu season in Australia. There are also high rates of COVID circulating, along with other respiratory viruses such as respiratory syncytial virus (RSV) and adenovirus.
Sometimes it’s clear when you need to spend the day in bed: you have a fever, aches and pains, and can’t think clearly. If it’s the flu or COVID, you’ll want to stay away from others, and to rest and recover.
But what about if your symptoms are mild? Are you sick enough to take the day off, or should you push through it? And what if you feel pressured to work?
Here’s what to consider.
Are you likely to spread it?
While it may seem like a good idea to continue working, especially when your symptoms are mild, going to work when infectious with a respiratory virus risks infecting your co-workers.
If you are in a client-facing role, such as a teacher or a salesperson, you may also infect others like students or customers.
The risks may be even greater for those working with vulnerable communities, such as in aged care work, where the consequences can be severe.
From an organisational perspective, you are likely less productive when you are not feeling well.
So, whenever possible, avoid going into work when you’re feeling unwell.
Should I work from home?
The COVID pandemic normalised working from home. Since then, more people work from home when they’re unwell, rather than taking sick leave.
Some employees join Zoom or Teams meetings out of guilt, not wanting to let their co-workers down. Others – and in particular, some men – feel the need to maintain their performance at work, even if it’s at the expense of their health.
A downside of powering through is that workers may prolong their illness by not looking after themselves.
Can you take leave when you need it?
Employees in Australia can take either paid or unpaid time off when they are unwell.
Most full-time employees get ten days of paid sick leave per year, while part-time employees get the equivalent pro-rata.
Employers can ask for reasonable evidence from employees to show they are unwell, such as asking for a medical certificate from a pharmacy or GP, or a statutory declaration. The type of evidence required may differ from organisation to organisation, with some awards and enterprise agreements specifying the type of evidence needed.
While taking a sick day helps many workers recuperate, a significant proportion of workers engaged in non-standard work arrangements do not receive these benefits. There are, for example, 2.6 million casual employees who don’t have access to paid sick leave.
Similarly, most self-employed people such as tradies and gig workers do not have any paid leave entitlements. Although these workers can still take unpaid leave, they are sacrificing income when they call in sick.
Research from the Australian Council of Trade Unions has found more than half of insecure workers don’t take time off when injured or sick.
So a significant proportion of workers in Australia simply cannot afford to call in sick.
Why pushing through isn’t the answer
“Presenteeism” is the phenomenon of people reporting for work even when they are unwell or not fully functioning, affecting their health and productivity.
While exact figures are hard to determine, since most organisations don’t systematically track it, estimates suggest 30%–90% of employees work while sick at least once a year.
People work while sick for different reasons. Some choose to because they love their job or enjoy the social side of work – this is called voluntary presenteeism.
But many don’t have a real choice, facing financial pressure or job insecurity. That’s involuntary presenteeism, and it’s a much bigger problem.
Research has found industry norms may be shaping the prevalence of “involuntary presenteeism”, with workers in the health and education sectors more likely to feel obligated to work when sick due to “at work” caring responsibilities.
What can organisations do about it?
Leaders set the tone, especially around health and wellbeing. When they role-model healthy behaviour and support time off, it gives others permission to do the same.
Supportive leaders can help reduce presenteeism, while pressure from demanding leaders can make it worse.
Your co-workers matter too. When teams step up and share the load, it creates a culture where people feel safe to take leave. A supportive environment makes wellbeing a shared responsibility.
But for some workers, leave isn’t an option. Fixing this requires policy change across industries and society more broadly, not just inside the workplace.
Alex Veen, Senior Lecturer and University of Sydney Business School Emerging Scholar Research Fellow, University of Sydney; Hannah Kunst, Lecturer in Leadership, University of Sydney, and Nate Zettna, Lecturer in Leadership, University of Sydney
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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The All-New Pain Therapy That “Switches Off Pain” Without Addiction
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When it comes to painkilling medications, they can generally be categorized into two broad kinds:
- non-opioids (e.g. ibuprofen, paracetamol/acetaminophen, aspirin)
- ones that actually work for something more serious than a headache
That’s an oversimplification of course, but as a strong general rule, when there is serious painkilling to be done, that’s when doctors consider it’s time to break out the opioids.
