
PFAS Exposure & Cancer: The Numbers Are High
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PFAS & Cancer Risk: The Numbers Are High

This is Dr. Maaike van Gerwen. Is that an MD or a PhD, you wonder? It’s both.
She’s also Director of Research in the Department of Otolaryngology at Mount Sinai Hospital in New York, Scientific Director of the Program of Personalized Management of Thyroid Disease, and Member of the Institute for Translational Epidemiology and the Transdisciplinary Center on Early Environmental Exposures.
What does she want us to know?
She’d love for us to know about her latest research published literally today, about the risks associated with PFAS, such as the kind widely found in non-stick cookware:
Per- and polyfluoroalkyl substances (PFAS) exposure and thyroid cancer risk
Dr. van Gerwen and her team tested this several ways, and the very short and simple version of the findings is that per doubling of exposure, there was a 56% increased rate of thyroid cancer diagnosis.
(The rate of exposure was not just guessed based on self-reports; it was measured directly from PFAS levels in the blood of participants)
- PFAS exposure can come from many sources, not just non-stick cookware, but that’s a “biggie” since it transfers directly into food that we consume.
- Same goes for widely-available microwaveable plastic food containers.
- Relatively less dangerous exposures include waterproofed clothing.
To keep it simple and look at the non-stick pans and microwavable plastic containers, doubling exposure might mean using such things every day vs every second day.
Practical take-away: PFAS may be impossible to avoid completely, but even just cutting down on the use of such products is already reducing your cancer risk.
Isn’t it too late, by this point in life? Aren’t they “forever chemicals”?
They’re not truly “forever”, but they do have long half-lives, yes.
See: Can we take the “forever” out of forever chemicals?
The half-lives of PFOS and PFOA in water are 41 years and 92 years, respectively.
In the body, however, because our body is constantly trying to repair itself and eliminate toxins, it’s more like 3–7 years.
That might seem like a long time, and perhaps it is, but the time will pass anyway, so might as well get started now, rather than in 3–7 years time!
Read more: National Academies Report Calls for Testing People With High Exposure to “Forever Chemicals”
What should we use instead?
In place of non-stick cookware, cast iron is fantastic. It’s not everyone’s preference, though, so you might also like to know that ceramic cookware is a fine option that’s functionally non-stick but without needing a non-stick coating. Check for PFAS-free status; they should advertise this.
In place of plastic microwaveable containers, Pyrex (or equivalent) glass dishes (you can get them with lids) are a top-tier option. Ceramic containers (without metallic bits!) are also safely microwaveable.
See also:
Here’s a List of Products with PFAS (& How to Avoid Them)
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Falling vaccination rates put children at risk of preventable diseases. Governments need a new strategy to boost uptake
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Child vaccination is one of the most cost-effective health interventions. It accounts for 40% of the global reduction in infant deaths since 1974 and has led to big health gains in Australia over the past two decades.
Australia has been a vaccination success story. Ten years after we begun mass vaccination against polio in 1956, it was virtually eliminated. Our child vaccination rates have been among the best in the world.
But after peaking in 2020, child vaccination in Australia is falling. Governments need to implement a comprehensive strategy to boost vaccine uptake, or risk exposing more children to potentially preventable infectious diseases.
Yuri A/Shutterstock Child vaccination has been a triumph
Thirty years ago, Australia’s childhood vaccination rates were dismal. Then, in 1997, governments introduced the National Immunisation Program to vaccinate children against diseases such as diphtheria, tetanus, and measles.
Measures to increase coverage included financial incentives for parents and doctors, a public awareness campaign, and collecting and sharing local data to encourage the least-vaccinated regions to catch up with the rest of the country.
What followed was a public health triumph. In 1995, only 52% of one-year-olds were fully immunised. By 2020, Australia had reached 95% coverage for one-year-olds and five-year-olds. At this level, it’s difficult even for highly infectious diseases, such as measles, to spread in the community, protecting both the vaccinated and unvaccinated.
