Almost half of antibiotic prescribing for surgery is inappropriate, new report shows

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Inappropriate antibiotic prescribing around the time of surgery and long-term prescribing in aged care are among a mixed bag of findings of a recent report into antibiotic use and resistance in Australia.

The report shows while fewer antibiotics are prescribed in the community than a decade ago, there is still room to improve antibiotic prescribing in hospitals.

We are both involved in antibiotic stewardship programs, primarily in hospitals, which aim to improve the use of antibiotics to improve patient care and reduce the potential for antimicrobial resistance.

Here’s why antibiotic resistance is so concerning and what the latest report tells us.

Why is antibiotic use and resistance important?

Factors driving antibiotic use tend to be different in hospitals and in the community.

In hospitals, there are more patients with infections, and these are also places where patients come to with resistant infections. Here, a common dilemma is making sure sick patients receive antibiotics quickly, balanced with not overusing them unnecessarily.

In the community, GPs often have to use careful clinical judgement to determine whether antibiotics are required, or if the patient will recover without them.

If we think of this issue at the level of individual patients, the risks may feel small. But at the population level, using the wrong antibiotic, or using it when it’s not needed, or for too long increases the risk of antibiotic resistance.

This is where bacteria become resistant to the usual treatment options, so infections may continue to progress despite treatment.

This occurs due to “selection pressure”. This means the bacteria acquire changes that enable them to evade the effect of antibiotics, and these resistant strains continue to grow and spread.

Why are antibiotics used in surgery?

Antibiotics are mostly used around the time of surgery as one way to prevent, rather than treat, an infection.

They are generally needed only for procedures where there is a higher risk of infection and for a short period (mostly a single dose before surgery or for up to 24 hours afterwards).

This report shows that just under half (42.7%) of antibiotic use for surgical procedures was not appropriate. The main areas that we need to work on are:

  • only using antibiotics for surgery where there is a high risk of infection
  • the time we administer the antibiotic dose, ideally within an hour before the skin is cut
  • the choice of antibiotic – sufficient to cover the organisms that could cause infection, but not unnecessarily broad that it may cause side effects or antibiotic resistance.

Inappropriate antibiotic use in surgery may have several consequences.

Giving the antibiotic at the wrong time (too early, or too late) reduces its effectiveness. Giving it for surgery where there is a low risk of infection, or for too long unnecessarily exposes patients to the risk of antibiotic side effects such as diarrhoea, as well as increasing the risk of antibiotic resistance.

How about aged-care facilities?

The report shows residents of aged-care homes receive high amounts of antibiotics.

Two striking statistics were that four in five residents (79.5%) received at least one antibiotic prescription each year. About one in three patients (34.7%) were given an antibiotic for more than six months.

Aged-care residents are at a higher risk of developing infections and it can sometimes be harder to spot the signs and symptoms of an infection.

So using antibiotics to prevent infection can sometimes be appropriate but should be a last resort. This is because infections that “break through” to cause infection despite preventative antibiotics are more likely to be resistant.

What else did the report find?

The report also included critical antimicrobial resistances. These microorganisms are a serious threat to some of our last-line antibiotics. These are very difficult to treat and require specialised antibiotics and medical care.

The reported number of these organisms more than doubled from 2022, to 3,389, or more than nine cases each day, in 2024.

The report also highlights that many of these organisms are acquired overseas, reinforcing the regional and global context of antibiotic resistance.

What can we do to reduce antibiotic resistance?

We’ve previously written about actions we can take to reduce antibiotic use. This latest report reinforces that we should:

  • raise awareness that many infections will get better by themselves, and don’t necessarily need antibiotics
  • for aged-care residents, regularly review medications, including antibiotics, and check if they are still needed
  • use the antibiotics we have more appropriately and for as short a time as possible, supported by appropriate oversight in hospitals, and at state and national levels
  • continue to monitor for infections due to resistant bacteria to inform control policies
  • reduce cross-transmission of resistant organisms in hospitals and in the community
  • prevent infections by other means, such as clean water, sanitation, hygiene and vaccines
  • continue to develop new antibiotics and alternatives to antibiotics, and ensure the right incentives are in place to encourage a continuous pipeline of new antibiotics.

The wider context

This report is only one part of the picture of how and where antibiotics are used in Australia.