Nor are all opioids created equal—there’s a noteworthy difference between codeine and morphine, for instance—but the problems of opioids are typically the same (tolerance, addiction, and eventual likelihood of overdose when one tries to take enough to make it work after developing a tolerance), and it becomes simply a matter of degree.
See also: I’ve been given opioids after surgery to take at home. What do I need to know?
But what if we’d like those benefits, without the tolerance, ease of addiction, and possibility of overdose?
The genetic advantage
Researchers (Dr. Corinna Oswell et al.) developed a targeted gene therapy that reduces pain by acting directly on specific brain circuits instead of broadly affecting the entire brain like opioids do.
In other words: this gene therapy reproduces pain relief without activating addiction-related brain pathways or dulling normal sensations.
Specifically, the therapy makes use of a brain-specific “off switch”, which dampens pain signals in the anterior cingulate cortex (a key brain area involved in the experience of pain). This way, it can target opioid-sensitive neurons and alter their activity, effectively recreating the beneficial effects of opioids at a circuit level, without having the usual adverse effects associated with opioids on the systemic level.
If you’re wondering how soon you can get it, that bad news is that the therapy is still in preclinical research, and further studies are needed before human clinical trials can confirm safety and effectiveness.
But it’s very promising so far; in animal models, the treatment provided sustained pain relief and reversed abnormal brain activity linked to chronic pain without impairing reflexes or sensory detection.
You can find the paper itself, here: Mimicking opioid analgesia in cortical pain circuits
So, how rapidly is science advancing? Well, this research comes a little more than a year after, a different team of scientists were pioneering a potential gene therapy for pain, with results that were very promising at the time, but not a patch on what we’ve been talking about today:
Structure-guided design of a peripherally restricted chemogenetic system
…which you can read about in pop-science terms (with diagrams!) here:
New gene therapy could alleviate chronic pain, researchers find
So… Pretty quickly, but we always love to see new advances!
Want to learn more?
If you’re looking for alternatives while you wait, we’ve written quite a bit about pain management, including:
- Before You Reach For That Tylenol…
- How To Stop Pain Spreading
- How To Dial Down Your Pain
- Managing Chronic Pain (Realistically!)
- Get The Right Help For Your Pain
- The 7 Approaches To Pain Management
- Science-Based Alternative Pain Relief (When Painkillers Aren’t Helping, These Things Might)
Take care!
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How Hitting “Snooze” Actually Steals Your Sleep
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There’s a common myth that older people require less sleep; the reality is that sleeping less and not dying of it does not equate to needing less.
See also: Sleep: Yes, You Really Do Still Need It! and How Sleep-Deprived Are You, Really?
Quantity is not everything though; quality absolutely matters too: The 6 Dimensions Of Sleep (And Why They Matter) ← duration is just one dimension out of the six!
Harvard declares: you snooze, you lose
Dr. Rebecca Robbins et al. did a huge study using data from the SleepCycle smartphone app, analyzing over 3 million sleep sessions from 21,222 users across four continents—of which, most participants lived in the US (nearly half), followed by the UK, Japan, Germany, and Australia (then other countries; the distribution was simply a matter of where the app is popular, and was not part of the study design).
So, firstly, how much did people use the snooze button? 55.6% of sleep sessions involved a snooze alarm. 45% of users were heavy snoozers, 28% moderate, 27% light.
❝Overall, users pressed the snooze alarm approximately 2.4 times daily and spent on average 10.8 minutes snoozing.
This is equivalent to a monthly loss of nearly one 6-hour night of sleep.