By 2020, 95% of children were vaccinated. Drazen Zigic/Shutterstock Gaps between regions and communities closed too. In 1999, the Northern Territory’s vaccination rate for one-year-olds was the lowest in the country, lagging the national average by six percentage points. By 2020, that gap had virtually disappeared.
The difference between vaccination rates for First Nations children and other children also narrowed considerably.
It made children healthier. The years of healthy life lost due to vaccine-preventable diseases for children aged four and younger fell by nearly 40% in the decade to 2015.
Some diseases have even been eliminated in Australia.
Our success is slipping away
But that success is at risk. Since 2020, the share of children who are fully vaccinated has fallen every year. For every child vaccine on the National Immunisation Schedule, protection was lower in 2024 than in 2020.
Gaps between parts of Australia are opening back up. Vaccination rates in the highest-coverage parts of Australia are largely stable, but they are falling quickly in areas with lower vaccination.
In 2018, there were only ten communities where more than 10% of one-year-old children were not fully vaccinated. Last year, that number ballooned to 50 communities. That leaves more areas vulnerable to disease and outbreaks.
While Noosa, the Gold Coast Hinterland and Richmond Valley (near Byron Bay) have persistently had some of the country’s lowest vaccination rates, areas such as Manjimup in Western Australia and Tasmania’s South East Coast have recorded big declines since 2018.
Missing out on vaccination isn’t just a problem for children.
One preprint study (which is yet to be peer-reviewed) suggests vaccination during pregnancy may also be declining.
Far too many older Australians are missing out on recommended vaccinations for flu, COVID, pneumococcal and shingles. Vaccination rates in aged care homes for flu and COVID are worryingly low.
What’s going wrong?
Australia isn’t alone. Since the pandemic, child vaccination rates have fallen in many high-income countries, including New Zealand, the United Kingdom and the United States.
Globally, in 2023, measles cases rose by 20%, and just this year, a measles outbreak in rural Texas has put at least 13 children in hospital.
Alarmingly, some regions in Australia have lower measles vaccination than that Texas county.
The timing of trends here and overseas suggests things shifted, or at least accelerated, during the pandemic. Vaccine hesitancy, fuelled by misinformation about COVID vaccines, is a growing threat.
This year, vaccine sceptic Robert F. Kennedy Jr was appointed to run the US health system, and Louisiana’s top health official has reportedly cancelled the promotion of mass vaccination.
In Australia, a recent survey found 6% of parents didn’t think vaccines were safe, and 5% believed they don’t work.
Those concerns are far more common among parents with children who are partially vaccinated or unvaccinated. Among the 2% of parents whose children are unvaccinated, almost half believe vaccines are not safe for their child, and four in ten believe vaccines didn’t work.
Other consequences of the pandemic were a spike in the cost of living, and a health system struggling to meet demand. More than one in ten parents said cost and difficulty getting an appointment were barriers to vaccinating their children.
There’s no single cause of sliding vaccination rates, so there’s no one solution. The best way to reverse these worrying trends is to work on all the key barriers at once – from a lack of awareness, to inconvenience, to lack of trust.
What governments should do
Governments should step up public health campaigns that counter misinformation, boost awareness of immunisation and its benefits, and communicate effectively to low-vaccination groups. The new Australian Centre for Disease Control should lead the charge.
Primary health networks, the regional bodies responsible for improving primary care, should share data on vaccination rates with GPs and pharmacies. These networks should also help make services more accessible to communities who are missing out, such as migrant groups and disadvantaged families.
State and local governments should do the same, sharing data and providing support to make maternal child health services and school-based vaccination programs accessible for all families.
Governments can communicate better about the benefits of vaccination. Yuri A/Shutterstock Governments should also be more ambitious about tackling the growing vaccine divides between different parts of the country. The relevant performance measure in the national vaccination agreement is weak. States must only increase five-year-old vaccination rates in four of the ten areas where it is lowest. That only covers a small fraction of low-vaccination areas, and only the final stage of child vaccination.
