
A New $16,000 Postpartum Depression Drug Is Here. How Will Insurers Handle It?
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A much-awaited treatment for postpartum depression, zuranolone, hit the market in December, promising an accessible and fast-acting medication for a debilitating illness. But most private health insurers have yet to publish criteria for when they will cover it, according to a new analysis of insurance policies.
The lack of guidance could limit use of the drug, which is both novel — it targets hormone function to relieve symptoms instead of the brain’s serotonin system, as typical antidepressants do — and expensive, at $15,900 for the 14-day pill regimen.
Lawyers, advocates, and regulators are watching closely to see how insurance companies will shape policies for zuranolone because of how some handled its predecessor, an intravenous form of the same drug called brexanolone, which came on the market in 2019. Many insurers required patients to try other, cheaper medications first — known as the fail-first approach — before they could be approved for brexanolone, which was shown in early trials reviewed by the FDA to provide relief within days. Typical antidepressants take four to six weeks to take effect.
“We’ll have to see if insurers cover this drug and what fail-first requirements they put in” for zuranolone, said Meiram Bendat, a licensed psychotherapist and an attorney who represents patients.
Most health plans have yet to issue any guidelines for zuranolone, and maternal health advocates worry that the few that have are taking a restrictive approach. Some policies require that patients first try and fail a standard antidepressant before the insurer will pay for zuranolone.
In other cases, guidelines require psychiatrists to prescribe it, rather than obstetricians, potentially delaying treatment since OB-GYN practitioners are usually the first medical providers to see signs of postpartum depression.
Advocates are most worried about the lack of coverage guidance.
“If you don’t have a published policy, there is going to be more variation in decision-making that isn’t fair and is less efficient. Transparency is really important,” said Joy Burkhard, executive director of the nonprofit Policy Center for Maternal Mental Health, which commissioned the study.
With brexanolone, which was priced at $34,000 for the three-day infusion, California’s largest insurer, Kaiser Permanente, had such rigorous criteria for prescribing it that experts said the policy amounted to a blanket denial for all patients, according to an NPR investigation in 2021.
KP’s written guidelines required patients to try and fail four medications and electroconvulsive therapy before they would be eligible for brexanolone. Because the drug was approved only for up to six months postpartum, and trials of typical antidepressants take four to six weeks each, the clock would run out before a patient had time to try brexanolone.
An analysis by NPR of a dozen other health plans at the time showed Kaiser Permanente’s policy on brexanolone to be an outlier. Some did require that patients fail one or two other drugs first, but KP was the only one that recommended four.
Miriam McDonald, who developed severe postpartum depression and suicidal ideation after giving birth in late 2019, battled Kaiser Permanente for more than a year to find effective treatment. Her doctors put her on a merry-go-round of medications that didn’t work and often carried unbearable side effects, she said. Her doctors refused to prescribe brexanolone, the only FDA-approved medication specifically for postpartum depression at the time.
“No woman should suffer like I did after having a child,” McDonald said. “The policy was completely unfair. I was in purgatory.”
One month after NPR published its investigation, KP overhauled its criteria to recommend that women try just one medication before becoming eligible for brexanolone.
Then, in March 2023, after the federal Department of Labor launched an investigation into the insurer — citing NPR’s reporting — the insurer revised its brexanolone guidelines again, removing all fail-first recommendations, according to internal documents recently obtained by NPR. Patients need only decline a trial of another medication.
“Since brexanolone was first approved for use, more experience and research have added to information about its efficacy and safety,” the insurer said in a statement. “Kaiser Permanente is committed to ensuring brexanolone is available when physicians and patients determine it is an appropriate treatment.”
“Kaiser basically went from having the most restrictive policy to the most robust,” said Burkhard of the Policy Center for Maternal Mental Health. “It’s now a gold standard for the rest of the industry.”
McDonald is hopeful that her willingness to speak out and the subsequent regulatory actions and policy changes for brexanolone will lead Kaiser Permanente and other health plans to set patient-friendly policies for zuranolone.
“This will prevent other women from having to go through a year of depression to find something that works,” she said.
Clinicians were excited when the FDA approved zuranolone last August, believing the pill form, taken once a day at home over two weeks, will be more accessible to women compared with the three-day hospital stay for the IV infusion. Many perinatal psychiatrists told NPR it is imperative to treat postpartum depression as quickly as possible to avoid negative effects, including cognitive and social problems in the baby, anxiety or depression in the father or partner, or the death of the mother to suicide, which accounts for up to 20% of maternal deaths.
