
Getting Rid Of Warts
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
It’s Q&A Day at 10almonds!
Have a question or a request? We love to hear from you!
In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!
As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!
So, no question/request too big or small 😎
❝How to get rid of warts and stop them from coming back?❞
First we’ll mention: it does matter what kind of warts they are, and where they are. It’s easier to treat a wart on a finger than on the genitals or an eyelid, for example.
To speak in broad terms, though, warts are caused by human papillomavirus (HPV).
There is a HPV vaccine; you can read about it here: Everything you need to know about cervical cancer*
You can get the vaccine as an adult, but it’s most readily given to children, as it is most effective before first encountering HPV. Otherwise, most people contract the virus at some point, and it is highly contagious, so prevention as early as possible is good. Sometimes people will hand-wring about giving a HPV vaccine to children as HPV is mostly transmitted sexually so it “shouldn’t” be necessary yet, but 1) better to get it done and not have to worry about it later 2) it’s not only transmitted sexually, but by touch in general, and it is highly resistant to disinfectants.
*You may be wondering: why is the link about cervical cancer? And the answer is that almost all cervical cancer is caused by HPV.
However, if you have warts, this does not mean you are necessarily at high risk of cervical cancer (or oropharyngeal cancer, which is also mostly caused by HPV).
There are hundreds of known types of HPV, and the most common wart-inducing types aren’t known to increase cancer risk. Please note that this is not an exhaustive statement though; there are a lot of types where it’s not fully known what they do. Also, many people will have numerous types of HPV.
You asked us about treating warts, so that’s important knowledge, but to save space on discussing the many types of HPV (which is a fascinating topic, especially when it comes to some of the most common types like 2 and 16, and the most risky types like 16 and 18), we’ll direct you to a convenient table of HPV types on Wikipedia that tells which kinds do what.
So, can we destroy the virus once infected?
Yes!
…ish. That is to say, we can destroy them locally (at the site of the wart), but we will still remain infected by the virus, meaning they can always reappear (we can reduce the risks though; more on that later).
Destroying it mostly comes in two main forms:
- Salicylic acid or similar chemical products: needs to be used every day, for weeks, but will destroy the wart tissue (and the virus contained within it) while leaving healthy tissue mostly unaffected (it’s only mildly corrosive to our own flesh) but still, try to get it only on the wart. Here’s an example product on Amazon.
- Liquid nitrogen or other freezing treatments: usually only takes a few treatments to destroy it completely. Liquid nitrogen is usually available only via a doctor, unless for some reason you happen to have access to it yourself, but we recommend getting professional (medical professional!) assistance, as otherwise it can very easily destroy your flesh too. Milder, at-home freezing treatments are not as effective as liquid nitrogen, but still much more effective than corrosive chemical treatments. The at-home kits usually involve a canister containing chemicals that produce an endothermic reaction when mixed, and this mixture is then either sprayed via a nozzle placed around the wart (to avoid getting other tissue), or else isn’t sprayed anywhere, and just cools a conductive metal element the tip of which is then placed on the wart to freeze it off. Here’s an example product on Amazon.
There are a lot of home remedies that people try; most of them do not work.
Here’s a list: 16 Natural Home Remedies for Warts ← we’re not recommending these, but we link them for your interest.
About avoiding reoccurrence
There are two main things here:
- don’t reinfect yourself: so for example try to avoid touching it (spreads it about anywhere else you touch), and consider anything you used on it physically (e.g. pumice stone, nail file, etc) contaminated and now capable of spreading it to other parts of you (or indeed other people, if it’s a shared item, so don’t share it). Remember, it’s very resistant to disinfecting, so unless you have a medical grade autoclave or seriously strong industrial chemicals, you’re very unlikely to successfully disinfect such items at home.
- look after your immune system: most warts go away by themselves in about 18 months. Is it because they just got fed up of being a nuisance? No, it’s because your immune system finally beat them (on that particular battleground, at least). So, look after your immune system, and it will not only help you get rid of extant warts more quickly, but also reduce the risk of reoccurrence.
Learn more about that latter: Beyond Supplements: The Real Immune-Boosters!
Take care!
Don’t Forget…
Did you arrive here from our newsletter? Don’t forget to return to the email to continue learning!
Recommended
Learn to Age Gracefully
Join the 98k+ American women taking control of their health & aging with our 100% free (and fun!) daily emails:
-
Barley Malt Flour vs chickpea flour – Which is Healthier?
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
Our Verdict
When comparing barley malt flour to chickpea flour, we picked the chickpea.