Heavy users chose the snooze alarm approximately 4 times daily, resulting in about 20 minutes of snoozing duration. On the other hand, light users chose the snooze alarm on average 1.2 times a day, resulting in 3 minutes of snoozing duration.❞
About that average user’s loss of the equivalent of nearly a night of sleep per month: What Harm Can One Sleepless Night Do?
You may be wondering: how did adding an extra 10.8 minutes (or 20, or 3) on average result in lost sleep, rather than more?
It comes down to sleep cycles, of which the shortest healthy sleep cycle is 20 minutes. Anything shorter than that, and it’ll be a broken cycle, and you’ll (unless in cases of very extreme sleep deprivation) be better off not having taken that at all.
This same principle goes for napping, by the way, though there’s more to it than that, sufficient that we wrote a whole main feature about this: How To Nap Like A Pro (No More “Sleep Hangovers”!)
Consequently, the time spent hitting the snooze button and then not getting a full sleep cycle in, was for functional purposes in terms of brain health time lost.
Furthermore, the researchers noted that frequent snooze-button use was linked to erratic sleep patterns, short and poor-quality sleep, and resultant sleep problems such as hypersomnia / compensatory sleep behavior.
See also: How Regularity Of Sleep Can Be Even More Important Than Duration ← A recent, large (n=72,269) 8-year observational study of adults aged 40-79 found a strong association between irregular sleep and major cardiovascular events, to such an extent that it was worse than undersleeping.
Back to the Harvard study: the paper itself, which we’ll link below, has a lot of data, including a lot of demographic information we didn’t include her for brevity. For example,
❝In addition, we observed that women’s snooze duration was slightly longer than men. It is possible the gender difference observed in snooze alarm behavior stems from the increased risk for insomnia among women as compared to men20. In addition, women shoulder a greater burden of childcare duties compared to men21, which may be on top of professional or other duties, therefore reducing the time available to women for sleep and increasing risk for sleep difficulties22, which may increase reliance upon snooze alarm.❞
Read the study paper in full: Snooze alarm use in a global population of smartphone users
What to do about this?
Well, “don’t use the snooze button” is a great start.
But also: Calculate (And Enjoy) The Perfect Night’s Sleep ← this is about getting all your sleep cycles right, not just the last one of the night!
And finally: Get Better Sleep: Beyond The Basics
Sweet dreams!
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Age & Strength Loss: What Happens When, & How Much Is Unavoidable?
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When it comes to aging and loss of strength, a lot of focus is placed on loss of muscle mass (sarcopenia).
We talked about this in our article: Protein vs Sarcopenia: How Much Do We Need, Really?
And that is important, but it’s not the whole story!
Strong at every age
You can be strong at every age, if and only if you’re very intentional about it.
Researchers (Dr. Maria Westerståhl et al.) followed 427 people for 47 years, repeatedly measuring fitness, strength, muscle endurance, and power from adolescence all the way through into older adulthood.
First, the bad news: physical performance overall peaks in early adulthood and begins declining at around 26 for women and 36 for men, with initially gradual losses that accelerate with advancing age.
About that acceleration: aerobic capacity and muscular endurance initially fall by about 0.3–0.6 percent per year, later speeding up to roughly 2.0–2.5 percent per year, and the deterioration in muscle power gets a similar age-related acceleration.
Next, the worse news: physical power specifically starts its decline even sooner than the other factors, with women having their peak around 19 and men having their peak around 27.
It does, however, get worse: total losses in physical capacity from peak to age 63 range from 30–48%, which latter end of the range is quite a dramatic loss of physical capacity indeed. Note that that’s the aggregate figure, so we’re not just talking about strength here.
Is there any good news? Yes: it’s never too late! People who became physically active in adulthood improved physical capacity by about 5–10%, showing that starting later still provides meaningful benefits. To be clear, that’s a net improvement of 5–10%, we’re not talking about shaving 5–10% off the 30–48% loss.
