Do We Simply Not Care About Old People?

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The covid-19 pandemic would be a wake-up call for America, advocates for the elderly predicted: incontrovertible proof that the nation wasn’t doing enough to care for vulnerable older adults.

The death toll was shocking, as were reports of chaos in nursing homes and seniors suffering from isolation, depression, untreated illness, and neglect. Around 900,000 older adults have died of covid-19 to date, accounting for 3 of every 4 Americans who have perished in the pandemic.

But decisive actions that advocates had hoped for haven’t materialized. Today, most people — and government officials — appear to accept covid as a part of ordinary life. Many seniors at high risk aren’t getting antiviral therapies for covid, and most older adults in nursing homes aren’t getting updated vaccines. Efforts to strengthen care quality in nursing homes and assisted living centers have stalled amid debate over costs and the availability of staff. And only a small percentage of people are masking or taking other precautions in public despite a new wave of covid, flu, and respiratory syncytial virus infections hospitalizing and killing seniors.

In the last week of 2023 and the first two weeks of 2024 alone, 4,810 people 65 and older lost their lives to covid — a group that would fill more than 10 large airliners — according to data provided by the CDC. But the alarm that would attend plane crashes is notably absent. (During the same period, the flu killed an additional 1,201 seniors, and RSV killed 126.)

“It boggles my mind that there isn’t more outrage,” said Alice Bonner, 66, senior adviser for aging at the Institute for Healthcare Improvement. “I’m at the point where I want to say, ‘What the heck? Why aren’t people responding and doing more for older adults?’”

It’s a good question. Do we simply not care?

I put this big-picture question, which rarely gets asked amid debates over budgets and policies, to health care professionals, researchers, and policymakers who are older themselves and have spent many years working in the aging field. Here are some of their responses.

The pandemic made things worse. Prejudice against older adults is nothing new, but “it feels more intense, more hostile” now than previously, said Karl Pillemer, 69, a professor of psychology and gerontology at Cornell University.

“I think the pandemic helped reinforce images of older people as sick, frail, and isolated — as people who aren’t like the rest of us,” he said. “And human nature being what it is, we tend to like people who are similar to us and be less well disposed to ‘the others.’”

“A lot of us felt isolated and threatened during the pandemic. It made us sit there and think, ‘What I really care about is protecting myself, my wife, my brother, my kids, and screw everybody else,’” said W. Andrew Achenbaum, 76, the author of nine books on aging and a professor emeritus at Texas Medical Center in Houston.

In an environment of “us against them,” where everybody wants to blame somebody, Achenbaum continued, “who’s expendable? Older people who aren’t seen as productive, who consume resources believed to be in short supply. It’s really hard to give old people their due when you’re terrified about your own existence.”

Although covid continues to circulate, disproportionately affecting older adults, “people now think the crisis is over, and we have a deep desire to return to normal,” said Edwin Walker, 67, who leads the Administration on Aging at the Department of Health and Human Services. He spoke as an individual, not a government representative.

The upshot is “we didn’t learn the lessons we should have,” and the ageism that surfaced during the pandemic hasn’t abated, he observed.

Ageism is pervasive. “Everyone loves their own parents. But as a society, we don’t value older adults or the people who care for them,” said Robert Kramer, 74, co-founder and strategic adviser at the National Investment Center for Seniors Housing & Care.

Kramer thinks boomers are reaping what they have sown. “We have chased youth and glorified youth. When you spend billions of dollars trying to stay young, look young, act young, you build in an automatic fear and prejudice of the opposite.”

Combine the fear of diminishment, decline, and death that can accompany growing older with the trauma and fear that arose during the pandemic, and “I think covid has pushed us back in whatever progress we were making in addressing the needs of our rapidly aging society. It has further stigmatized aging,” said John Rowe, 79, professor of health policy and aging at Columbia University’s Mailman School of Public Health.

“The message to older adults is: ‘Your time has passed, give up your seat at the table, stop consuming resources, fall in line,’” said Anne Montgomery, 65, a health policy expert at the National Committee to Preserve Social Security and Medicare. She believes, however, that baby boomers can “rewrite and flip that script if we want to and if we work to change systems that embody the values of a deeply ageist society.”

Integration, not separation, is needed. The best way to overcome stigma is “to get to know the people you are stigmatizing,” said G. Allen Power, 70, a geriatrician and the chair in aging and dementia innovation at the Schlegel-University of Waterloo Research Institute for Aging in Canada. “But we separate ourselves from older people so we don’t have to think about our own aging and our own mortality.”