So far, only one of the country’s six largest private insurers, Centene, has set a policy for zuranolone. It is unclear what criteria KP will set for the new pill. California’s Medicaid program, known as Medi-Cal, has not yet established coverage criteria.
Insurers’ policies for zuranolone will be written at a time when the regulatory environment around mental health treatment is shifting. The U.S. Department of Labor is cracking down on violations of the Mental Health Parity and Addiction Equity Act of 2008, which requires insurers to cover psychiatric treatments the same as physical treatments.
Insurers must now comply with stricter reporting and auditing requirements intended to increase patient access to mental health care, which advocates hope will compel health plans to be more careful about the policies they write in the first place.
In California, insurers must also comply with an even broader state mental health parity law from 2021, which requires them to use clinically based, expert-recognized criteria and guidelines in making medical decisions. The law was designed to limit arbitrary or cost-driven denials for mental health treatments and has been hailed as a model for the rest of the country. Much-anticipated regulations for the law are expected to be released this spring and could offer further guidance for insurers in California setting policies for zuranolone.
In the meantime, Burkhard said, patients suffering from postpartum depression should not hold back from asking their doctors about zuranolone. Insurers can still grant access to the drug on a case-by-case basis before they formalize their coverage criteria.
“Providers shouldn’t be deterred from prescribing zuranolone,” Burkhard said.
This article is from a partnership that includes KQED, NPR and KFF Health News.
KFF Health News is a national newsroom that produces in-depth journalism about health issues and is one of the core operating programs at KFF—an independent source of health policy research, polling, and journalism. Learn more about KFF.
Subscribe to KFF Health News’ free Morning Briefing.
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A free shower is the least older people can expect, but aged care funding misses one key point
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This week, we learned older people in home-based aged care will no longer have to pay out-of-pocket for showering, dressing and continence care.
This backflip will provide relief for those currently receiving services under the Support at Home program and the 100,000 or so people on the waiting list for home care.
For people with continence issues, wounds and other issues that make showering essential, this is welcome news and something both advocates and consumers have been calling for.
This announcement comes as the government grapples with the cost of providing health care in various forms, prompting major changes to the National Disability Insurance Scheme, aged care and private health insurance.
In fact, the government plans to pay for increased funding for aged care, including the Support at Home program, by scrapping the additional private health insurance rebate for the over-65s.
One key issue now is how Australia subsidises this type of aged care without shifting excessive costs onto future generations.
Jacob Wackerhausen/Getty Equitable but at what cost?
A key push of the Support at Home program, which started in November 2025, is that people who can afford it should fund more of their own care. The aim of this so-called “vertical equity” is to ensure the system is sustainable.
In theory, this protects funding for those who need it most. In practice, it has raised questions about whether it has undermined access to necessary care.
There’s a list with three types of services requiring the person receiving care to contribute at different levels:
- Clinical support services require no co-contribution, regardless of means. This includes services such as wound care or podiatry.
- Independence (including personal care) requires a contribution of 5–50% of the fee depending on income and assets. This currently includes services such as showering, social support and respite care.
- Everyday living requires the biggest contribution of 17.5–80%. This includes cleaning, home maintenance and gardening.
Let’s see what this means in dollar terms. Currently, if a shower costs about A$100 an hour (not unreasonable given this hourly rate has to include superannuation, travel, workers compensation, for instance), a person on a full aged pension would have to pay $5 per shower and a person at full rates would pay $50.
You can see how this adds up quickly with payments also required for other services, such as cleaning and gardening eating into a fixed age pension. Getting help to shower every day becomes impossible – particularly with higher rates paid at the weekend.
Some people may be able to get friends and neighbours to help with some tasks, such as mowing the lawn or putting out the bins. But showering is intensely personal. It isn’t something you want to have to ask of a friend.
However, the recent announcement means personal care – showering, dressing, continence care – moves from being classified as “independence” which attracted a co-payment to “clinical support”, which requires the participant to pay nothing out-of-pocket.
This ensures a different type of equity, known as “horizontal equity”. In other words, everyone with similar clinical needs can access the same support.
But there’s a flip side. This change means people who could afford to contribute to personal care will no longer need to do so. This increases the share of costs borne by taxpayers.
Why are there different subsidies?
When people start to have difficulty managing their daily activities, they often turn to requesting help doing the cleaning, cooking and gardening rather than working on improving or regaining their capacity to do those tasks.
The idea behind setting varied prices for the different types of services is to shift this pattern.