We have previously estimated that around 60% of antibiotics in Australia are used in animals.

This issue was highlighted by recent use of the antibiotic florfenicol in Tasmanian salmon farms. This is closely related to chloramphenicol, an antibiotic used in humans.

This reinforces the need to take a co-ordinated strategy across different sectors, an approach that has worked before in Australia.

There would also be benefits from responding to antibiotic resistance in a similar way to how we respond to other public health threats. So bringing the national response into the Australian Centre for Disease Control, which was launched officially at the start of 2026, should strengthen our efforts.

Allen Cheng, Professor of Infectious Diseases, Monash University and Kelly Cairns, PhD Candidate studying antimicrobial resistance, Department of Infectious Diseases, The Alfred Hospital and School of Translational Medicine, Monash University

This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • Scars? How To Minimize & Heal Them

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    It’s Q&A Day at 10almonds!

    Have a question or a request? We love to hear from you!

    In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!

    As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!

    So, no question/request too big or small 😎

    ❝I had a surgery 7 months ago, and while everything is healed there’s more of a raised scar than I’d like, is there anything better than just moisturizes that will actually reduce it?❞

    First of all: congratulations on your presumably successful surgery, and your good healing since!

    Scar management/reduction is mostly a matter of helping your body to heal itself, except for the most extreme cases. We will assume yours is not one of the extreme cases, and as such we are discounting the possibility of a revisional surgery being on your to-do list, and will focus on gentler approaches.

    With that in mind…

    First, limit any further scarring

    By this we don’t mean “avoid future surgeries and injuries”, though of course, also do that if reasonably possible. But we do mean:

    Yes, moisturize first and moisturize often.

    The reason this is important is because scar tissue loses water faster than normal tissue, and tissue that is not hydrated properly cannot function properly and certainly cannot heal properly.

    As for what moisturizer is best for this: something that’s actually hydrating.

    So, put aside the coconut oil, the castor oil, or anything else like that. Instead, opt for a moisturizer that has:

    1. water as its main ingredient
    2. glycerin and urea high on the list after water

    As usual, we’re not just saying things; there has been research done about this, for example:

    Commonly recommended moisturising products: effect on transepidermal water loss and hydration in a scar model

    The top-scoring moisturizer in that study, by the way, was Eucerin Advanced Repair Cream ← you can get it on Amazon if you want some 😎

    Next, encourage your body to heal itself

    Your body is an incredibly efficient organism. It might not always feel like it, but it is!

    However, this efficiency can sometimes manifest as a sort of thriftiness, that is to say, your body usually won’t voluntarily do more than it thinks necessary; it has its own idea of what is “good enough”, and after that point, it won’t “waste” further resources on it unless you give it a reason to.

    We see this with muscle-building, for example. Your body will not put on more muscle in a given place unless you are exerting that muscle sufficiently that the body thinks “hmm, we need more muscle there”.

    When it comes to scar healing, the same principle applies. So far as the body is concerned, “we’ve closed the wound, you’re not bleeding, there is no further chance of infection, it doesn’t hurt, what more do you want from me?”

    So, it will be necessary to provoke the body into getting back into “healing things” mode.

    So, paradoxically, it will be necessary to create a perceived threat for the body to respond to. Options include:

    • Microneedling / dermarolling: these are basically the same thing, except that the former is automated and the latter is manual. It pokes many very tiny holes in the skin; the body detects this and shifts back into “healing this bit” mode. Here’s an example microneedling kit on Amazon, and here’s a dermaroller if you prefer a low-tech approach.
    • Phototherapy: includes red light therapy (RLT), which there’s a lot of science for in terms of how it promotes wound healing and stimulates collagen production; we’ve written about it before, here. However, while previously we recommended a RLT mask, that’s probably not a good option unless the scar is on your face, so you might consider a red light therapy mat, instead. That link’s for quite a deluxe one; if you want something smaller and cheaper, then perhaps this one. Laser therapy may also be an option (works exactly the same way as RLT, but is more localized and more intense), but for safety reasons that’s more of an in-salon thing, so you might want to check with local salons to see what’s available. Honestly, unless you want to throw a lot of money at salon sessions, an at-home RLT kit (even if springing for an expensive one) is the more economic choice for most people, as a “buy it once and then it’s yours” option, rather than the more ongoing “pay every time” situation of “give a salon a financial incentive to never quite finish treating you”.
    • Retinoids: these promote (to varying degrees, depending on which you opt for) localized inflammation, which in turn promotes more rapid skin cell turnover, and thus, more rapid skin repair. There are a variety of options here, so here’s a guide for choosing the one that’s best for you: Retinoids: Retinol vs Retinal vs Retinoic Acid vs..?
    • Topical vitamin C: if you use this and a retinoid, it’s usually recommended to use vitamin C in the morning and the retinoid in the evening, as they don’t go well together, and since retinoids temporarily increase photosensitivity, it’s best to use that one at night. If you use it alone, you might want to do it at night, for reasons covered amongst the professional tips shared in: Is Vitamin C Worth The Hype?
    • Collagen supplementation: assuming you’re not vegetarian/vegan, collagen supplementation may be worth considering. If you are vegetarian/vegan, don’t worry, your body can make its own collagen; you’ll just want to make sure you’re taking extra care to give your body plenty of the things it needs to do that, so check out: The Best Foods For Collagen Production