The solution: “We have to find ways to better integrate older adults in the community as opposed to moving them to campuses where they are apart from the rest of us,” Power said. “We need to stop seeing older people only through the lens of what services they might need and think instead of all they have to offer society.”

That point is a core precept of the National Academy of Medicine’s 2022 report Global Roadmap for Healthy Longevity. Older people are a “natural resource” who “make substantial contributions to their families and communities,” the report’s authors write in introducing their findings.

Those contributions include financial support to families, caregiving assistance, volunteering, and ongoing participation in the workforce, among other things.

“When older people thrive, all people thrive,” the report concludes.

Future generations will get their turn. That’s a message Kramer conveys in classes he teaches at the University of Southern California, Cornell, and other institutions. “You have far more at stake in changing the way we approach aging than I do,” he tells his students. “You are far more likely, statistically, to live past 100 than I am. If you don’t change society’s attitudes about aging, you will be condemned to lead the last third of your life in social, economic, and cultural irrelevance.”

As for himself and the baby boom generation, Kramer thinks it’s “too late” to effect the meaningful changes he hopes the future will bring.

“I suspect things for people in my generation could get a lot worse in the years ahead,” Pillemer said. “People are greatly underestimating what the cost of caring for the older population is going to be over the next 10 to 20 years, and I think that’s going to cause increased conflict.”

KFF Health News is a national newsroom that produces in-depth journalism about health issues and is one of the core operating programs at KFF—an independent source of health policy research, polling, and journalism. Learn more about KFF.

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  • GLP-1 Drugs Delay Alcohol’s Effects!

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    GLP-1 drugs were designed as antidiabetic drugs, and took over the market as weight loss drugs.

    Their usefulness to reduce cravings has been noted to also reduce non-food cravings, including for some addictive substances, and some compulsive behaviors.

    See: Can a drug like Ozempic help treat addictions to alcohol, opioids or other substances?

    But, there’s more to it than that…

    It’s not just about drinking less

    Researchers (Dr. Alexandra DiFeliceantonio et al.) have found that GLP-1 agonists such as semaglutide, tirzepatide, and liraglutide slow the rate at which alcohol enters the bloodstream, resulting in delayed (and weaker) intoxicating effects.

    What they tested: in a randomized controlled trial, all participants fasted, ate a standardized snack, then consumed an alcoholic drink within 10 minutes. Breath alcohol, glucose, blood pressure, and pulse were then measured repeatedly over the next four hours.

    What they found is that those on GLP-1 drugs had slower increases in breath alcohol concentration and consistently reported feeling less intoxicated than those not taking such.

    How it works: the current hypothesis is that GLP-1 drugs likely reduce alcohol’s effects by slowing gastric emptying, delaying alcohol absorption, rather than directly affecting the brain. Because alcohol will then still be processed by the liver, it simply means the liver can process it little by little.

    This is important, because it means that (so far as the data so far can tell us) it doesn’t run into the same problem as occurs when people take cannabis edibles, think “hmm, I don’t feel it”, and then take more, and then end up overdosing, because everything was just delayed batch-by-batch, rather than slowed down in a continuous process.

    You can find the paper itself here: A preliminary study of the physiological and perceptual effects of GLP-1 receptor agonists during alcohol consumption in people with obesity

    You may be thinking: “with obesity? Isn’t that protective against alcohol’s effects?”, and the answer is that in the case of adiposity (as opposed to being muscular) there’s a mixed effect that cancels itself out rather; yes, alcohol has a per-kg effect, but a kg of muscle is a lot more helpful metabolically than a kg of fat, which is in most cases more of a metabolic problem than a solution. Still, it cannot be said with certainty that the conclusions will applicable to all people of all body types; more research will be needed to make a definitive declaration about that.

    GLP-1 drugs can protect the liver in one more way, too

    It’s also known that GLP-1 drugs lower liver levels of an enzyme known by the snappy name of Cyp2e1, which normally breaks alcohol down into acetaldehyde, the highly toxic metabolite responsible for much of alcohol’s liver damage.

    You can read more about this, here: GLP-1 receptor agonism results in reduction in hepatic ethanol metabolism

    Want to learn more?