It’s to encourage people to get the clinical support they need and promote capacity building – via using services with no out-of-pocket costs – so people can continue to manage daily living at home. This may mean bringing in a physiotherapist to help someone move about, and maintain muscle mass and stability, making it easier for them to manage at home.
This logic makes sense early on, where people are capable of reversing or preventing frailty. We want to encourage people to stay active and well. But this isn’t always possible.
Requiring co-payments for support services – such as support to prepare meals or do the laundry under the everyday living category – when capacity can’t be regained can feel like a punitive measure. It’s this part of the funding equation that the latest announcement doesn’t touch on.
How about the future?
Currently, we don’t know if the Support at Home program is delivering its intended effect of increasing access to clinical and capacity building services while charging more for those who can afford it to pay for their care.
But we have a great opportunity to find out. We can compare the types of services people receive under the previous version of the home aged-care scheme before November 2025 (which some people are still on) with the current scheme.
As the Support at Home program matures, we also need to review the level and type of services that attract co-payments. We need to understand if people are forgoing some types of care due to the co-payments and whether other adjustments to the program are needed.
As people progress and need more care, we may need to consider whether co-payments for certain services are still a good idea, or are creating new inequities. As one example, cleaning may need to be provided without a co-payment for people with greater care needs and less ability to pay.
We also need to consider whether wealthier older people should pay more.
A delicate balance
This announcement addresses a clear and important equity concern by removing financial barriers to essential personal care. But it also highlights the delicate balance governments must strike in designing a sustainable aged-care system – one that protects access for those with the greatest needs, while fairly sharing costs across the community.
As Support at Home matures, equity will need to be monitored and government must be prepared to make changes where needed.
Getting that balance right will be crucial to ensuring older Australians can age with dignity, without causing intergenerational inequity by shifting excessive costs onto future generations.
Tracy Comans, Professor, School of Public Health, The University of Queensland; The University of Melbourne
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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A drug that can extend your life by 25%? Don’t hold your breath
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Every few weeks or months, the media reports on a new study that tantalisingly dangles the possibility of a new drug to give us longer, healthier lives.
The latest study centres around a drug involved in targeting interleukin-11, a protein involved in inflammation. Blocking this protein appeared to help mice stave off disease and extend their life by more than 20%.
If only defying the ravages of time could be achieved through such a simple and effort-free way – by taking a pill. But as is so often the case, the real-world significance of these findings falls a fair way short of the hype.
Halfpoint/Shutterstock The role of inflammation in disease and ageing
Chronic inflammation in the body plays a role in causing disease and accelerating ageing. In fact, a relatively new label has been coined to represent this: “inflammaging”.
While acute inflammation is an important response to infection or injury, if inflammation persists in the body, it can be very damaging.
A number of lifestyle, environmental and societal drivers contribute to chronic inflammation in the modern world. These are largely the factors we already know are associated with disease and ageing, including poor diet, lack of exercise, obesity, stress, lack of sleep, lack of social connection and pollution.
While addressing these issues directly is one of the keys to addressing chronic inflammation, disease and ageing, there are a number of research groups also exploring how to treat chronic inflammation with pharmaceuticals. Their goal is to target and modify the molecular and chemical pathways involved in the inflammatory process itself.
What the latest research shows
This new interleukin-11 research was conducted in mice and involved a number of separate components.
In one component of this research, interleukin-11 was genetically knocked out in mice. This means the gene for this chemical mediator was removed from these mice, resulting in the mice no longer being able to produce this mediator at all.
In this part of the study, the mice’s lives were extended by over 20%, on average.
Another component of this research involved treating older mice with a drug that blocks interleukin-11.
Injecting this drug into 75-week old mice (equivalent to 55-year-old humans) was found to extend the life of mice by 22-25%.
These treated mice were less likely to get cancer and had lower cholesterol levels, lower body weight and improved muscle strength and metabolism.
From these combined results, the authors concluded, quite reasonably, that blocking interleukin-11 may potentially be a key to mitigating age-related health effects and improving lifespan in both mice and humans.
Why you shouldn’t be getting excited just yet
There are several reasons to be cautious of these findings.
First and most importantly, this was a study in mice. It may be stating the obvious, but mice are very different to humans. As such, this finding in a mouse model is a long way down the evidence hierarchy in terms of its weight.
Research shows only about 5% of promising findings in animals carry over to humans. Put another way, approximately 95% of promising findings in animals may not be translated to specific therapies for humans.
Second, this is only one study. Ideally, we would be looking to have these findings confirmed by other researchers before even considering moving on to the next stage in the knowledge discovery process and examining whether these findings may be true for humans.