    You may be wondering about silicon sheets, and topical collagen:

    Take care, and happy healing!

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  • When Science Brings Hope

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    There’s a lot of bad news out there at present, including in the field of healthcare. So as some measure of respite from that, here’s some good news from the world of health science, including some actionable things to do:

    Run for your life! Or casually meander for your life; that’s fine too.

    Those who enjoy the equivalent of an average of 160mins slow (3mph) walking per day also enjoy the greatest healthspan. Now, there may be an element of two-way causality here (moving more means we live longer, but also, sometimes people move less because of having crippling disabilities, which are themselves not great for healthspan, as well as having the knock-on effect of reducing movement, and so such conditions yield and anti-longevity double-whammy), but for any who are able to, increasing the amount of time per day spend moving, ultimately results (on average) in a lot of extra days in life that we’ll then get to spend moving.

    Depending on how active or not you are already, every extra 1 hour walked could add two hours and 49 minutes to life expectancy:

    Read in full: Americans over 40 could live extra 5 years if they were all as active as top 25% of population, modeling study suggests

    Related: The Doctor Who Wants Us To Exercise Less & Move More

    Re-teaching your brain to heal itself

    Cancer is often difficult to treat, and brain tumors can be amongst the most difficult with which to contend. Not only is everything in there very delicate, but also it’s the hardest place in the body to get at—not just surgically, but even chemically, because of the blood-brain barrier. To make matters worse, brain tumors such as glioblastoma weaken the function of T-cells (whose job it is to eliminate the cancer) by prolonged exposure.

    Research has found a way to restore the responsiveness of these T-cells to immune checkpoint inhibitors, allowing them to go about their cancer-killing activities unimpeded:

    Read in full: New possibilities for treating intractable brain tumors unveiled

    Related: 5 Ways To Beat Cancer (And Other Diseases)

    Here’s to your good health!

    GLP-1 receptor agonists, originally developed to fight diabetes and now enjoying popularity as weight loss adjuvants, work in large part by cutting down food cravings by interfering with the chemical messaging about such.

    As a bonus, it seems that they also can reduce alcohol cravings, especially by targetting the brain’s reward center; this was based on a large review of studies looking at how GLP-1RA use affects alcohol use, alcohol-related health problems, hospital visits, and brain reactions to alcohol cues:

    Read in full: Diabetes medication may be effective in helping people drink less alcohol, research finds

    Related: How To Reduce Or Quit Alcohol

    Take care!

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  • Marathons in Mid- and Later-Life

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    It’s Q&A Day at 10almonds!

    Have a question or a request? You can always hit “reply” to any of our emails, or use the feedback widget at the bottom!

    In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!

    As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!

    So, no question/request too big or small

    We had several requests pertaining to veganism, meatless mondays, and substitutions in recipes—so we’re going to cover those on a different day!

    As for questions we’re answering today…

    Q: Is there any data on immediate and long term effects of running marathons in one’s forties?