If you want to go through all these numbers (and more) in detail, here’s the paper: Rise and Fall of Physical Capacity in a General Population: A 47-Year Longitudinal Study
As for what this means in realistic terms: you’re probably not only not as strong as you used to be, but also not as fit, fast, mobile, and so forth. Your power (explosive power, like sprints or best-effort lifts) and endurance (like long-distance cardio, or isometric holds) are probably not what they used to be either.
- On the one hand, you can improve them.
- On the other hand, you do have to actually do it—merely knowing about it will not help if you don’t take action!
So, how to do that?
Read on…
Want to learn more?
Here are some very good starting points:
- Resistance Is Useful! (Especially As We Get Older)
- Overdone It? How To Speed Up Recovery After Exercise
- How To Do HIIT (Without Wrecking Your Body)
- HIIT, But Make It HIRT ← this is about high-intensity resistance training (HIRT); confusing the muscles like one confuses the heart in HIIT, which thus yields improved results
- Exercises To Do (And Ones To Avoid) If You Have Osteoporosis ← an important consideration for many
And if you’re really serious about it, then for a much deeper dive than we have room for here, we highly recommend this excellent book we reviewed a while back:
Unbreakable: A Woman’s Guide to Aging with Power – by Dr. Vonda Wright ← So, she wants us to avoid the train of sarcopenia → osteopenia → osteoporosis → fractures → infections → death, by reducing our risk factors early, and staying more robust and biologically younger.
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Yes, adults can develop food allergies. Here are 4 types you need to know about
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If you didn’t have food allergies as a child, is it possible to develop them as an adult? The short answer is yes. But the reasons why are much more complicated.
Preschoolers are about four times more likely to have a food allergy than adults and are more likely to grow out of it as they get older.
It’s hard to get accurate figures on adult food allergy prevalence. The Australian National Allergy Council reports one in 50 adults have food allergies. But a US survey suggested as many as one in ten adults were allergic to at least one food, with some developing allergies in adulthood.
What is a food allergy
Food allergies are immune reactions involving immunoglobulin E (IgE) – an antibody that’s central to triggering allergic responses. These are known as “IgE-mediated food allergies”.
Food allergy symptoms that are not mediated by IgE are usually delayed reactions and called food intolerances or hypersensitivity.
Food allergy symptoms can include hives, swelling, difficulty swallowing, vomiting, throat or chest tightening, trouble breathing, chest pain, rapid heart rate, dizziness, low blood pressure or anaphylaxis.
Symptoms include hives. wisely/Shutterstock IgE-mediated food allergies can be life threatening, so all adults need an action management plan developed in consultation with their medical team.
Here are four IgE-mediated food allergies that can occur in adults – from relatively common ones to rare allergies you’ve probably never heard of.
1. Single food allergies
The most common IgE-mediated food allergies in adults in a US survey were to:
- shellfish (2.9%)
- cow’s milk (1.9%)
- peanut (1.8%)
- tree nuts (1.2%)
- fin fish (0.9%) like barramundi, snapper, salmon, cod and perch.
In these adults, about 45% reported reacting to multiple foods.
This compares to most common childhood food allergies: cow’s milk, egg, peanut and soy.
Overall, adult food allergy prevalence appears to be increasing. Compared to older surveys published in 2003 and 2004, peanut allergy prevalence has increased about three-fold (from 0.6%), while tree nuts and fin fish roughly doubled (from 0.5% each), with shellfish similar (2.5%).
While new adult-onset food allergies are increasing, childhood-onset food allergies are also more likely to be retained into adulthood. Possible reasons for both include low vitamin D status, lack of immune system challenges due to being overly “clean”, heightened sensitisation due to allergen avoidance, and more frequent antibiotic use.
Some adults develop allergies to cow’s milk, while others retain their allergy from childhood. Sarah Swinton/Unsplash 2. Tick-meat allergy
Tick-meat allergy, also called α-Gal syndrome or mammalian meat allergy, is an allergic reaction to galactose-alpha-1,3-galactose, or α-Gal for short.