Why?
First, some notes:
About chickpea flour: this is also called besan flour, gram flour, and garbanzo bean flour; they are all literally the same thing by different names, and are all flour made from ground chickpeas.
About barley malt flour: barley is a true grain, and does contain gluten. We’re not going to factor that into today’s decision, but if you are avoiding gluten, avoid barley. As for “malt”; malting grains means putting them in an environment (with appropriate temperature and humidity) that they can begin germination, and then drying them with hot air to stop the germination process from continuing, so that we still have grains to make flour out of, and not little green sprouting plants. It improves the nutritional qualities and, subjectively, the flavor.
To avoid repetition, we’re just going to write “barley” instead of “barley malt” now, but it’s still malted.
Now, let’s begin:
Looking at the macros first, chickpea flour has 2x the protein and also more fiber, while barley flour has more carbs. An easy win for chickpea flour.
In the category of vitamins, chickpea flour has more of vitamins A, B1, B5, B9, E, and K, while barley flour has more of vitamins B2, B3, B6, and C. A modest 6:4 victory for chickpea flour.
When it comes to minerals, things are much more one-sided; chickpea flour has more calcium, copper, iron, magnesium, manganese, phosphorus, potassium, and zinc, while barley flour has more selenium. An overwhelming win for chickpea flour.
Adding up these three wins for chickpea flour makes for a convincing story in favor of using that where reasonably possible as a flour! It has a slight nutty taste, so you might not want to use it in everything, but it is good for a lot of things.
Want to learn more?
You might like to read:
- Grains: Bread Of Life, Or Cereal Killer?
- Gluten: What’s The Truth?
- Sprout Your Seeds, Grains, Beans, Etc
Take care!
Share This Post
-
Demystifying C-Reactive Protein (CRP)!
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
Most people over a certain age who are at least somewhat invested in their health know that c-reactive protein (CRP) is a blood biomarker that’s considered an indicator of cardiovascular health (or: illness, as the case may be), but often know little more than that.
So, what is it and what does it mean?
Oh, CRP
CRP is produced by the liver in response to immune activation from infections, tissue damage, autoimmune disease, obesity, and diabetes.
As for what its actual job is (because the body does produce it for reasons other than for its usefulness as a biomarker in blood tests),
❝Similar to immunoglobulin (Ig)G, it activates complement, binds to Fc receptors and acts as an opsonin for various pathogens. Interaction of CRP with Fc receptors leads to the generation of proinflammatory cytokines that enhance the inflammatory response. Unlike IgG, which specifically recognizes distinct antigenic epitopes, CRP recognizes altered self and foreign molecules based on pattern recognition. Thus, CRP is though to act as a surveillance molecule for altered self and certain pathogens. This recognition provides early defense and leads to a proinflammatory signal and activation of the humoural, adaptive immune system.❞
Source: Function of C-reactive protein
Translating that from sciencese: CRP acts like a first-responder version of an antibody. Like IgG antibodies, it can activate the complement* system, latch onto immune cell receptors, and tag microbes so they are easier for immune cells to destroy. When CRP binds to these receptors, it also triggers the release of inflammatory signals that dial up the immune response.
The key difference is how it recognizes threats: antibodies such as IgG are very specific—they are custom-made to recognize one precise target and nothing else. CRP, in contrast, looks for broad patterns that signal damage or danger, whether from invading microbes or the body’s own altered cells. Thus, CRP acts as an immune surveillance sensor, providing early warning and kick-starting inflammation and the wider adaptive immune response. Which, in the case of an actual infection or similar, is a good thing.
*You may be wondering what, in turn, the complement system is and what activating it means. In simple terms, it starts off as a bunch of proteins circulating in your blood in an inactive form. When CRP (or an antibody) binds to a microbe or a damaged cell, it can activate this cascade, which then joints the fight and also does the latching on and tagging that we mentioned, by:
- directly damaging microbes: in some cases, complement proteins punch holes in bacterial membranes, leading to their death.
- tagging the target for destruction: complement proteins typically coat the surface of the microbe or damaged cell, making it easier for immune cells to recognize and engulf it.
- dialling up inflammation: small complement fragments act like chemical alarms, attracting immune cells and increasing local inflammation.
Again, if there’s actually a genuine threat to respond to, these are all good things for it to be doing.