    Here’s an unusually balanced overview of GLP-1 drugs when it comes to many aspects of life, rather than providing a glowing report or a terrible condemnation:

    Magic Pill – by Johann Hari

    And if GLP-1 drugs aren’t your thing, then we cover some other approaches for those who wish to drink alcohol and minimize its harmful effects:

    How To Make Drinking Less Harmful ← our main feature on such

    Take care!

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  • Cherries vs Elderberries – Which is Healthier?

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    Our Verdict

    When comparing cherries to elderberries, we picked the elderberries.

    Why?

    Both are great! But putting them head-to-head…

    In terms of macros, cherries have slightly more protein (but we are talking miniscule numbers here, 0.34mg/100g), while elderberries have moderately more carbs and more than 4x the fiber. This carbs:fiber ratio difference means that elderberries have the lower glycemic index by far, as well as simply more grams/100g fiber, making this an easy win for elderberries.

    In the category of vitamins, cherries have more of vitamins A, B9, E, K, and choline, while elderberries have more of vitamins B1, B2, B3, B6, and C. The margins of difference mean that elderberries have the very slightly better overall vitamin coverage, but it’s so slight that we’ll call this a 5:5 tie.

    When it comes to minerals, cherries have more copper, magnesium, and manganese, while elderberries have more calcium, iron, phosphorus, potassium, selenium, and zinc. A nice easy win to top it off for elderberries.

    On the polyphenols (and other phytochemicals) front, both are great in different ways, nothing that’d we’d consider truly sets one ahead of the other.

    All in all, adding up the sections, an overall win for elderberries, but by all means enjoy either or both!

    Want to learn more?

    You might like to read:

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  • 3 Ways To Increase Your Push-Ups (In Just 30-Days!)

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    Cori Lefkowitz, of “Strong at Every Age”, shows us how:

    Pushing it up

    A lot of people who struggle with push-ups will do make-it-easier modifications; doing them one one’s knees is a popular one, for example. However, more reps of a modified push-up only makes you stronger at that modification, not at the full push-up.

    So, how to get around this problem?

    Three ways:

    1. Cluster sets: do 3–5 rounds at the start of your workout; set a target of 6–10 total reps per round, and do 1–3 reps of the hardest variation you can, resting 15–30 seconds between mini-sets until the round is complete (rest for at least a minute between rounds).
    2. Slow eccentric push-ups: for 3–5 seconds, focus only on lowering yourself down, then reset at the top. This lets you train harder variations and build control even if you can’t push back up yet.
    3. Push-up holds: hold the push-up at weak points (e.g. bottom, halfway, or top—whatever it is for you) to build slow-twitch tension and improve your form (so that you no longer find yourself wobbly). This helps develop mind–muscle connection, which in turn helps pretty much all other parts of this endeavor.

    For an extra upwards push, you can combine these three ways with incline push-ups. As a very strong general rule, it’s almost always better to push towards harder variations rather than higher reps of the same easier version.

    Why “almost always”? Well, if you’re doing some push-up challenge and specifically want to do very many reps for the sake of it, then building rep count will be what you want. But for anything that’s not “high reps for the sake high reps”, the above method will stand you in better stead.

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like:

    How To Get Your First Pull-Up

    Take care!

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  • Elderhood – by Dr. Louise Aronson

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    Where does “middle age” end, and “old age” begin? By the United States’ CDC’s categorization, human life involves:

    • 17 stages of childhood, deemed 0–18
    • 5 stages of adulthood, deemed 18–60
    • 1 stage of elderhood, deemed 60+

    Isn’t there something missing here? Do we just fall off some sort of conveyor belt on our sixtieth birthdays, into one big bucket marked “old”?

    Yesterday you were 59 and enjoying your middle age; today you have, apparently, the same medical factors and care needs as a 114-year-old.

    Dr. Louise Aronson, a geriatrician, notes however that medical science tends to underestimate the differences found in more advanced old age, and underresearch them. That elders consume half of a country’s medicines, but are not required to be included in clinical trials. That side effects not only are often different than for younger adults, but also can cause symptoms that are then dismissed as “Oh she’s just old”.

    She explores, mostly through personal career anecdotes, the well-intentioned disregard that is frequently given by the medical profession, and—importantly—how we might overcome that, as individuals and as a society.

    Bottom line: if you are over the age of 60, love someone over the age of 60, this is a book for you. Similarly if you and/or they plan to live past the age of 60, this is also a book for you.