Australia needs to set tougher goals, and back them with funding.
Governments should fund tailored interventions in areas with the lowest rates of vaccination. Proven initiatives include training trusted community members as “community champions” to promote vaccinations, and pop-up clinics or home visits for free vaccinations.
At this time of year, childcare centres and schools are back in full swing. But every year, each new intake has less protection than the previous cohort. Governments are developing a new national vaccination strategy and must seize the opportunity to turn that trend around. If it commits to a bold national plan, Australia can get back to setting records for child vaccination.
Peter Breadon, Program Director, Health and Aged Care, Grattan Institute and Wendy Hu, Associate, Health Program, Grattan Institute
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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Eat To Beat Chronic Fatigue!
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How To Eat To Beat Chronic Fatigue
Chronic fatigue is on the rise, and it can make life a living Hell. Days blur into one, and you try to take each day as it comes, but sometimes several days gang up on you at once.
You probably know some lifestyle changes that might help—if only you had the energy to implement them.
You’d like to eat well, but you need to…
- Buy the fresh produce (and take a little rest after)
- Put the groceries away (and take a little rest after)
- Wash the vegetables (and take a little rest after)
- Chop the things as necessary (and take a little rest after)
- Cook dinner (and take a little rest after)
…and now you’re too exhausted to eat it.
So, what can be done?
First, avoid things that cause inflammation, as this is a major contributor to chronic fatigue. You might like our previous main feature:
Next up, really do stay hydrated. It’s less about quantity, and more about ubiquity. Hydrate often.
Best is if you always have some (hydrating) drink on the go.
Do experiment with your diet, and/but keep a food journal of what you eat and how you feel 30–60 minutes after eating it. Only make one change at a time, otherwise you won’t know which change made the difference.
Notice what patterns emerge over time, and adjust your ingredients accordingly.
Limit your caffeine intake. We know that sometimes it seems like the only way to get through the day, but you will always crash later, because it was only ever taxing your adrenal system (thus: making you more tired in the long run) and pulling the wool over the eyes of your adenosine receptors (blocking you from feeling how tired you are, but not actually reducing your body’s tiredness).
Put simply, caffeine is the “payday loan” of energy.
Eat more non-starchy vegetables, and enjoy healthy fats. Those healthy fats can come from nuts and seeds, avocado, or fish (not fried, though!).
The non-starchy vegetables will boost your vitamins and fiber while being easy on your beleaguered metabolism, while the healthy fats will perk up your energy levels without spiking insulin like sugars would.
Pay the fatigue tax up front. What this means is… Instead of throwing away vegetables that didn’t get used because it would take too much effort and you just need an easier dinner today, buy ready-chopped vegetables, for example.
And if you buy vegetables frozen, they’re also often not only cheaper, but also (counterintuitively) contain more nutrients.
A note of distinction:
Many more people have chronic fatigue (the symptom: being exhausted all the time) than have chronic fatigue syndrome (the illness: myalgic encephalomyelitis).
This is because fatigue can be a symptom of many, many other conditions, and can be heavily influenced by lifestyle factors too.
A lot of the advice for dealing with chronic fatigue is often the same in both cases, but some will be different, because for example:
- If your fatigue is from some other condition, that condition probably impacts what lifestyle factors you are (and are not) able to change, too
- If your fatigue is from lifestyle factors, that hopefully means you can change those and enjoy less fatigue…
- But if it’s not from lifestyle factors, as in ME/CFS, then advice to “exercise more” etc is not going to help so much.
There are ways to know the difference though:
Check out: Do You Have Chronic Fatigue Syndrome?
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Artichoke vs Onion – Which is Healthier?
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Our Verdict
When comparing artichoke to onion, we picked the artichoke.
Why?