CRP as a biomarker
CRP is a very useful biomarker of low-grade inflammation, and evidence from decades of research shows it predicts heart attacks and strokes better than LDL cholesterol and lipoprotein(a), and at least as well as blood pressure: Inflammation, Cholesterol, Lipoprotein(a), and 30-Year Cardiovascular Outcomes in Women
Same goes for when we look at mortality: C-reactive protein concentration and risk of coronary heart disease, stroke, and mortality: an individual participant meta-analysis
For this reason, the American College of Cardiology recommended universal CRP screening alongside cholesterol testing to improve cardiovascular risk assessment. You can read that in full, here:
You may be wondering about numbers: CRP under 1 mg/dL indicates low inflammation and lower risk, while CRP above 3 mg/dL signals higher inflammation and higher risk.
How likely is it that you are at risk? Well, about 52% of Americans have elevated CRP, so that’s not a promising figure to start with. Now, 10almonds readers are doubtlessly healthier than the average American as a general demographic, but still, it’s worth bearing in mind and not assuming that it could only apply to other people.
And as for how to improve your numbers? Same deal as most heart health advice that we give here: enjoy a plants-forward (if not entirely plant-based) diet, especially making sure to get a lot of fiber and as many different sources of polyphenols as is reasonable. Get good sleep, do exercise often, and don’t smoke or drink.
About the dietary aspects specifically, see:
- What Matters Most For Your Heart?
- What Do The Different Kinds Of Fiber Do? 30 Foods That Rank Highest
- 21 Most Beneficial Polyphenols & What Foods Have Them
Want to learn more?
If you’d like a comprehensive guide to reducing your heart disease risk, no matter your starting point, then you might like to consider:
Dr. Dean Ornish’s Program For Reversing Heart Disease – by Dr. Dean Ornish
Take care!
Share This Post
-
Finish What You Start – by Peter Hollins
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
For some people, getting started is the problem. For others of us, getting started is the easy part! We just need a little help not dropping things we started.
There are summaries at the starts and ends of sections, and many “quick tips” to get you back on track.
As a taster: one of these is “temptation bundling“, combining unpleasant things with pleasant. A kind of “spoonful of sugar” approach.
Hollins also discusses hyperbolic discounting (the way we tend to value rewards according to how near they are, and procrastinate accordingly). He offers a tool to overcome this, too, the “10–10–10 rule“.
Also dealt with is “the preparation trap“, and how to know when you have enough information to press on.
For a lot of us, the places we’re most likely to drop a project is 20% in (initial enthusiasm wore off) or 80% in (“it’s nearly done; no need to worry about it”). Those are the times when the advices in this book can be particularly handy!
All in all, a great book for seeing a lot of things to completion.
Share This Post
Related Posts
-
How (And Why) To Do Foot-Rolling
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
Foot rolling isn’t just for pain relief—it helps rewire nerve connections, improve balance, and support overall well-being.
There are some things to bear in mind if you want to benefit, though:
On the ball
With the ball underfoot, roll the ball back and forth and in small circles for a couple of minutes, and:
- if you find a tender spot, press there and roll for about 15 seconds before continuing
- if doing this while seated, lean forwards slightly to apply your weight to it at the correct angle
- if you can, do it while standing though, as this will enable you to get the full benefits of not just the foot pressure, but also the improvement to your balance and stabilization muscles
Because of the nerve connections, this can (especially if you use a massage ball, the kind with many small blunt spikes) also engage your parasympathetic nervous system, thus activating your relaxation response, which means lowered cortisol levels, improved digestion and healing, and various other “rest and digest” benefits that one doesn’t get while in fight-or-flight mode or even in neutral.
For more on this plus a visual demonstration, enjoy:
Click Here If The Embedded Video Doesn’t Load Automatically!
Want to learn more?
You might also like:
The Vagus Nerve (And How You Can Make Use Of It) ← the vagus nerve doesn’t go all the way down to the feet itself, of course, but the message will get up to the vagus nerve anyway
Take care!
Don’t Forget…
Did you arrive here from our newsletter? Don’t forget to return to the email to continue learning!
Learn to Age Gracefully
Join the 98k+ American women taking control of their health & aging with our 100% free (and fun!) daily emails:
-
What Your Brain Is Really Doing When You’re Doing “Nothing”
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
Unless we are dead, our brain is never truly inactive. And it’s not just a matter of regulating autonomic functions, either…
Default Mode Network
When the brain is at rest but not necessarily asleep, the Default Mode Network (DMN) engages. This makes up for around 20% of the brain’s overall activity, and contributes to complex cognitive processes.
What constitutes “at rest”: the DMN activates when external tasks stop and is engaged during self-reflection, mind-wandering, and relaxed memory recall (i.e. reminiscing, rather than answering questions in a difficult test, for example).