We generally require a larger body of evidence before we get too excited about any new research findings and even consider the possibility of human trials.
Third, even if everything remains positive and follow-up studies support the findings of this current study, it can take decades for a new finding like this to be translated to successful therapies in humans.
Until then, we can focus on doing the things we already know make a huge difference to health and longevity: eating well, exercising, maintaining a healthy weight, reducing stress and nurturing social relationships.
Hassan Vally, Associate Professor, Epidemiology, Deakin University
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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5 unexpected ways your oral health affects your overall health
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What you need to know
- The health of your teeth and gums affects the health of your whole body.
- Research shows that poor oral health can increase your risk of heart disease, dementia, breathing issues, and even cancer.
- You can protect your oral and overall health by cleaning your teeth daily, getting regular dentist checkups, eating a healthy diet, and avoiding tobacco.
When you brush and floss daily, you don’t just prevent cavities and gum disease: You also support your overall health.
Research shows that poor dental health may put you at risk for serious health issues, including infections, certain types of cancer, and dementia. People who are older, are pregnant, or have chronic health conditions like diabetes and heart disease are the most vulnerable to oral health-related risks.
Here are some unexpected ways that dental health impacts your overall health and well-being.
Cardiovascular health
For decades, researchers have studied a potential connection between oral health and cardiovascular diseases (conditions affecting the heart and blood vessels). Although it isn’t clear that dental issues cause cardiovascular issues, there is evidence that the two are linked.
“Some studies have found that people with gum disease may be twice as likely to have a heart attack or a stroke,” said Dr. Nicholas Ruthmann, a cardiologist at the Cleveland Clinic, on a 2021 episode of the “Love Your Heart” podcast.
Our mouths are home to an entire ecosystem of bacteria, some of which form plaque that sticks to your teeth and cause gum disease. These bacteria can also travel through blood vessels to other parts of your body. This causes inflammation, which can damage your blood vessels and even lead to blood clots and other cardiovascular issues.
Research suggests that gum disease-related inflammation can have a profound impact on your cardiovascular health. A 2017 review outlined multiple ways that the oral bacteria that cause tooth plaque and gum disease may also contribute to the buildup of harmful plaque in the arteries.
“Gum disease can also create a portal for bacteria to enter the bloodstream,” added Ruthmann. “Research has shown that the same bacteria from common oral infections has also been found in plaques and blockages that form in our heart arteries.”
Alzheimer’s disease and dementia
Like the heart, the brain is also susceptible to the germs that cause oral health issues. A 2019 study found that one of the bacteria that causes gum disease may be linked to Alzheimer’s disease. The bacteria, Porphyromonas gingivalis, travels from the mouth to the brain, where it can damage brain cells. High levels of the bacteria are found in the brains of people with Alzheimer’s disease.
A larger study the following year supported these results, showing that older adults with a history of gum disease were more likely to develop Alzheimer’s disease. Meanwhile, two 2022 studies found that an enzyme released by Porphyromonas gingivalis can contribute to the buildup of the two major proteins involved in Alzheimer’s disease.
Several studies have also found a connection between dementia risk and tooth loss, which may result from gum disease, cavities, or physical trauma. A 2021 study showed that dementia risk increased with each tooth lost.
The findings “underscore the importance of maintaining good oral health and its role in helping to preserve cognitive function,” said Bei Wu, co-author of the study and co-director of New York University’s Aging Incubator.
Cancer
In recent years, scientists have identified a type of bacteria most commonly found in the mouth that may play a role in some cancers. Fusobacterium nucleatum is a normal part of the community of bacteria that live in our mouths. But when there’s too much of the bacteria, it can cause infections like gingivitis.
The bacteria flourish in the intestines of people with colon cancer—and one type appears to drive tumor growth, according to a 2024 study. The study, conducted in mice, found that the bacteria improved conditions for tumor formation and increased tumor growth.
“Patients who have high levels of this bacteria in their colorectal tumors have a far worse prognosis,” said Susan Bullman, one of the study’s authors and an immunologist at the University of Texas MD Anderson Cancer Center, in a 2024 NBC News article.
“They don’t respond as well to chemotherapy and they have an increased risk of recurrence,” she added.
Previous research found that a history of gum disease is associated with a significantly higher risk of stomach and esophageal cancers, as well as breast cancer. Notably, Fusobacterium nucleatum is found at high levels in breast cancer tumors, suggesting that the bacteria may also play a role in one of the most common types of cancer.
