The Mediterranean Diet Cookbook for Beginners – by Jessica Aledo

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There are a lot of Mediterranean Diet books on the market, and not all of them actually stick to the Mediterranean Diet. There’s a common mistake of thinking “Well, this dish is from the Mediterranean region, so…”, but that doesn’t make, for example, bacon-laden carbonara part of the Mediterranean Diet!

Jessica Aledo does better, and sticks unwaveringly to the Mediterranean Diet principles.

First, she gives a broad introduction, covering:

  • The Mediterranean Diet pyramid
  • Foods to eat on the Mediterranean Diet
  • Foods to avoid on the Mediterranean Diet
  • Benefits of the Mediterranean Diet

Then, it’s straight into the recipes, of which there are 201 (as with many recipe books, the title is a little misleading about this).

They’re divided into sections, thus:

  • Breakfasts
  • Lunches
  • Snacks
  • Dinners
  • Desserts

The recipes are clear and simple, one per double-page, with high quality color illustrations. They give ingredients/directions/nutrients. There’s no padding!

Helpfully, she does include a shopping list as an appendix, which is really useful!

Bottom line: if you’re looking to build your Mediterranean Diet repertoire, this book is an excellent choice.

Get your copy of The Mediterranean Diet Cookbook for Beginners from Amazon today!

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  • Basil vs Oregano – Which is Healthier?

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    Our Verdict

    When comparing basil to oregano, we picked the basil.

    Why?

    You may be thinking: these are just herbs; we don’t eat enough of these for the nutritional values to be relevant!

    And to this we say: there’s nothing stopping you :p Herbs are full of flavor and goodness and there is really no reason to deny yourself. On this note, check out the sabzi khordan (traditional Levantine herb platter), linked below. You’ll start thinking about herbs in new ways, and you can thank us later!

    Now, in terms of macros, nominally basil has more protein and oregano has more carbs and fiber, but the numbers are so close in each case that we’re going to call this category a tie.

    When it comes to vitamins, things get more interesting: basil has more of vitamins B2, B3, B6, B9, K, and choline, while oregano has more of vitamins A, B1, B5, C, and E. This means a 6:5 win for basil, but note how the two herbs together give an impressive vitamin coverage. In other words, they complement each other nutritionally, not just culinarily!

    In the category of minerals, basil has more calcium, copper, iron, magnesium, manganese, phosphorus, potassium and zinc, while oregano has more selenium. Now, this is obviously a clear win for basil, but we’d like to highlight that both of these herbs are incredibly rich in minerals (i.e. oregano is a very good source of all those minerals we listed for basil, too!); it’s just that basil has even more of most of them.

    When looking at any nutrient-dense food (which most herbs are), it’s worth looking at polyphenols. In this case, both are very abundant in polyphenols, and/but their respective numbers are close enough to be within each other’s margin of variation (i.e. exact numbers will depend on the individual plant’s life history), so this category is a tie.

    Adding up the sections makes for an overall clear win for basil, but absolutely please do enjoy both unless you have a good reason not to—they complement each other so well, in nutrients as well as in flavor!

    Want to learn more?

    You might like to read:

    Enjoy!

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  • 7 Essential Devices For Hand Arthritis: Regain Control of Your Life

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Dr. Diana Girnita is a double board-certified physician in rheumatology and internal medicine. With a PhD in immunology (on top of her MD), and training at Harvard and top universities, she founded Rheumatologist OnCall, offering integrative medicine to broaden rheumatology access. Here’s what she has to say about things that make life easier:

    Get your hands on these…

    The seven devices that Dr. Girnita recommends are:

    • Hand grip strengthener: helps build grip strength with a spring-loaded mechanism. Regular use can improve strength and reduce pain.
    • Finger exerciser: different device; similar principle: it strengthens hand and finger muscles using resistance, enhancing hand function.
    • Moisturizing paraffin bath: a heated paraffin wax bath that soothes hands, providing heat therapy and moisturizing the skin.
    • Weighted silverware: weighted utensils (knives, forks, spoons) make gripping easier and provide stability for eating.
    • Foam tubing grips: foam covers to make kitchen tools, toothbrushes, and hairbrushes easier to grip.
    • Electric can-opener: reduces strain in opening cans, making meal preparation more accessible.
    • Compression gloves: provide gentle compression to reduce swelling and pain, improving hand flexibility and circulation.
    • Door knob cover grips: make it easier to turn doorknobs by providing a larger surface to grip.
    • Wider-grip pens: ergonomically designed pens with a larger diameter and softer grip reduce hand strain while writing.

    This writer, who does not have arthritis but also does not have anything like the grip strength she used to, also recommends a jar opener like this one.

    As a bonus, if you spend a lot of time writing at a computer, an ergonomic split keyboard like this one goes a long way to avoiding carpal tunnel syndrome, and logically must be better for arthritis than a regular keyboard; another excellent thing to have (that again this writer uses and swears by) is an ergonomic vertical mouse like this one (aligns the wrist bones correctly; the “normal” horizontal version is woeful for the carpal bones). These things are both also excellent to help avoid worsening peripheral neuropathy (something that troubles this writer’s wrists if she’s not careful, due to old injuries there).

    For more on the seven things otherwise listed above, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like to read:

    Take care!

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  • DVT Risk Management Beyond The Socks

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    It’s Q&A Day at 10almonds!

    Have a question or a request? We love to hear from you!

    In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!

    As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!

    So, no question/request too big or small

    ❝I know I am at higher risk of DVT after having hip surgery, any advice beside compression stockings?❞

    First of all, a swift and easy recovery to you!

    Surgery indeed increases the risk of deep vein thrombosis (henceforth: DVT), and hip or knee surgery especially so, for obvious reasons.

    There are other risk factors you can’t control, like genetics (family history of DVT as an indicator) and age, but there are some that you can, including:

    • smoking (so, ideally don’t; do speak to your doctor before quitting though, in case withdrawal might be temporarily worse for you than smoking)
    • obesity (so, losing weight is good if overweight, but if this is going to happen, it’ll mostly happen in the kitchen not the gym, which may be a relief as you’re probably not the very most up for exercise at present)
    • sedentariness (so, while you’re probably not running marathons right now, please do try to keep moving, even if only gently)

    Beyond that, yes compression socks, but also frequent gentle massage can help a lot to avoid clots forming.

    Also, no surprises, a healthy diet will help, especially one that’s good for general heart health. Check out for example the Mediterranean DASH diet:

    Four Ways To Upgrade The Mediterranean Diet

    Also, obviously, speak with your doctor/pharmacist if you haven’t already about possible medications, including checking whether any of your current medications increase the risk and could be swapped for something that doesn’t.

    Take care!

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Related Posts

  • Science of Stretch – by Dr. Leada Malek
  • Sweet Dreams Are Made Of Cheese (Or Are They?)

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    It’s Q&A Day at 10almonds!

    Have a question or a request? You can always hit “reply” to any of our emails, or use the feedback widget at the bottom!

    In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!

    As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!

    So, no question/request too big or small

    ❝In order to lose a little weight I have cut out cheese from my diet – and am finding that I am sleeping better. Would be interested in your views on cheese and sleep, and whether some types of cheese are worse for sleep than others. I don’t want to give up cheese entirely!❞

    In principle, there’s nothing in cheese that, biochemically, should impair sleep. If anything, its tryptophan content could aid good sleep.

    Tryptophan is found in many foods, including cheese, which (of common foods, anyway), for example cheddar cheese ranks second only to pumpkin seeds in tryptophan content.

    Tryptophan can be converted by the body into 5-HTP, which you’ve maybe seen sold as a supplement. Its full name is 5-hydroxytryptophan.

    5-HTP can, in turn, be used to make melatonin and/or serotonin. Which of those you will get more of, depends on what your body is being cued to do by ambient light/darkness, and other environmental cues.

    If you are having cheese and then checking your phone, for instance, or otherwise hanging out where there are white/blue lights, then your body may dutifully convert the tryptophan into serotonin (calm wakefulness) instead of melatonin (drowsiness and sleep).

    In short: the cheese will (in terms of this biochemical pathway, anyway) augment some sleep-inducing or wakefulness-inducing cues, depending on which are available.

    You may be wondering: what about casein?

    Casein is oft-touted as producing deep sleep, or disturbed sleep, or vivid dreams, or bad dreams. There’s no science to back any of this up, though the following research review is fascinating:

    Dreams of the Rarebit Fiend: food and diet as instigators of bizarre and disturbing dreams

    (it largely supports the null hypothesis of “not a causal factor” but does look at the many more likely alternative explanations, ranging from associated actually casual factors (such as alcohol and caffeine) and placebo/nocebo effect)

    Finally, simple digestive issues may be the real thing at hand:

    Association between digestive symptoms and sleep disturbance: a cross-sectional community-based study

    Worth noting that around two thirds of all people, including those who regularly enjoy dairy products, have some degree of lactose intolerance:

    Lactose Intolerance in Adults: Biological Mechanism and Dietary Management

    So, in terms of what cheese may be better/worse for you in this context, you might try experimenting with lactose-free cheese, which will help you identify whether that was the issue!