As for its neurophysiology, the DMN is connected to the hippocampus and plays a key role in episodic, prospective, and semantic memory (memories of experiences, future plans, and general knowledge), as well as being involved in self-reflection, social cognition, and understanding others’ thoughts (theory of mind). The DMN thus also helps integrate memories and thoughts to create a cohesive internal narrative and sense of self.
However, it doesn’t work alone: the DMN interacts with other networks like the salience network, which switches attention to external stimuli. Disruptions between these networks are linked to psychiatric disorders (e.g., schizophrenia, Alzheimer’s, depression), in various different ways depending on the nature of the disruption.
Sometimes, for some people in some circumstances, the option to disrupt the DMN is useful. For example, research shows that psilocybin disrupts the DMN, leading to changes in brain activity and potential therapeutic benefits for depression* and other psychiatric disorders by enhancing neuroplasticity.
*Essentially, kicking the brain out of the idling gear it got stuck in, and into action
For more on all of this, enjoy:
Click Here If The Embedded Video Doesn’t Load Automatically!
Want to learn more?
You might also like to read:
- The Wandering Mind – by Dr. Michael Corballis ← a book, largely about the DMN and how to use it beneficially
- Taking A Trip Through The Evidence On Psychedelics ← for a shorter read, touching on psilocybin
Take care!
Don’t Forget…
Did you arrive here from our newsletter? Don’t forget to return to the email to continue learning!
Learn to Age Gracefully
Join the 98k+ American women taking control of their health & aging with our 100% free (and fun!) daily emails:
-
Antiviral Gum Gives Epidemiologists Something To Chew On
10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.
With viruses on the rise, of course one of our biggest weapons against them is vaccination, but that approach has its limitations:
- In some places such as the US, anti-vaccine sentiments are high, and a vaccine is only as good as its uptake (i.e. if people don’t take it, they will more likely catch the disease and pass it on, including to some people who cannot be vaccinated, so non-vaccinators create a hole in herd immunity)
- Many vaccines can become outdated when viruses mutate more quickly than vaccines can be developed (we’ve seen a lot of this with COVID and Flu viruses, and that’s why we keep needing new ones)
- There are some viruses for which we simply do not yet have vaccines; sometimes this is the case even for very common viruses like Herpes simplex. or, indeed, the common cold (Rhinovirus sp.).
So, antivirals definitely have their place too. To be clear about the difference:
- A vaccine forewarns the immune system “watch out for this thing that you might encounter in the future, and prepare a defense for it according to these specifications” (it only helps if you aren’t already infected with the thing it’s vaccinating against, because otherwise the warning is too late and your body is already trying to mount a defense)
- An antiviral kills, inactivates, or otherwise severely inconveniences the virus directly (it only helps if there is a virus there to fight)
How the antiviral gum works
In few words: you chew it, the antiviral substance is then in your saliva, and it kills/inactivates/inconveniences the virus at the site of infection (e.g. your respiratory tract)
In the case of this specific antiviral gum, it’s more in the category of “severely inconveniences”, because the antiviral substance is a protein trap that binds to the virus, rendering it near-harmless.
In essence, therefore, it works less like a vaccine and more like a facemask (except it’s trapping the virus on the molecular level, rather than trying to stop aerosolized droplets from moving around on the macro level).
This was first developed as a possible tool against COVID:
…and this in turn was based on previous work quite early in 2020:
And yes, those are lablab beans, as in Lablab purpureus, also called hyacinth beans, which may not be available in all supermarkets, but are not very obscure either (common throughout most of Africa and the tropics).
Most recently, researchers have found that 40mg of the broad-spectrum (as in, it affects many viruses) antiviral trap protein, as delivered by a 2g piece of gum, was sufficient to reduce viral loads by more than 95%, including for SARS-CoV-2 as well as H5N1, H3N2, and H7N9 (various kinds of bird flu that affect humans), and HSV-1 and HSV-2 (the two most common variants of herpes, including cold sores):
You can also read a pop-science article about it, with links to more details, here:
Antiviral chewing gum shows promise in reducing influenza and herpes spread
Want to learn more?
Check out:
Winning The Biological Arms Race: Could This Be “The Ultimate Booster”?
Take care!
Don’t Forget…
Did you arrive here from our newsletter? Don’t forget to return to the email to continue learning!
Learn to Age Gracefully
Join the 98k+ American women taking control of their health & aging with our 100% free (and fun!) daily emails:







