Eating Disorders: More Varied (And Prevalent) Than People Think

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Disordered Eating Beyond The Stereotypes

Around 10% of Americans* have (or have had) an eating disorder. That might not seem like a high percentage, but that’s one in ten; do you know 10 people? If so, it might be a topic that’s near to you.

*Source: Social and economic cost of eating disorders in the United States of Americ

Our hope is that even if you yourself have never had such a problem in your life, today’s article will help arm you with knowledge. You never know who in your life might need your support.

Very misunderstood

Eating disorders are so widely misunderstood in so many ways that we nearly made this a Friday Mythbusting edition—but we preface those with a poll that we hope to be at least somewhat polarizing or provide a spectrum of belief. In this case, meanwhile, there’s a whole cluster of myths that cannot be summed up in one question. So, here we are doing a Psychology Sunday edition instead.

“Eating disorders aren’t that important”

Eating disorders are the second most deadly category of mental illness, second only to opioid addiction.

Anorexia specifically has the highest case mortality rate of any mental illness:

Source: National Association of Anorexia Nervosa & Associated Disorders: Eating Disorder Statistics

So please, if someone needs help with an eating disorder (including if it’s you), help them.

“Eating disorders are for angsty rebellious teens”

While there’s often an element of “this is the one thing I can control” to some eating disorders (including anorexia and bulimia), eating disorders very often present in early middle-age, very often amongst busy career-driven individuals using it as a coping mechanism to have a feeling of control in their hectic lives.

13% of women over 50 report current core eating disorder symptoms, and that is probably underreported.

Source: as above; scroll to near the bottom!

“Eating disorders are a female thing”

Nope. Officially, men represent around 25% of people diagnosed with eating disorders, but women are 5x more likely to get diagnosed, so you can do the math there. Women are also 1.5% more likely to receive treatment for it.

By the time men do get diagnosed, they’ve often done a lot more damage to their bodies because they, as well as other people, have overlooked the possibility of their eating being disordered, due to the stereotype of it being a female thing.

Source: as above again!

“Eating disorders are about body image”

They can be, but that’s far from the only kind!

Some can be about control of diet, not just for the sake of controlling one’s body, but purely for the sake of controlling the diet itself.

Still yet others can be not about body image or control, like “Avoidant/Restrictive Food Intake Disorder”, which in lay terms sometimes gets dismissed as “being a picky eater” or simply “losing one’s appetite”, but can be serious.

For example, a common presentation of the latter might be a person who is racked with guilt and/or anxiety, and simply stops eating, because either they don’t feel they deserve it, or “how can I eat at a time like this, when…?” but the time is an ongoing thing so their impromptu fast is too.

Still yet even more others might be about trying to regulate emotions by (in essence) self-medicating with food—not in the healthy “so eat some fruit and veg and nuts etc” sense, but in the “Binge-Eating Disorder” sense.

And that latter accounts for a lot of adults.

You can read more about these things here:

Psychology Today | Types of Eating Disorder ← it’s pop-science, but it’s a good overview

Take care! And if you have, or think you might have, an eating disorder, know that there are organizations that can and will offer help/support in a non-judgmental fashion. Here’s the ANAD’s eating disorder help resource page, for example.

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  • How To Be 7.5x More Likely To Develop Chronic Fatigue Syndrome

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    First, what is it?

    Many more people have chronic fatigue, which is the symptom of being exhausted all the time, than have chronic fatigue syndrome (CFS) which is the illness of myalgic encephalomyelitis (ME).

    This is because fatigue can be a symptom of many, many other conditions, and can be heavily influenced by lifestyle factors too.

    A lot of the advice for dealing with chronic fatigue is often the same in both cases, but some will be different, because for example:

    • If your fatigue is from some other condition, that condition probably impacts what lifestyle factors you are (and are not) able to change, too
    • If your fatigue is from lifestyle factors, that hopefully means you can change those and enjoy less fatigue…
      • But if it’s not from lifestyle factors, as in ME/CFS, then advice to “exercise more” etc is not going to help so much.

    There are ways to know the difference though:

    Check out: Do You Have Chronic Fatigue Syndrome?

    The chronic disease pipeline

    While it had been strongly suspected that COVID infection could lead to CFS, with long COVID having chronic fatigue as one of its characteristic symptoms, a research team led by Dr. Suzanne Vernon has now established the nature of the relationship.

    It was a large (n=13,224) longitudinal observational cohort study of people with no pre-existing ME/CFS, grouped according to their COVID infection status:

    1. acute infected, enrolled within 30 days of infection or enrolled as uninfected who became infected (n=4,515)
    2. post-acute infected, enrolled greater than 30 days after infection (n=7,270)
    3. uninfected (n=1,439).