It wasn’t close:
In terms of macros, artichoke has more than 3x the fiber, more than 3x the protein, and about the same carbs. An easy first-round win for artichoke.
In the category of vitamins, artichoke has more of vitamins A, B1, B2, B3, B5, B7. B9, C, E, and K, while onions are not higher in any vitamins, though they do at least equal artichoke for vitamin B6. In any case, an overwhelming win for artichokes in this round.
Looking at minerals next, artichokes have more calcium, copper, iron, magnesium, manganese, phosphorus, potassium, and zinc, while onions have more selenium, yielding an 8:1 victory to artichokes here.
In other considerations, artichokes are much higher in polyphenols, so that’s another point in their favor.
Adding up the sections makes for a very clear overall win for artichoke, but by all means enjoy either or both, as diversity is best!
Want to learn more?
You might like:
What’s Your Plant Diversity Score?
Enjoy!
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Thinking, Fast and Slow – by Dr. Daniel Kahneman
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We all try to make the best decisions we can with the information available… Don’t we?
Yet, somehow, a survival chance of 90% seems better than a mortality rate of 10%, and as it turns out, we as fallible humans are prey to all manner of dubious heuristics.
Nobel Prize winner Dr. Daniel Kahneman lays out for us two sytems of thought process:
- Fast, intuitive, emotional
- Slow, deliberate, logical
He makes the case for how and why we do need both, but often end up using the wrong one. He notes how the first is required for efficiency, or we would spend all day deciding what socks to wear… The second, meanwhile, is required for high-stakes decisions, but is lazy by nature, and often we don’t engage it when we ought to.
Over the course of many diverse examples, Dr. Kahneman shows how again and again, the second system is slowly cogitating at the back of the class, while the first system is bouncing up and down with its hand in the air saying “I know! I know!”, even when, in fact, it does not know.
For a book largely founded in economics (it’s a massive takedown of the notion of the rational consumer), it is not at all dry, and is very readable in style. It’s engaging throughout, and readers far removed from Wall Street will find plenty of ways it relates to our everyday lives.
Bottom line: if you’d like to avoid making many mistakes in what you’d assumed to be rational decisions, this book is critical reading.
Click here to check out “Thinking, Fast And Slow”, and enjoy the results of better decisions!
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Focusing on how and why you eat, not just what, may be the key to healthy eating
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When most people think about “healthy eating”, they usually focus on what they eat. That might mean trying to eat more fruit and vegetables or less fast food, or counting calories.
But there’s a lot more to healthy eating than just dietary intake. Behaviours and attitudes around food are also important.
Take, for example, orthorexia nervosa, which is an obsessive preoccupation with consuming only “healthy” foods. If healthy eating only means ingesting healthy foods, then people with orthorexia are super healthy.
But people who live with this eating disorder often struggle with relationships and report poor quality of life, among other issues.
Research suggests that shifting the focus from food itself to our experience of eating can have a range of health benefits. Let’s take a look.
Hinterhaus Productions/Getty Why are we so obsessed with diet?
Equating “healthy eating” with “healthy diet” may have taken off in the early 1980s with panic over the “obesity epidemic” in Western countries – defined as a rapid rise in the prevalence of people in the population with a body mass index (BMI) of 30 or greater.
But causes of obesity are complex and poorly understood, with numerous possible explanations beyond simply what a person eats. And admonishing overweight people to eat “healthier” has done nothing to reduce population rates of obesity.
There is some evidence that this fixation on weight has resulted in increased rates of disordered eating and eating disorders – both of which involve problematic eating behaviours and distorted attitudes towards food, weight, shape and appearance.
Clearly, something needs to change in how we think about healthy eating.
Listening to your body
A growing body of research on intuitive eating has found this approach has an array of health benefits.
Intuitive eating means trusting internal body cues that tell us when, what and how much to eat. For example, tuning into your stomach growling telling you it’s time to eat, or noticing feeling full or satisfied, or that you may crave certain foods because your body wants specific nutrients (such as protein after exercising).