    Click here to check out Elderhood, and empower yours!

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  • Ticks Are Migrating, Raising Disease Risks if They Can’t Be Tracked Quickly Enough

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    Biologist Grant Hokit came to this small meadow in the mountains outside Condon, Montana, to look for ticks. A hiking path crossed the expanse of long grasses and berry bushes.

    As Hokit walked the path, he carried a handmade tool made of plastic pipes taped together to hold a large rectangle of white flannel cloth.

    He poked fun at this “sophisticated” device, but the scientific survey was quite serious: He was sweeping the cloth over the shrubs and grass, hoping that “questing” ticks would latch on.

    Along the summer trail, ticks dangle from blades of grass, sticking their legs out and waiting for a passing mammal.

    “We got one,” Hokit said.

    “So that came off of this sedge grass right here,” he said. “Simply pick them off with our fingers. We’ve got a vial that we pop them in.”

    Any captured ticks would go back to Hokit’s lab in Helena for identification. Most of them would probably be identified as Rocky Mountain wood ticks.

    But Hokit also wanted to find out whether new species are making their way into the state.

    As human-driven climate change makes winters shorter, ticks are spending less time hibernating and have more active months when they can hitch rides on animals and people. Sometimes the ticks carry themselves — and diseases — to new parts of the country.

    Hokit found deer ticks for the first time in northeastern Montana earlier this year. Deer ticks are infamous for transmitting Lyme disease and can infect people with other pathogens.

    Knowing a new species like the deer tick has arrived in Montana or other states is important for doctors.

    Neil Ku is an infectious disease specialist at the Billings Clinic in eastern Montana. He said most patients don’t come in right after they get bitten by a tick. They usually show up later, when they start feeling sick from a tick-borne illness.

    “Fever, some chills, they may just feel bad, similar to many infections we may encounter throughout the year,” he said.

    It’s rare that patients connect a tick bite to those symptoms, and even more rare that they capture and keep the tick that bit them. Sorting out whether someone might have a tick-borne illness can be complicated.

    Knowing what kinds of ticks are in the region will help doctors know that they might start encountering patients infected with new diseases after a tick bite, Ku said.

    That’s partially why the state is on the hunt for new tick species.

    “The more we know about what’s in Montana, the better we can inform our physicians, the better care you can receive,” said Devon Cozart, a zoonotic illness and vector-borne disease epidemiologist with the Montana Department of Public Health and Human Services.

    Cozart collects and tests the ticks from field surveys in Montana to see whether they are carrying any pathogens.

    Whether a tick can get a human sick depends on the species, but the kind of mammal it feeds on also plays a role.

    “Usually it’s a rodent that might be carrying, for example, Rocky Mountain spotted fever,” she said. “So, the tick will feed on that rodent, then will get the pathogen as well.”

    Because the prevalence of a particular disease can vary in mammal populations, ticks in one part of the state could be more or less likely to get you sick. That’s also important information for medical providers, Cozart said.

    This kind of surveillance and testing isn’t happening in every county or state. A 2023 survey of nearly 500 health departments throughout the country found that roughly a quarter do some kind of tick surveillance.

    Not all surveillance efforts are equal, said Chelsea Gridley-Smith, director of environmental health at the National Association of City and County Health Officials.

    Field surveys can be expensive. For numerous local and state health departments, tick surveillance relies on a less expensive, more passive approach: Concerned patients, veterinarians, and doctors must collect and send in ticks for identification.

    “It does provide a little information about what ticks are actually interacting with people and animals, but it doesn’t get into the weeds of how common ticks are in that area and how often do those ticks carry pathogens,” Gridley-Smith said.

    She said more health departments want to start tick surveillance, but getting funding is hard — and might get harder as federal public health grants from agencies like the Centers for Disease Control and Prevention dry up.

    Montana receives about $60,000 from a federal grant annually, but the bulk of that funding goes toward mosquito surveillance, which is more intensive and costly. What’s left funds trips into the field to look for ticks.

    Hokit said he doesn’t have enough funding for his small team to survey everywhere he would like to in a state as large as Montana. That means he’s unable to monitor emerging populations of deer ticks as closely as he would like.

    He found those new deer ticks in two Montana counties, but he doesn’t have enough data to determine whether they have begun reproducing there, establishing a local population.