    (to be clear, that last means “never infected”, or else they would be in group 2)

    Note: people who had COVID and were hospitalized for it were excluded from the study, so this risk is the risk represented by even just more “moderate” infections.

    What they found:

    ❝The proportion of all RECOVER-Adult participants that met criteria for ME/CFS following SARS-CoV-2 infection was 4.5% (531 of 11,785) compared to 0.6% (9 of 1439) in uninfected participants.❞

    There are then different numbers if we look per 100 person-years, as the study also did—in which case, we get a re-modelled increase in risk of 5x instead of 7.5x, but a) that’s still not good b) the “here-and-now” figures of 4.5% vs 0.6% are also relevant.

    Read in full: Incidence and Prevalence of Post-COVID-19 Myalgic Encephalomyelitis: A Report from the Observational RECOVER-Adult Study

    The killer nobody wants to talk about anymore

    Of course, as we all know the pandemic is over, because politicians declared it so, which is very reassuring.

    Nevertheless, COVID is currently the still 4th leading cause of death in the US, placing it higher than stroke, Alzheimer’s, diabetes, and others.

    See also: Emergency or Not, Covid Is Still Killing People. Here’s What Doctors Advise to Stay Safe

    So, while it’s very good to take care of our hearts, brains, blood sugars, and so forth, let’s at the very least continue to keep on top of our vaccinations, avoid enclosed crowded spaces where possible, etc.

    And for extra boosts to one’s chances: Why Some People Get Sick More (And How To Not Be One Of Them)

    What if I do get (or already have) long COVID and/or ME/CFS?

    Well, that is definitely going to suck, but there are still some things that can be done.

    Here’s a big one: How To Eat To Beat Chronic Fatigue ← this will not, of course, cure you, but it’s a way of getting maximum nutrition for minimum effort, given that for someone with chronic fatigue, effort is a very finite resource that must be used sparingly

    Finally, here are some further resources:

    Support For Long COVID & Chronic Fatigue

    Take care!

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  • What Nobody Teaches You About Strengthening Your Knees

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    Strengthening unhappy knees can seem difficult, because many obvious exercises like squats may hurt, and can feel like they are doing harm (and if your knees are bad enough, maybe they are; it depends on many factors). Here’s a way to improve things:

    The muscle nobody talks about

    Well, not nobody. But, it’s a muscle that’s rarely talked about; namely, the tibialis anterior.

    It plays a key role in decelerating knee motion—in other words, the movement that hurts if you have bad knees. It’s essential for absorbing shock during activities like walking, climbing stairs, and stepping off curbs

    So, of course, strengthening this muscle supports knee health.

    The exercise this video recommends for strengthening it involves leaning against a wall with feet about a foot away (closer feet make it easier, further makes it harder). Note, this is a lean, not a “Roman chair”.

    The exercise involves squeezing the quadriceps, lifting toes toward the nose, and engaging the tibialis anterior muscle. If you’re wondering what to do with your hands, they can be held out with palms open to work on posture, or hanging by the sides. Do this for about 1½–2 minutes.

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    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

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  • Your Heart In Their Hands: Surgeon Preferences & Survival Rates

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    Unless you are paying entirely out-of-pocket for a heart surgery, you will not usually get final say over which surgeon you get.

    The surgeon, however, will have final say over what they actually do when they open you up.

    And their preferences, it seems, can make all the difference:

    MAG vs SAG

    When doing coronary artery bypass grafting, (CABG), surgeons may prefer to do multi-arterial grafting (MAG) or single-arterial grafting (SAG).

    Recently, there was a study analysing more than a million Americans who underwent CABG on Medicare over an 18-year period, looking at outcomes for MAG vs SAG.

    The superficial news: those who received MAG had much better long-term survival chances than those who received SAG.

    However: this may be less to do with the relative merits of the procedures themselves, and more to do with the preferences of the surgeon.

    The “eyeball test”

    If surgeons look at a patient and think they will not have many years to live after surgery, they may opt for the SAG, as the long-term benefits of the MAG will only manifest in the long-term.

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    Or you might just not have enough donor artery tissue available to pick and choose; after all, a person having a coronary artery bypass quite possibly won’t have great arteries in their arm or leg, either.

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    Why it might be ok that things are like this

    When factoring in surgeon preference for MAG or SAG as an instrumental variable, no significant difference in long-term survival was observed. This may explain inconsistencies with randomized controlled trials like the Arterial Revascularization Trial (ART), which also found no survival benefit of MAG over SAG.

    Also, MAG recipients were generally younger, healthier, and from more resourceful areas, which likely had a further impact on MAG-giving decisions, and/but at the same time, may also have increased survival chances for reasons other than that they got MAG rather than SAG.

