Apricot vs Kiwi – Which is Healthier?

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Our Verdict

When comparing apricots to kiwi, we picked the kiwi.

Why?

Both are great! But…

In terms of macros, kiwi has more fiber and carbs, as well as the better ratio of fiber:carbs, and so wins this round.

In the category of vitamins, apricot has more of vitamins A, B1, B2, B3, and B5, while kiwi has more of vitamins B6, B7, B9, C, E, K, and choline. Yes, it’s a little unusual the distribution of vitamins were divided alphanumerically like that (from A to B5 for apricots, then B6 onwards for kiwi, as though they were neighboring sections in a library), but nature can be strange like that sometimes. It’s also fun apricots are a really good source of vitamin A (24x more) and kiwis are a really good source of vitamin K (12x more), but when all the vitamins are added up, kiwi has a clear numerical advantage over all, and wins this round.

When it comes to minerals, things are no longer distributed alphabetically, because apricots have more iron and zinc, while kiwis have more calcium, copper, magnesium, manganese, phosphorus, potassium, and selenium. Another win for kiwis.

Both of these fruits have abundant polyphenols and specific anticancer properties, so there’s nothing between them here in that regard.

Adding up the sections makes for a clear overall win for kiwis, but by all means enjoy either or both; diversity is good!

Want to learn more?

You might like:

Top 8 Fruits That Prevent & Kill Cancer

Enjoy!

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  • Build Your Neck Mobility

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    The neck (so: the cervical spine) is, when healthy, the most mobile part of the spine.

    It’s essential for everyday function and overhead lifts, and yet it’s often neglected in physical training, despite being strained by forward-facing habits like screen time and driving.

    Here’s how to make it better:

    It’s time to raise the bar

    Literally, you will need a bar. You don’t have to get sportier about it than you want to; a broom handle is equally good!

    For each of the exercises in this routine, maintaining rib-hip connection and shoulder stability is important:

    1. Chin retraction: place a stick at the base of your skull (occipital lobe) and pull the chin straight back (not downwards) to create a double chin. Maintain a neutral spine, keep your rib cage stable, and repeat.
    2. Neck flexion: from the retracted position, slowly curl your chin downward towards your chest—moving only the neck, not the mid-back. Maintain retraction and stable ribs. Repeat, deepening the range each time.
    3. Neck extension: starting in retraction, slowly tilt your head backwards so that it moves behind the body without arching your spine or crunching your neck. Return to neutral and repeat.
    4. Neck rotation: with the stick behind your occipital lobe, maintain chin retraction while applying isometric tension. Rotate your head to one side, keeping the back of your head pressed into the stick. Return to center and repeat to the other side.
    5. Neck lateral flexion: put the stick across your shoulders to stabilize them. Retract your chin, then slowly drop one ear toward the same-side shoulder. Isolate your neck—do not let your shoulders move. Hold briefly, return to center, and repeat on both sides.
    6. Partial neck CARs*: begin with lateral flexion (ear to shoulder), then rotate your chin towards that shoulder, return to lateral flexion, and come back to the center. Repeat on the other side.
    7. Neck CARs with flexion: begin in lateral flexion, rotate your chin towards the shoulder, and trace your collarbone across into full neck flexion. End with lateral flexion and return to upright. Reverse the motion to complete one rep.
    8. Neck CARs with extension: from lateral flexion, draw a semicircle with your chin outward and around into extension. Pass through the opposite side’s lateral flexion, rotate your chin towards the shoulder, and return to the start. Reverse for a complete rep.
    9. Full neck CARs: combine flexion, rotation, lateral flexion, and extension into a slow, smooth chin circle. Keep the stick across your shoulders and the rest of your spine still.

    *CARs = controlled articular rotations

    For more on all of this plus visual demonstrations, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like:

    3 Unconventional Ways To Fix Your Stiff Neck (Without Stretching) ← these ones are because often, your neck is not the actual problem

    Take care!