Australian immunologists first reported links between α-Gal syndrome and tick bites in 2009, with cases also reported in the United States, Japan, Europe and South Africa. The US Centers for Disease Control estimates about 450,000 Americans could be affected.
The α-Gal contains a carbohydrate molecule that is bound to a protein molecule in mammals.
The IgE-mediated allergy is triggered after repeated bites from ticks or chigger mites that have bitten those mammals. When tick saliva crosses into your body through the bite, antibodies to α-Gal are produced.
When you subsequently eat foods that contain α-Gal, the allergy is triggered. These triggering foods include meat (lamb, beef, pork, rabbit, kangaroo), dairy products (yoghurt, cheese, ice-cream, cream), animal-origin gelatin added to gummy foods (jelly, lollies, marshmallow), prescription medications and over-the counter supplements containing gelatin (some antibiotics, vitamins and other supplements).
Tick-meat allergy reactions can be hard to recognise because they’re usually delayed, and they can be severe and include anaphylaxis. Allergy organisations produce management guidelines, so always discuss management with your doctor.
3. Fruit-pollen allergy
Fruit-pollen allergy, called pollen food allergy syndrome, is an IgE-mediated allergic reaction.
In susceptible adults, pollen in the air provokes the production of IgE antibodies to antigens in the pollen, but these antigens are similar to ones found in some fruits, vegetables and herbs. The problem is that eating those plants triggers an allergic reaction.
The most allergenic tree pollens are from birch, cypress, Japanese cedar, latex, grass, and ragweed. Their pollen can cross-react with fruit and vegetables, including kiwi, banana, mango, avocado, grapes, celery, carrot and potato, and some herbs such as caraway, coriander, fennel, pepper and paprika.
Fruit-pollen allergy is not common. Prevalence estimates are between 0.03% and 8% depending on the country, but it can be life-threatening. Reactions range from itching or tingling of lips, mouth, tongue and throat, called oral allergy syndrome, to mild hives, to anaphylaxis.
4. Food-dependent, exercise-induced food allergy
During heavy exercise, the stomach produces less acid than usual and gut permeability increases, meaning that small molecules in your gut are more likely to escape across the membrane into your blood. These include food molecules that trigger an IgE reaction.
If the person already has IgE antibodies to the foods eaten before exercise, then the risk of triggering food allergy reactions is increased. This allergy is called food-dependent exercise-induced allergy, with symptoms ranging from hives and swelling, to difficulty breathing and anaphylaxis.
This type of allergy is extremely rare. Ben O’Sullivan/Unsplash Common trigger foods include wheat, seafood, meat, poultry, egg, milk, nuts, grapes, celery and other foods, which could have been eaten many hours before exercising.
To complicate things even further, allergic reactions can occur at lower levels of trigger-food exposure, and be more severe if the person is simultaneously taking non-steroidal inflammatory medications like aspirin, drinking alcohol or is sleep-deprived.
Food-dependent exercise-induced allergy is extremely rare. Surveys have estimated prevalence as between one to 17 cases per 1,000 people worldwide with the highest prevalence between the teenage years to age 35. Those affected often have other allergic conditions such as hay fever, asthma, allergic conjunctivitis and dermatitis.
Allergies are a growing burden
The burden on physical health, psychological health and health costs due to food allergy is increasing. In the US, this financial burden was estimated as $24 billion per year.
Adult food allergy needs to be taken seriously and those with severe symptoms should wear a medical information bracelet or chain and carry an adrenaline auto-injector pen. Concerningly, surveys suggest only about one in four adults with food allergy have an adrenaline pen.
If you have an IgE-mediated food allergy, discuss your management plan with your doctor. You can also find more information at Allergy and Anaphylaxis Australia.
Clare Collins, Laureate Professor in Nutrition and Dietetics, University of Newcastle
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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