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  • Prolonged Grief: A New Mental Disorder?

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    The issue is not whether certain mental conditions are real—they are. It is how we conceptualize them and what we think treating them requires.

    The latest edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) features a new diagnosis: prolonged grief disorder—used for those who, a year after a loss, still remain incapacitated by it. This addition follows more than a decade of debate. Supporters argued that the addition enables clinicians to provide much-needed help to those afflicted by what one might simply consider a too much of grief, whereas opponents insisted that one mustn’t unduly pathologize grief and reject an increasingly medicalized approach to a condition that they considered part of a normal process of dealing with loss—a process which in some simply takes longer than in others.    

    By including a condition in a professional classification system, we collectively recognize it as real. Recognizing hitherto unnamed conditions can help remove certain kinds of disadvantages. Miranda Fricker emphasizes this in her discussion of what she dubs hermeneutic injustice: a specific sort of epistemic injustice that affects persons in their capacity as knowers1. Creating terms like ‘post-natal depression’ and ‘sexual harassment’, Fricker argues, filled lacunae in the collectively available hermeneutic resources that existed where names for distinctive kinds of social experience should have been. The absence of such resources, Fricker holds, put those who suffered from such experiences at an epistemic disadvantage: they lacked the words to talk about them, understand them, and articulate how they were wronged. Simultaneously, such absences prevented wrong-doers from properly understanding and facing the harm they were inflicting—e.g. those who would ridicule or scold mothers of newborns for not being happier or those who would either actively engage in sexual harassment or (knowingly or not) support the societal structures that helped make it seem as if it was something women just had to put up with. 

    For Fricker, the hermeneutical disadvantage faced by those who suffer from an as-of-yet ill-understood and largely undiagnosed medical condition is not an epistemic injustice. Those so disadvantaged are not excluded from full participation in hermeneutic practices, or at least not through mechanisms of social coercion that arise due to some structural identity prejudice. They are not, in other words, hermeneutically marginalized, which for Fricker, is an essential characteristic of epistemic injustice. Instead, their situation is simply one of “circumstantial epistemic bad luck”2. Still, Fricker, too, can agree that providing labels for ill-understood conditions is valuable. Naming a condition helps raise awareness of it, makes it discursively available and, thus, a possible object of knowledge and understanding. This, in turn, can enable those afflicted by it to understand their experience and give those who care about them another way of nudging them into seeking help. 

    Surely, if adding prolonged grief disorder to the DSM-5 were merely a matter of recognizing the condition and of facilitating assistance, nobody should have any qualms with it. However, the addition also turns intense grief into a mental disorder—something for whose treatment insurance companies can be billed. With this, significant forces of interest enter the scene. The DSM-5, recall, is mainly consulted by psychiatrists. In contrast to talk-therapists like psychotherapists or psychoanalysts, psychiatrists constitute a highly medicalized profession, in which symptoms—clustered together as syndromes or disorders—are frequently taken to require drugs to treat them. Adding prolonged grief disorder thus heralds the advent of research into various drug-based grief therapies. Ellen Barry of the New York Times confirms this: “naltrexone, a drug used to help treat addiction,” she reports, “is currently in clinical trials as a form of grief therapy”, and we are likely to see a “competition for approval of medicines by the Food and Drug Administration.”3

    Adding diagnoses to the DSM-5 creates financial incentives for players in the pharmaceutical industry to develop drugs advertised as providing relief to those so diagnosed. Surely, for various conditions, providing drug-induced relief from severe symptoms is useful, even necessary to enable patients to return to normal levels of functioning. But while drugs may help suppress feelings associated with intense grief, they cannot remove the grief. If all mental illnesses were brain diseases, they might be removed by adhering to some drug regimen or other. Note, however, that ‘mental illness’ is a metaphor that carries the implicit suggestion that just like physical illnesses, mental afflictions, too, are curable by providing the right kind of physical treatment. Unsurprisingly, this metaphor is embraced by those who stand to massively benefit from what profits they may reap from selling a plethora of drugs to those diagnosed with any of what seems like an ever-increasing number of mental disorders. But metaphors have limits. Lou Marinoff, a proponent of philosophical counselling, puts the point aptly:

    Those who are dysfunctional by reason of physical illness entirely beyond their control—such as manic-depressives—are helped by medication. For handling that kind of problem, make your first stop a psychiatrist’s office. But if your problem is about identity or values or ethics, your worst bet is to let someone reify a mental illness and write a prescription. There is no pill that will make you find yourself, achieve your goals, or do the right thing.