Studies have shown this approach can lead to better physical and mental health as well as better diet quality, and is associated with lower BMIs.
Research also shows eating at regular intervals and eating with other people also lead to better overall health and diet.
But if you find it hard, you’re not alone
Most of us are surrounded by food environments that make healthy eating difficult.
Unhealthy food environments promote overeating and encourage us to override our innate signals of hunger and fullness.
When we’re surrounded by cheap and accessible sugary snacks, fast foods and large portions – and lots of marketing – it can be hard to develop a positive relationship with food.
The issue is particularly acute for people in more disadvantaged communities.
For example, in our research with rural Australians about food and eating, most told us they wanted to eat more healthily, but found it difficult for many reasons, These included busy schedules and the cost of healthier food.
Habits and emotional eating can also make healthy eating difficult.
So, what works?
For most people, healthy behaviours and attitudes to eating mean a balanced, flexible and non-judgmental approach, without fear of “bad” foods. It means paying attention to hunger and fullness cues.
But it also means recognising that food is a source of social and cultural connection. A healthy attitude to food doesn’t ignore nutritional information – it incorporates this knowledge into a broader and more joyous approach to eating.
Here are three suggestions to get you started.
1. Recognising signs of hunger and fullness
These may differ from person to person. Can you hear your stomach start to growl or your energy begin to dip? Is it a while since you ate? And while eating, is there a point where the hunger has gone away and you no longer feel a strong desire to continue eating? Some people find using hunger and fullness scales useful.
2. Reframing “bad” foods
Is there a food you really like but don’t eat because you consider it “bad” or “forbidden”? Try incorporating a small amount into your next meal or snack. You may find that doing so brings greater joy to your eating while simultaneously taking away its power.
3. Eating with people
If you normally eat by yourself or “grab and go”, see if there’s a way to plan more time for meals and include other people – whether this is more family meals or group lunches with coworkers.
But some people have to follow a specific diet
People with medical conditions that require a particular type of diet – such as those with diabetes or coeliac disease – need to follow that advice. But they may still be able to have healthy behaviours and attitudes towards food even within these constraints.
For example, one 2020 study of people with type 2 diabetes found that more intuitive eaters had better control of their blood sugar levels.
The bottom line
So – if you don’t have a medical condition that prevents it – go ahead and have some of that birthday cake. And then listen to your body when it tells you you’ve had enough.
If you feel that you have an unhealthy relationship with food that is interfering with your life, please contact your GP to discuss your options. You may also want to contact the Butterfly Foundation for support.
Nina Van Dyke, Associate Professor and Associate Director, Mitchell Institute, Victoria University and Rosemary V. Calder, Professor, Health Policy, Victoria University
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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Your gluten sensitivity might be something else entirely, new study shows
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Social media and lifestyle magazines have turned gluten – a protein in wheat, rye and barley – into a dietary villain.
Athletes and celebrities have promoted gluten-free eating as the secret to better health and performance.
But our review in The Lancet published today challenges that idea.
By examining decades of research, we found that for most people who think they react to gluten, gluten itself is rarely the cause.
Daisy-Daisy/Getty Symptoms but not coeliac
Coeliac disease is when the body’s immune system attacks itself when someone eats gluten, leading to inflammation and damage to the gut.
But people with gut or other symptoms after eating foods containing gluten can test negative for coeliac disease or wheat allergy. They are said to have non-coeliac gluten sensitivity.
We wanted to understand whether gluten itself, or other factors, truly cause their symptoms.
What we did and what we found
Our study combined more than 58 studies covering symptom changes and possible ways they could arise. These included studying the immune system, gut barrier, microbes in the gut, and psychological explanations.
Across studies, gluten-specific reactions were uncommon and, when they occurred, changes in symptoms were usually small. Many participants who believed they were “gluten sensitive” reacted equally – or more strongly – to a placebo.