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  • Artichoke vs Green Beans – Which is Healthier?

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Our Verdict

    When comparing artichoke to green beans, we picked the artichoke.

    Why?

    In terms of macros, artichoke has nearly 2x the fiber, nearly 2x the protein, and slightly more carbs, making it to “more food per food” option in this category easily.

    In the category of vitamins, artichokes have more of vitamins B3, B5, B6, B7, and B9, while green beans have more of vitamins A, B1, B2, E, and K, for a 5:5 tie in this round.

    Looking at minerals, artichokes have more copper, iron, magnesium, phosphorus, potassium, and zinc, while green beans have more manganese and selenium, yielding to artichoke a 6:2 win here.

    In other considerations, artichokes are also higher in polyphenols, so that’s another round in their favor.

    Adding up the sections makes for a clear overall win for artichokes, but by all means enjoy either or both, as diversity is best!

    Want to learn more?

    You might like:

    Level-Up Your Fiber Intake! (Without Difficulty Or Discomfort)

    Enjoy!

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  • Norepinephrine vs Alzheimer’s Disease

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Norepinephrine (or noradrenaline, in the rest of the world outside of the US), is a hormone and neurotransmitter generally associated with stress, fight-or-flight responses, and hypertension.

    Like any of our hormones, it has its place, and we wouldn’t do well without it (same deal for cortisol, which has a very bad reputation, but again, we do need it or else we would not, for example, wake up in the morning).

    When it comes to neurochemistry, a shortage of norepinephrine can result in lethargic listlessness, which is why some antidepressants work on the noradrenergic* system (as opposed to the more common SSRIs, which work on the serotonergic system), to boost flagging norepinephrine levels and perk us** up.

    *Yes, it’s called that even in the US where the hormone/neurotransmitter itself is called norepinephrine rather than noradrenaline.

    **this article brought to you by the power of this writer taking mirtazapine, a selective norepinephrine reuptake inhibitor (SNRI) antidepressant, that thus increases the amount of available norepinephrine in her brain.

    So… How does it protect against Alzheimer’s disease?

    Dialing down the brain’s immune system

    Because of the blood-brain barrier, there are many things that happen either only inside of, or only outside of, our brain—which because of the unique nature of the brain’s anatomy, means that we often have a specialized system doing the same job inside the brain as a different system does outside of the brain, but in a different way.

    See for example how the glymphatic system (a portmanteau of glial cells and lymphatic system) in the brain does approximately the same job as the lymphatic system does in the rest of the body:

    How To Clean Your Brain (Glymphatic Health Primer) ← this helps protect us against Alzheimer’s, Parkinson’s, and other neurodegenerative conditions

    And those glial cells? Some of them do the job otherwise done by parts of our immune system that can’t operate inside our brain.

    Specifically, microglia do approximately the same job inside our brain as macrophages do outside of it: “eating” things that shouldn’t be there—ranging from actual invading pathogens, to bits of debris that are also in the way.

    That our brain has an immune response is, generally speaking, a good thing. But much like the immune system in the rest of our body, things can get out of hand.

    As with how chronic inflammation (and/or autoimmune disorders) causes problems in the rest of the body, neuroinflammation can cause problems in the brain—not least of all: it can lead to Alzheimer’s.

    The microglia are involved in the cleanup of the β-amyloid proteins that can otherwise build up into harmful plaque resulting in neuronal damage and with it, neurodegeneration), so calming them down a bit means they can do their actual assigned job better for longer.

    Dr. Ania Majewska et al. did a study into how norepinephrine’s inhibitory effect via β2 adrenergic receptors (β2AR) in microglia has an anti-inflammatory effect, and found that it has potential as an Alzheimer’s preventative.