    An interesting and very specific question! We didn’t find an overabundance of studies specifically for the short- and long-term effects of marathon-running in one’s 40s, but we did find a couple of relevant ones:

    The first looked at marathon-runners of various ages, and found that…

    • there are virtually no relevant running time differences (p<0.01) per age in marathon finishers from 20 to 55 years
    • the majority of middle-aged and elderly athletes have training histories of less than seven years of running

    From which they concluded:

    ❝The present findings strengthen the concept that considers aging as a biological process that can be considerably speeded up or slowed down by multiple lifestyle related factors.❞

    See the study: Performance, training and lifestyle parameters of marathon runners aged 20–80 years: results of the PACE-study

    The other looked specifically at the impact of running on cartilage, controlled for age (45 and under vs 46 and older) and activity level (marathon-runners vs sedentary people).

    The study had the people, of various ages and habitual activity levels, run for 30 minutes, and measured their knee cartilage thickness (using MRI) before and after running.

    They found that regardless of age or habitual activity level, running compressed the cartilage tissue to a similar extent. From this, it can be concluded that neither age nor marathon-running result in long-term changes to cartilage response to running.

    Or in lay terms: there’s no reason that marathon-running at 40 should ruin your knees (unless you are doing something wrong).

    That may or may not have been a concern you have, but it’s what the study looked at, so hey, it’s information.

    Here’s the study: Functional cartilage MRI T2 mapping: evaluating the effect of age and training on knee cartilage response to running

    Q: Information on [e-word] dysfunction for those who have negative reactions to [the most common medications]?

    When it comes to that particular issue, one or more of these three factors are often involved:

    • Hormones
    • Circulation
    • Psychology

    The most common drugs (that we can’t name here) work on the circulation side of things—specifically, by increasing the localized blood pressure. The exact mechanism of this drug action is interesting, albeit beyond the scope of a quick answer here today. On the other hand, the way that they work can cause adverse blood-pressure-related side effects for some people; perhaps you’re one of them.

    To take matters into your own hands, so to speak, you can address each of those three things we just mentioned:

    Hormones

    Ask your doctor (or a reputable phlebotomy service) for a hormone test. If your free/serum testosterone levels are low (which becomes increasingly common in men over the age of 45), they may prescribe something—such as testosterone shots—specifically for that.

    This way, it treats the underlying cause, rather than offering a workaround like those common pills whose names we can’t mention here.

    Circulation

    Look after your heart health; eat for your heart health, and exercise regularly!

    Cold showers/baths also work wonders for vascular tone—which is precisely what you need in this matter. By rapidly changing temperatures (such as by turning off the hot water for the last couple of minutes of your shower, or by plunging into a cold bath), your blood vessels will get practice at constricting and maintaining that constriction as necessary.

    Psychology

    [E-word] dysfunction can also have a psychological basis. Unfortunately, this can also then be self-reinforcing, if recalling previous difficulties causes you to get distracted/insecure and lose the moment. One of the best things you can do to get out of this catch-22 situation is to not worry about it in the moment. Depending on what you and your partner(s) like to do in bed, there are plenty of other equally respectable options, so just switch track!

    Having a conversation about this in advance will probably be helpful, so that everyone’s on the same page of the script in that eventuality, and it becomes “no big deal”. Without that conversation, misunderstandings and insecurities could arise for your partner(s) as well as yourself (“aren’t I desirable enough?” etc).

    So, to recap, we recommend:

    • Have your hormones checked
    • Look after your circulation
    • Make the decision to have fun!

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  • Apple vs Papaya – Which is Healthier?

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    Our Verdict

    When comparing apple to papaya, we picked the papaya.

    Why?

    Both do have their merits, but ultimately, it wasn’t close:

    In terms of macros, apples have more fiber and carbs, while papaya has more protein. Since the protein is negligible and the fiber difference is greatest, this means a marginal win for apples here.

    In the category of vitamins, apples are not higher in any vitamin, while papayas have more of vitamins A, B1, B3, B5, B7, B9, C, E, and K, for a total win in this round.

    Looking at minerals, apples have the tiniest bit more phosphorus (so close that simply using slightly different soil could swing it), while papayas have more calcium, copper, iron, magnesium, manganese, potassium, selenium, and zinc, winning easily.

    Adding up the sections makes for an overall win for papaya, but by all means enjoy either or both, as diversity is good, and apple’s fiber content is great!

    Want to learn more?

    You might like:

    What Do The Different Kinds Of Fiber Do? 30 Foods That Rank Highest

    Enjoy!