One landmark trial looked at the role of fermentable carbohydrates (known as FODMAPs) in people who said they were sensitive to gluten (but didn’t have coeliac disease). When people ate a low-FODMAP diet – avoiding foods such as certain fruits, vegetables, legumes and cereals – their symptoms improved, even when gluten was reintroduced.
Another showed fructans – a type of FODMAP in wheat, onion, garlic and other foods – caused more bloating and discomfort than gluten itself.
This suggests most people who feel unwell after eating gluten are sensitive to something else. This could be FODMAPs such as fructans, or other wheat proteins. Another explanation could be that symptoms reflect a disorder in how the gut interacts with the brain, similar to irritable bowel syndrome.
Some people may be truly sensitive to gluten. However, current evidence suggests this is uncommon.
People expected symptoms
A consistent finding is how expecting to have symptoms profoundly shapes people’s symptoms.
In blinded trials, when people unknowingly ate gluten or placebo, symptom differences almost vanished.
Some who expected gluten to make them unwell developed identical discomfort when exposed to a placebo.
This nocebo effect – the negative counterpart of placebo – shows that belief and prior experience influence how the brain processes signals from the gut.
Brain-imaging research supports this, showing that expectation and emotion activate brain regions involved in pain and how we perceive threats. This can heighten sensitivity to normal gut sensations.
These are real physiological responses. What the evidence is telling us is that focusing attention on the gut, coupled with anxiety about symptoms or repeated negative experiences with food, has real effects. This can sensitise how the gut interacts with the brain (known as the gut–brain axis) so normal digestive sensations are felt as pain or urgency.
Recognising this psychological contribution doesn’t mean symptoms are imagined. When the brain predicts a meal may cause harm, gut sensory pathways amplify every cramp or sensation of discomfort, creating genuine distress.
This helps explain why people remain convinced gluten is to blame even when blinded studies show otherwise. Symptoms are real, but the mechanism is often driven by expectation rather than gluten.
So what else could explain why some people feel better after going gluten-free? Such a change in the diet also reduces high-FODMAP foods and ultra-processed products, encourages mindful eating and offers a sense of control. All these can improve our wellbeing.
People also tend to eat more naturally gluten-free, nutrient-dense foods such as fruits, vegetables, legumes and nuts, which may further support gut health.
The cost of going gluten-free
For the approximately 1% of the population with coeliac disease, avoiding gluten for life is essential.
But for most who feel better gluten-free, gluten is unlikely to be the true problem.
There’s also a cost to going gluten-free unnecessarily. Gluten-free foods are, on average, 139% more expensive than standard ones. They are also often lower in fibre and key nutrients.
Avoiding gluten long term can also reduce diversity in your diet, alter your gut microbes and reinforce anxiety about eating.
Is it worth getting tested?
Unlike coeliac disease or a wheat allergy, non-coeliac gluten sensitivity has no biomarker – there’s no blood test or tissue marker that can confirm it.
Diagnosis instead relies on excluding other conditions and structured dietary testing.
Based on our review, we recommend clinicians:
- rule out coeliac disease and wheat allergy first
- optimise the quality of someone’s overall diet
- trial a low-FODMAP diet if symptoms persist
- only then, consider a four to six-week dietitian-supervised gluten-free trial, followed by a structured re-introduction of gluten-containing foods to see whether gluten truly causes symptoms.
This approach keeps restriction targeted and temporary, avoiding unnecessary long-term exclusion of gluten.
If gluten doesn’t explain someone’s symptoms, combining dietary guidance with psychological support often works best. That’s because expectation, stress and emotion influence our symptoms. Cognitive-behavioural or exposure-based therapies can reduce food-related fear and help people safely reintroduce foods they once avoided.
This integrated model moves beyond the simplistic “gluten is bad” narrative toward personalised, evidence-based gut–brain care.
Jessica Biesiekierski, Associate Professor of Human Nutrition, The University of Melbourne
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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