    In their words, “β2AR manipulations can alter disease pathology”, which is a great example of how carefully scientists say things, but the series of declarations adds up to the same; we’ll quote some points directly from the paper’s abstract:

    • NE inhibits surveillance activity of microglia, the brain’s resident immune cells, via their β2 adrenergic receptors (β2ARs)
    • Microglial β2AR signaling is an important modulator of amyloid pathology.
    • Endogenous β2AR signaling degenerates as a function of amyloid pathology and aging.
    • In AD, microglia downregulate β2AR expression early and progressively.
    • β2AR manipulations can alter disease pathology.
    • Importantly, dampening microglial β2AR signaling worsened plaque load and the associated neuritic damage, while stimulating microglial β2AR signaling attenuated amyloid pathology.
    • Our results suggest that microglial β2AR could be explored as a potential therapeutic target to modify AD pathology.

    Translating from sciencese (if you’ll pardon that we’ll still use some big words, but only ones we’ve already explained):

    Norepinephrine activates certain receptors in microglia, and those receptors tell the microglia to “keep calm & carry on”. In the case of Alzheimer’s disease, those receptors stop working correctly, leading to increased neuroinflammation. Thus, stimulating those receptors with norepinephrine reduces neuroinflammation, allowing the microglia to calmly carry on with their actual job of getting rid of the amyloid that leads to Alzheimer’s disease.

    You can read the paper itself here:

    Noradrenergic signaling controls Alzheimer’s disease pathology via activation of microglial β2 adrenergic receptors

    Want to learn more?

    Check out:

    Take care!

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  • Parents find Health Star Ratings confusing and unhelpful. We need a better food labelling system

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    Food labels are intended to support healthy choices. But not all labelling schemes are equal.

    Australia currently uses a voluntary Health Star Rating system. Food manufacturers can choose to add a star label to their packaging to indicate how it compares to other similar products. Or they can choose not to show a star rating on a product at all.

    The Australian government is now considering making it mandatory.

    But our new research on parenting and food in Australia found the Health Star Ratings are often confusing, misunderstood and have little credibility among shoppers.

    If Health Stars are mandated, the system will also need a major overhaul to be trusted and useful for shoppers.

    Gustavo Fring/Pexels

    How do Health Star Ratings work?

    The government set up the front-of-pack Health Star Rating system in 2014 in collaboration with the food industry, public health and consumer groups.

    Product ratings range from (bad) ½ to (good) 5 stars.

    Calories, saturated fat, sugars and sodium decrease the rating. Fibre, protein, and the content of fruit, vegetables, nuts and legumes increase it.

    The good and bad offset each other. This means companies can strategically formulate products to boost the rating and mask unhealthy ingredients.

    Processing and additives – such as sweeteners, colouring, emulsifiers, preservatives and artificial flavourings – are not part of the calculation.

    Previous research has found the ratings can incentivise ultra-processed foods over minimally and unprocessed foods, and misrepresent healthfulness. Some researchers have also suggested practical ways to modify the rating algorithm to account for processing.

    The Health Star Rating’s own consumer research found 74% of consumers do not understand that the rating cannot be used to compare dissimilar products.

    What parents told us

    In our interviews with 34 parents in Australia, participants often described the Health Star Ratings as “misleading”, “not helpful” and “on the wrong product”. One participant called it the “fake health star rating”.

    They gave many examples:

    Like you might buy 100% orange juice or fruit juice and it might have only half a star health star rating, but then you can buy like a box of processed muesli bars and it will have five stars. – Mother of three high school aged children, urban WA

    Coco Pops or Nutrigrain have three and a half star rating, and what exactly does that mean? – Mother of one primary school aged child, urban WA

    Participants wondered if the Health Stars were something companies paid for, a “marketing thing”.

    Positivity bias

    Part of the problem with the Health Stars is the positivity bias of the symbol. As one participant put it, “All stars are good. Right?”

    Another noted their children comment on the stars, saying “but look Mum, it’s five stars.”