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  • An Accessible New Development Against Alzheimer’s

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    Dopamine vs Alzheimer’s

    One of the key hallmarks of Alzheimer’s disease is the formation of hardened beta-amyloid plaques around neurons. The beta-amyloid peptides themselves are supposed to be in the brain, but the hardened pieces of them that form the plaques are not.

    While the full nature of the relationship between those plaques and Alzheimer’s disease is not known for sure (there are likely other factors involved, and “the amyloid hypothesis” is at this stage nominally just that, a hypothesis), one thing that has been observed is that increasing or reducing the plaques increases or reduces (respectively) Alzheimer’s symptoms such as memory loss.

    Neprilysin

    There is an enzyme, neprilysin, that can break down those plaques.

    Neprilysin is made naturally in the brain, and/but we cannot take it as a supplement or medication, because it’s too big to pass through the blood-brain barrier.

    A team of researchers led by Dr. Takaomi Saido genetically manipulated mice to produce more neprilysin, and those mice resultantly experienced fewer beta-amyloid plaques and better memory in their old age.

    However wonderful for the mice (and a great proof of principle) the above approach is not useful as a treatment for humans whose genomes weren’t modified at our conception in a lab.

    Since (as mentioned before) we also can’t take it as a medication/supplement, that leaves one remaining option: find a way to make our already-existing brains produce more of it.

    The team’s previous research allowed them to narrow this down to “there is probably a hormone made in the hypothalamus that modulates this”, so they began experimenting with making the mice produce more hormones there.

    The DREADD switch

    DREADDs, or Designer Receptors Exclusively Activated by Designer Drugs, were the next tool in the toolbox. The scientists attached these designer receptors to dopamine-producing neurons in the mice, so that they could be activated by the appropriate designer drugs—basically, allowing for a “make more dopamine” button, without having to literally wire up the brains with electrodes. The “button” gets triggered instead by a chemical trigger, the designer drug. You can read more about them here:

    DREADDs for Neuroscientists: A Primer

    The result was positive; when the mice made more dopamine, the result was that they also made more neprilysin. So far, the hypothesis is that the presence of dopamine upregulates the production of neprilysin. In other words, the increased neprilysin levels were caused by the increased dopamine levels (the alternatives would have been: they were both caused by the same thing—in this case that’d be the DREADD activation—or the increase was caused by something else entirely that hadn’t been controlled for).

    As to how the causal relationship was determined…

    “But I don’t have (or want) a DREADD switch in my head”

    Happily for us (and probably happily for the mice too, because dopamine causes feelings of happiness), the experiments continued.

    This time, instead of using the DREADD system, they tried simply supplementing the mouse food with l-dopa, a dopamine precursor. L-dopa is often used in the treatment of Parkinson’s disease, because the molecules are small enough to pass through the blood-brain barrier, and can be converted to full dopamine inside the brain itself. So, taking l-dopa normally raises dopamine levels.

    The results? The mice who were given l-dopa enjoyed:

    • higher dopamine levels
    • higher neprilysin levels
    • lower beta-amyloid plaque levels
    • better memory in tests

    The next step for the researchers is to investigate how exactly dopamine regulates neprilysin in the brain, but for now, the relationship between l-dopa consumption and the reduction of Alzheimer’s symptoms seems clear.

    You can read about the study here:

    The dopaminergic system promotes neprilysin-mediated degradation of amyloid-β in the brain

    Is there a catch?

    L-dopa has common side effects that are not pleasant; the list begins with nausea and vomiting, and continues with things that one might expect from having “too much of a good thing” when it comes to dopamine, such as dyskinesia (extra movements) and hallucinations.

    You can read about it more here at the Parkinson’s Foundation:

    Parkinson’s Foundation | Levodopa

    However! All is not lost. Rather than reaching for the heavy guns by taking l-dopa unnecessarily, there are other dopamine precursors that don’t have those side effects (and are consequently less restricted, to the point they can be purchased as supplements, or indeed, enjoyed where they occur naturally in some foods).

    Top of the list of such safe* and readily-available dopamine precursors is…

    N-Acetyl L-Tyrosine (NALT): The Dopamine Precursor & More

    If you’d like to try that, here’s an example product on Amazon… Or you could eat fish, white beans, tofu, natto, or pumpkin seeds 😉

    *Quick note on safety: “safe” is a relative term and may vary from person to person. Please speak with your own doctor to be sure, check with your pharmacist in case of any meds interactions, and be especially careful taking anything that increases dopamine levels if you have bipolar disorder or are otherwise prone to psychosis of any kind. For most people, this shouldn’t be an issue as our brains have a built-in mechanism for scrubbing excess dopamine and ensuring we don’t end up with too much, but for some people whose dopamine regulation is not so good in that regard, it can cause problems. So again, speak with your doctor to be sure, because we are not doctors, let alone your doctor.