    Much more could be said about the differences between psychotherapy, psychiatry, and the newcomer in the field: philosophical counselling. Interested readers may benefit from consulting Marinoff’s work. Written in a provocative, sometimes alarmist style, it is both entertaining and—if taken with a substantial grain of salt—frequently insightful. My own view is this: from Fricker’s work, we can extract reasons to side with the proponents of adding prolonged grief disorder to the DSM-5. Creating hermeneutic resources that allow us to help raise awareness, promote understanding, and facilitate assistance is commendable. If the addition achieves that, we should welcome it. And yet, one may indeed worry that practitioners are too eager to move from the recognition of a mental condition to the implementation of therapeutic interventions that are based on the assumption that such afflictions must be understood on the model of physical disease. The issue is not whether certain mental conditions are real—they are. It is how we conceptualize them and what we think treating them requires.

    No doubt, grief manifests physically. It is, however, not primarily a physical condition—let alone a brain disease. Grief is a distinctive mental condition. Apart from bouts of sadness, its symptoms typically include the loss of orientation or a sense of meaning. To overcome grief, we must come to terms with who we are or can be without the loved one’s physical presence in our life. We may need to reinvent ourselves, figure out how to be better again and whence to derive a new purpose. What is at stake is our sense of identity, our self-worth, and, ultimately, our happiness. Thinking that such issues are best addressed by popping pills puts us on a dangerous path, leading perhaps towards the kind of dystopian society Aldous Huxley imagined in his 1932 novel Brave New World. It does little to help us understand, let alone address, the moral and broader philosophical issues that trouble the bereaved and that lie at the root not just of prolonged grief but, arguably, of many so-called mental illnesses.

    Footnotes:

    1 For this and the following, cf. Fricker 2007, chapter 7.

    2 Fricker 2007: 152

    3 Barry 2022

    References:

    Barry, E. (2022). “How Long Should It Take to Grieve? Psychiatry Has Come Up With an Answer.” The New York Times, 03/18/2022, URL = https://www.nytimes.com/2022/03/18/health/prolonged-grief-
    disorder.html [last access: 04/05/2022])
    Fricker, M. (2007). Epistemic Injustice. Power & the Ethics of knowing. Oxford/New York: Oxford University Press.
    Huxley, A. (1932). Brave New World. New York: Harper Brothers.
    Marinoff, L. (1999). Plato, not Prozac! New York: HarperCollins Publishers.

    Professor Raja Rosenhagen is currently serving as Assistant Professor of Philosophy, Head of Department, and Associate Dean of Academic Affairs at Ashoka University. He earned his PhD in Philosophy from the University of Pittsburgh and has a broad range of philosophical interests (see here). He wrote this article a) because he was invited to do so and b) because he is currently nurturing a growing interest in philosophical counselling.

    This article is republished from OpenAxis under a Creative Commons license. Read the original article.

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  • “Skinny Fat” Explained (& How To Fix It)

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    “Skinny fat” is a term you may have seen floating around social media. It describes people who have a low body weight but a high body fat percentage, often resulting in flabby appearance despite being within a weight range considered healthy. Many try dieting and exercising, only to find that neither work.

    This video explains what’s going wrong, and how to fix it:

    Diet & exercise won’t work if it’s not right

    This problem occurs because common weight-loss approaches, such as restrictive dieting and excessive cardio, fail to improve body composition:

    • Restrictive dieting reduces both fat and lean mass, keeping the body fat percentage unchanged
    • Cardio burns some calories but the underlying metabolic issue hasn’t meaningfully changed, so any loss will be temporary (and most of any immediate loss will be water weight, anyway)

    The key to overcoming skinny fat is resistance training. Lifting weights or doing bodyweight exercises helps build muscle, which not only lowers body fat percentage (by simple mathematics; add more muscle and the percentages of other things must go down even if the total amount is the same) and improves overall definition, which is something most people consider nice. However, the real value here is that it actually addresses the underlying metabolic issue—because muscle costs calories to maintain, one’s basal metabolic rate will now be faster, even when you’re sleeping.

    This then becomes… Not quite a self-sustaining system, because you do have to still eat well and continue to do resistance training, but your body will be doing most of the work for you, and you’ll find it’s a lot easier to maintain a healthy body composition than to get one in the first place, for exactly the metabolic reason we described.

    For more on all of this, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like:

    Visceral Belly Fat & How To Lose It ← this is a different, but adjacent issue (and very important for avoiding metabolic disease risks)

    Take care!

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