    However, parents were not convinced:

    A lot of packaged stuff is rated as five stars. I’m like yeah, well, don’t know about that. It’s still packaged. – Mother of two primary school aged children, urban NSW

    Participants thought discretionary foods should not have any stars. As one participant said:

    The other day, we saw a mud cake and it has a two out of five star health rating. How can that be a two out of five star?… Like there should not even be a star available for this. – Mother of pre-school aged child, urban NSW

    Burden on parents

    Parents often disregarded the rating. For example:

    This particular thing, you know, had all sorts of additives, had actually had a much higher rating than something that actually didn’t have any additives… what I ended up buying was rated slightly lower. – Mother of two primary school aged children, rural Victoria

    Instead participants used ingredients lists, apps such as Yuka, and “hours of internet research” to guide healthier choices.

    But there was a sense of frustration that the burden was on them. Participants said:

    I feel like food labels are extremely deceptive and by producers, purposely confusing. – Mother of one primary school aged child, urban SA

    It has to be government driven because companies won’t change unless they’re forced to by the government. – Father of two primary school aged children, urban Tasmania

    We need a food labelling system that works

    Still, the parents we spoke to think a front-of-pack system is valuable. As one participant explained:

    I do think if I had a better system for that, that would get a lot of use. – Mother of two primary school aged children, urban NSW

    Parents repeatedly stated a desire for transparency over food, for information they can trust and food policies that prioritise consumer health.

    As one mother put it, the “multi-billion dollar” food industry will not do this on their own, and “that’s where the government needs to step in.”

    If Health Stars are mandatory, how could labelling be overhauled?

    Chile, Mexico, Brazil and other countries, including Canada from 2026, are now using “stop-sign” warnings to steer consumers away from the least healthy products. Large Black Octagons alert consumers to high sugar, sodium and saturated fats, and ultra-processing.

    New Canadian food labelling system
    Starting in 2026, a new front-of-package symbol will be required on many Canadian foods and drinks that are high in saturated fat, sugars or salt. Canada.ca/en/health

    Evidence shows these warning labels have improved nutrition and public health in other countries and could be an option for Australia.

    We need to mandate a fit-for-purpose food labelling system that supports healthy eating. Governments should centre the voices of consumers in these and other national food policies to ensure they work as intended.

    Juliet Bennett, Postdoctoral Research Fellow, University of Sydney; Alex Broom, Professor of Sociology & Director, Sydney Centre for Healthy Societies, University of Sydney, and David Raubenheimer, Leonard P. Ullman Chair in Nutritional Ecology, Nutrition Theme Leader Charles Perkins Centre, Chair Sydney Food and Nutrition Network, University of Sydney

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • You Are the One You’ve Been Waiting For – by Dr. Richard Schwartz

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    As self-therapy approaches go, the title here could be read two ways: as pop-psychology fluff, or a suggestion of something deeper. And, while written in a way to make it accessible to all, we’re happy to report the content consists of serious therapeutic ideas, presented clearly.

    Internal Family Systems (IFS) is a large, internationally recognized, and popular therapeutic approach. It’s also an approach that lends itself quite well to self-therapy, as this book illustrates.

    Dr. Schwartz kicks off by explaining not IFS, but the problem that it solves… We (most of us, anyway) have over the course of our lives tried to plug the gaps in our own unmet psychological needs. And, that can cause resentment, strain, and can even be taken out on others if we’re not careful.

    The real meat of the book, however, is in its illustrative explanations of how IFS works, and can be applied by an individual. The goal is to recognize all the parts that make us who we are, understand what they need in order to be at peace, and give them that. Spoiler: most what they will need is just being adequately heard, rather than locked in a box untended.

    One of the benefits of using this book for self-therapy, of course, is that it requires a lot less vulnerability with a third party.

    But, speaking of which, what of these intimate relationships the subtitle of the book referenced? Mostly the benefits to such come from a “put your own oxygen mask on first” angle… but the book does also cover discussions between intimate partners, and approaches to love, including what the author calls “courageous love”.

    Bottom line: this is a great book if you want to do some “spring-cleaning of the soul” and live a little more lightly as a result.