    Lastly…

    If you’d like an entirely drug-free approach, that’s skipping even the “nutraceuticals”, you might enjoy:

    Short On Dopamine? Science Has The Answer

    Take care!

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  • A Very Cheap Way To Slow Biological Aging

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    “Multivitamins and minerals are good for longevity” is an interesting one as a topic, because:

    1. On the one hand, that is already popularly assumed
    2. On the other hand, multivitamins and minerals are equally popularly much-derided as a waste of money

    And indeed, they have their limitations, and yes, it’s still better to get everything from one’s diet.

    Warning: do not, however, fall into the trap that many do, and think “I don’t need to take supplements because it’s better to get it from my diet” and then not, in fact, get a full coverage from your diet

    So, for whatever reason you might choose to take a multivitamin and mineral, let’s take a look at…

    Cause and effect

    Researchers (Dr. Sidong Li et al.) found that taking a daily multivitamin for two years slowed biological aging by the equivalent of about four months.

    So, that’s basically as if every time you went forwards 6 years, you went back 1 year.

    Compared to what, you ask?

    Good question, because if it’s “compared to not taking it”, and if it’s an observational study, then there can be all kinds of confounding factors, most of them rooted in the theme “people who do thing 1 for their health are more likely to also do things 2, 3, 4, and 5 for their health”.

    The answer, happily, is: “compared to placebo”. It was a randomized controlled trial.

    Next up, some may be wondering what “biological age” is. In fact, biological age is not one thing but quite a lot of things, each of which can age at different rates.

    See for example: Age & Aging: What Can (And Can’t) We Do About It?

    One of the best ways to try to boil it down to a composite figure, however, is to look at epigenetic clocks.

    First let’s quickly cover the question of: what does “epigenetic” mean? In few words and put simply, epigenetics is the study of “around genetics”, i.e. the things that are not the genes themselves, but modulate how (and indeed, whether or not) genes are expressed.

    In this case, Dr. Li and her team used epigenetic clocks that estimate biological age based on patterns of DNA methylation, which influence gene expression and naturally change with age. The quicker the progression of the clock, the faster the aging.

    The analysis included DNA methylation data from blood samples of 958 healthy participants with an average chronological age of 70 who were randomized to receive combinations of cocoa extract, multivitamins & minerals, or placebo.

    • Good news: participants taking a multivitamin enjoyed slowing across all five epigenetic aging clocks tested, with strongest effects in the two clocks most strongly linked to mortality risk.
    • Bad news: the cocoa extract supplement was a bit of a flop. Didn’t help. Or rather, it had no effect whatsoever on the 5 epigenetic clocks tested.

    The reason why she tested cocoa extract for this, is because of: Cocoa vs Biological Aging!

    …in which Dr. Li (yes, the same Dr. Li) and her team (mostly the same team, but not entirely the same) found that daily cocoa extract reduced hsCRP (a key inflammation marker tied to heart disease) by about 8.4% per year compared with placebo, suggesting anti-inflammatory and cardio-protective effects, and also noted that this cocoa extract supplementation reduced cardiovascular mortality by 27%.

    Which is relevant, because epigenetic clocks and inflammatory markers are two out of three of: The 3 Best Predictors Of How Well You’ll Age

    If you’d like to read Dr. Li’s latest work, you can find it here: Effects of daily multivitamin–multimineral and cocoa extract supplementation on epigenetic aging clocks in the COSMOS randomized clinical trial ← published literally today, at time of writing this article. Never let it be said we don’t bring you cutting edge health science news!

    Want to learn more?

    You might like this book we reviewed a while back

    Eat Your Vitamins – by Mascha Davis, RDN ← This book methodically discusses an assortment of vitamins, minerals, and other nutrients; the “other nutrients” category including amino acids (branched chain and essential), prebiotics and probiotics, and triglycerides of various kinds.

    Enjoy!

    Don’t Forget…

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