    Here’s a pop-science article that goes into more detail about this:

    Surgeon preferences may explain differences in CABG survival rates

    How to look out for yourself, and advocate for yourself

    …or your loved one, of course. Now, having a coronary artery bypass surgery of any kind is not a fun activity; it will be dangerous, it’ll be stressful before and after, and the recovery will often not be an easy time either. However, it is possible to learn more about what is going on / what will happen, ask the right questions, and get the best options for you (which may not always be the same as the best options for someone else).

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  • How much does your phone’s blue light really delay your sleep? Relax, it’s just 2.7 minutes

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    It’s one of the most pervasive messages about technology and sleep. We’re told bright, blue light from screens prevents us falling asleep easily. We’re told to avoid scrolling on our phones before bedtime or while in bed. We’re sold glasses to help filter out blue light. We put our phones on “night mode” to minimise exposure to blue light.

    But what does the science actually tell us about the impact of bright, blue light and sleep? When our group of sleep experts from Sweden, Australia and Israel compared scientific studies that directly tested this, we found the overall impact was close to meaningless. Sleep was disrupted, on average, by less than three minutes.

    We showed the message that blue light from screens stops you from falling asleep is essentially a myth, albeit a very convincing one.

    Instead, we found a more nuanced picture about technology and sleep.

    Mangostar/Shutterstock

    What we did

    We gathered evidence from 73 independent studies with a total of 113,370 participants of all ages examining various factors that connect technology use and sleep.

    We did indeed find a link between technology use and sleep, but not necessarily what you’d think.

    We found that sometimes technology use can lead to poor sleep and sometimes poor sleep can lead to more technology use. In other words, the relationship between technology and sleep is complex and can go both ways.

    How is technology supposed to harm sleep?

    Technology is proposed to harm our sleep in a number of ways. But here’s what we found when we looked at the evidence:

    • bright screen light – across 11 experimental studies, people who used a bright screen emitting blue light before bedtime fell asleep an average of only 2.7 minutes later. In some studies, people slept better after using a bright screen. When we were invited to write about this evidence further, we showed there is still no meaningful impact of bright screen light on other sleep characteristics including the total amount or quality of sleep
    • arousal is a measure of whether people become more alert depending on what they’re doing on their device. Across seven studies, people who engaged in more alerting or “exciting” content (for example, video games) lost an average of only about 3.5 minutes of sleep compared to those who engaged in something less exciting (for example, TV). This tells us the content of technology alone doesn’t affect sleep as much as we think
    • we found sleep disruption at night (for example, being awoken by text messages) and sleep displacement (using technology past the time that we could be sleeping) can lead to sleep loss. So while technology use was linked to less sleep in these instances, this was unrelated to being exposed to bright, blue light from screens before bedtime.

    Which factors encourage more technology use?

    Research we reviewed suggests people tend to use more technology at bedtime for two main reasons:

    There are also a few things that might make people more vulnerable to using technology late into the night and losing sleep.

    We found people who are risk-takers or who lose track of time easily may turn off devices later and sacrifice sleep. Fear of missing out and social pressures can also encourage young people in particular to stay up later on technology.

    What helps us use technology sensibly?

    Last of all, we looked at protective factors, ones that can help people use technology more sensibly before bed.

    The two main things we found that helped were self-control, which helps resist the short-term rewards of clicking and scrolling, and having a parent or loved one to help set bedtimes.

    Mother looking over shoulder of teen daughter sitting on sofa using smartphone
    We found having a parent or loved one to help set bedtimes encourages sensible use of technology. fast-stock/Shutterstock

    Why do we blame blue light?

    The blue light theory involves melatonin, a hormone that regulates sleep. During the day, we are exposed to bright, natural light that contains a high amount of blue light. This bright, blue light activates certain cells at the back of our eyes, which send signals to our brain that it’s time to be alert. But as light decreases at night, our brain starts to produce melatonin, making us feel sleepy.

    It’s logical to think that artificial light from devices could interfere with the production of melatonin and so affect our sleep. But studies show it would require light levels of about 1,000-2,000 lux (a measure of the intensity of light) to have a significant impact.

    Device screens emit only about 80-100 lux. At the other end of the scale, natural sunlight on a sunny day provides about 100,000 lux.

    What’s the take-home message?

    We know that bright light does affect sleep and alertness. However our research indicates the light from devices such as smartphones and laptops is nowhere near bright or blue enough to disrupt sleep.

    There are many factors that can affect sleep, and bright, blue screen light likely isn’t one of them.

    The take-home message is to understand your own sleep needs and how technology affects you. Maybe reading an e-book or scrolling on socials is fine for you, or maybe you’re too often putting the phone down way too late. Listen to your body and when you feel sleepy, turn off your device.

    Chelsea Reynolds, Casual Academic/Clinical Educator and Clinical Psychologist, College of Education, Psychology and Social Work, Flinders University

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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