    Click here to check out “You Are The One You’ve Been Waiting For” on Amazon today, and try out IFS for yourself!

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  • GLP-1 RAs For Weight Loss (But How Much Of That Loss Is Muscle?)

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    First introduced as a diabetes medication, GLP-1 drugs quickly took hold for off-label use as weight loss aids, even when the science was still very young.

    Here’s one of our first articles on that, back in the day: Semaglutide’s Surprisingly Big Research Gap

    As for that popularity? Check out: 1 in 5 US Women Aged 50–64 Has Used GLP-1 RAs: What We’ve Learned

    Spoiler, one of the things we’ve learned is: Most People Who Start GLP-1 RAs Quit Them Within A Year (Here’s Why)

    One of the main things in their favor is, of course, that (for most people, anyway), they work (except when they don’t: Why Intermittent Fasting (& GLP-1 Drugs!) Might Not Work For You).

    But it seems that even that comes with a drawback of its own, and in this case, it’s a drawback that keeps on giving taking.

    Wrong weight loss!

    Recent research has shown that 25–40% of the weight lost on GLP-1 drugs comes from fat-free mass (mostly muscle), compared to only 8% per decade lost naturally with age.

    As we wrote about in an older article of ours:

    Of the four studies that actually looked at the macros (unlike most studies), they found that on average, protein intake decreased by 17.1%. Which is a big deal!

    It’s an especially big deal, because while protein’s obviously important for everyone, it’s especially important for anyone trying to lose weight, because muscle mass is a major factor in metabolic base rate—which in turn is much important for fat loss/maintenance than exercise, when it comes to how many calories we burn by simply existing.

    A reasonable hypothesis, therefore, is that one of the numerous reasons people who quit GLP-1 agonists immediately put fat back on, is because they probably lost muscle mass in amongst their weight loss, meaning that their metabolic base rate will have decreased, meaning that they end up more disposed to put on fat than before.❞

    Read in full: Semaglutide’s Surprisingly Big Research Gap ← our older article that we quoted above

    And now the very latest research (by Dr. Charlotte Suetta et al.) puts even more weight behind our hypothesis that we wrote about back in the day, and adds new numbers to it.

    All incretin-based drugs (GLP-1–related therapies) tested were associated with a higher proportion of muscle loss relative to total weight loss compared with placebo or lifestyle interventions.

    As for the numbers: the median proportion of weight loss from muscle-related tissue was 34.9%, with 68% of studies exceeding the 25% benchmark.

    Since, as we said, muscle plays a key role in metabolism, glucose regulation, energy expenditure, and immune function, this becomes quite dire, because its loss can keep on self-perpetuating down the line as metabolic health worsens.

    In the words of Dr. Suetta herself:

    Treatment success should not be defined by kilograms lost alone. This is particularly true in older adults and in patients with low muscle reserve or functional limitations.

    The question is no longer whether incretin-based therapies reduce body weight; the question now is whether we can ensure that the weight lost is predominantly fat while preserving the muscle needed for metabolic health, physical function and healthy aging.❞

    You can find her paper itself, here: Beyond Weight Loss: Preserving Muscle in the Era of Incretin Therapy

    As well as a systematic review that found the same: Effect of Incretin-Based and Nonpharmacologic Weight Loss on Body Composition: A Systematic Review

    And if you’d prefer to do better, then consider: The 5 Training Rules To Build Your Metabolism (Not Just Lose Weight)

    And if you like books, then we highly recommend: Strong: The Definitive Guide To Active Ageing – by Jacqueline HootonThe author, herself in her 60s, knows her stuff when it comes to fitness (female fitness in particular) and aging (or: ageing, as you’ll see in this book, with its British English).

    Want to learn more?

    You might also like this one that we reviewed a little while back:

    Magic Pill: The Extraordinary Benefits and Disturbing Risks of the New Weight-Loss Drugs – by Johann Hari

    Take care!

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