    In the meantime, Hokit uses data on climate and vegetation to make predictions about where deer ticks might thrive in the state. He has his eye on particular areas of western Montana, like the Flathead Valley.

    He said that will help him and his team narrow down where to look next so they can let the public know when deer ticks — and the diseases they can carry — arrive.

    This article is part of a partnership with NPR and Montana Public Radio

    KFF Health News is a national newsroom that produces in-depth journalism about health issues and is one of the core operating programs at KFF—an independent source of health policy research, polling, and journalism. Learn more about KFF.

    Subscribe to KFF Health News’ free Morning Briefing.

    This article first appeared on KFF Health News and is republished here under a Creative Commons Attribution-NoDerivatives 4.0 International License.

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  • How Your Emotions Affect GLP-1 Drug Results!

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    GLP-1 receptor agonist drugs (such as Ozempic, Wegovy, Mounjaro, and others) have a good reputation for working well, and the most talked-about downside is that they often have unpleasant side effects:

    Most People Who Start GLP-1 RAs Quit Them Within A Year (Here’s Why) ← there are 4 main reasons

    …and of course, some do work better than others: Better Than Ozempic?

    As with almost any drug, some people are simply “non-responders”, meaning that for some reason (often a genetic factor, often not known for sure why), the drug will simply not work as it does for most people.

    For GLP-1 receptor agonists, there is a portion of the general population for whom they simply will not work, and so far there is no known way of predicting it (probably at someone point it’ll be figured out, and this writer’s money would be on it being either a SNP mutation or a microbiome thing). So, you roll the dice, you take the GLP-1 drug, you wait and see, and there’s a 15% chance (that doesn’t sound like a lot, but it’s about 1 in 6, in other words, the same probability as rolling a “1” on a fair, six-sided die).

    You can read about how that can go, here: Ozempic didn’t work for me. I was furious—and ashamed

    But why?

    The key is in why you are overeating in the first place (and if you’re looking to lose weight and/but are not overeating, then probably GLP-1 drugs are not for you, since that is primarily how they work).

    In few words:

    • If you are overeating in response to the sight and/or smell of tasty food, then probably you will benefit well from GLP-1 RAs in the long-term
    • If you are overeating for emotional reasons (e.g. because of depression, or as a coping strategy to deal with stress/anxiety, for example) then probably GLP-1 RAs cannot be replied on to help you.

    By “cannot be relied on” does not mean you will necessarily be a “non-responder” as described above, but it does mean that it’s likely your results will be intermittent at best. Which, after all, is not a big improvement on regular yo-yo dieting, an approach that is famously Not Good™.

    Indeed, per the categories in the study we’re about to cite:

    • Emotional eaters (eating due to negative feelings, not hunger) responded best to glp-1 drugs, showing greater weight loss and better blood sugar improvements
    • External eaters (eating because food looks or smells appealing) were less likely to benefit in the long term
    • Restrained eaters (deliberately restricting diet to lose weight) exercised more restraint temporarily, but returned to baseline by 12 months

    Notably, all three categories of eaters here were people with type 2 diabetes—in other words, the very people that GLP-1 drugs were first developed to help, before they took off as weight-loss drugs.

    So in theory, these should be the people for whom GLP-1 RAs work best—and yet, as we see, it’s still not always so, and is highly dependent on what goes on between one’s ears.

    You can read this paper in full, here: Association between eating behavior patterns and the therapeutic efficacy of GLP-1 receptor agonists in individuals with type 2 diabetes: a multicenter prospective observational study

    Want a different approach?

    It is possible to get many of the effects of GLP-1 RAs without taking GLP-1 RAs, by enjoying foods that increase incretin, a hormone group (the most well-known of which is GLP-1) that slows down stomach emptying, which means a gentler blood sugar curve and feeling fuller for longer. It also acts on the hypothalamus, controlling appetite via the brain too (signalling fullness and reducing hunger).

    For what foods to focus on, see: 5 Ways To Naturally Boost The “Ozempic Effect” ← this is from Dr. Jason Fung, who is perhaps most well-known for his work in functional medicine for reversing diabetes, and he’s once again giving us sound advice about metabolic hormone-hacking with dietary tweaks!

    Or to curb emotional eating specifically, check out: Emotional Eating And The Five Pillars Of Craving Control

    Or for a deeper dive, you might like this book we reviewed not long back:

    Breaking Free from Emotional Eating – by Geneen Roth

    Take care!

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