Respiratory infections
Many bacteria can travel from our teeth, gums, and saliva to the lungs. Healthy lungs can typically fight harmful oral bacteria, preventing any harm.
But in people with existing respiratory conditions like asthma and chronic obstructive pulmonary disease, these bacteria can trigger inflammation and infections, worsening the conditions.
A 2022 review highlighted the evidence for a link between mouth bacteria and pneumonia, COPD, asthma, and other lung conditions. Bacteria and viruses that cause respiratory disease have also been found in tooth plaque, gums, and saliva.
Several studies have identified a connection between poor oral health and respiratory illness, although the exact relationship remains unclear. For example, data “strongly suggest” an association between gum disease and asthma and “higher odds” of tooth loss in people with asthma and COPD.
Pregnancy
Pregnancy is known to take a toll on oral health. But there’s also evidence that the health of your mouth can affect the health of your pregnancy. In fact, gum disease during pregnancy has been linked to serious pregnancy risks, including early labor, low birth weight, and preeclampsia.
Preeclampsia, a potentially serious pregnancy complication that causes high blood pressure, is the second most common cause of maternal death. Research suggests that hormonal fluctuations during pregnancy can worsen gum disease, which may in turn trigger an increased risk of preeclampsia.
Because of these risks, health care providers recommend maintaining dental hygiene, getting regular dental care, and quickly addressing any oral health concerns before and during pregnancy.
Tips to improve oral health
- Brush and floss daily: The American Dental Association recommends brushing your teeth twice a day with a fluoride toothpaste and flossing every day. You may also use a mouthwash for additional protection.
- Go to the dentist regularly: Regular dental cleanings help prevent gum disease and catch issues like cavities before they become a major concern. Don’t put off dental care until a problem arises. Prevention is better—and cheaper—than treatment.
- No dental insurance? Find low-cost dental care: Don’t forgo regular care if you don’t have dental insurance. Dental schools, public and free clinics, community health centers, nonprofit organizations like United Way, and local and state health centers may offer affordable options. Learn more about accessing free or low-cost dental care here and here.
- Eat a balanced diet with limited sugar and acid: Foods and drinks that are high in sugar and acid can cause tooth decay and weaken enamel, the protective outer layer of the tooth.
- Avoid tobacco: All types of tobacco use, including vaping, have been linked to gum disease, inflammation, and oral cancer.
For more information, talk to your dentist or health care provider.
This article first appeared on Public Good News and is republished here under a Creative Commons Attribution-NoDerivatives 4.0 International License.
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3 signs your diet is causing too much muscle loss – and what to do about it
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When trying to lose weight, it’s natural to want to see quick results. So when the number on the scales drops rapidly, it seems like we’re on the right track.
But as with many things related to weight loss, there’s a flip side: rapid weight loss can result in a significant loss of muscle mass, as well as fat.
So how you can tell if you’re losing too much muscle and what can you do to prevent it?
EvMedvedeva/Shutterstock Why does muscle mass matter?
Muscle is an important factor in determining our metabolic rate: how much energy we burn at rest. This is determined by how much muscle and fat we have. Muscle is more metabolically active than fat, meaning it burns more calories.
When we diet to lose weight, we create a calorie deficit, where our bodies don’t get enough energy from the food we eat to meet our energy needs. Our bodies start breaking down our fat and muscle tissue for fuel.
A decrease in calorie-burning muscle mass slows our metabolism. This quickly slows the rate at which we lose weight and impacts our ability to maintain our weight long term.
How to tell you’re losing too much muscle
Unfortunately, measuring changes in muscle mass is not easy.
The most accurate tool is an enhanced form of X-ray called a dual-energy X-ray absorptiometry (DXA) scan. The scan is primarily used in medicine and research to capture data on weight, body fat, muscle mass and bone density.
But while DEXA is becoming more readily available at weight-loss clinics and gyms, it’s not cheap.
There are also many “smart” scales available for at home use that promise to provide an accurate reading of muscle mass percentage.
Some scales promise to tell us our muscle mass. Lee Charlie/Shutterstock However, the accuracy of these scales is questionable. Researchers found the scales tested massively over- or under-estimated fat and muscle mass.
Fortunately, there are three free but scientifically backed signs you may be losing too much muscle mass when you’re dieting.
1. You’re losing much more weight than expected each week
Losing a lot of weight rapidly is one of the early signs that your diet is too extreme and you’re losing too much muscle.
Rapid weight loss (of more than 1 kilogram per week) results in greater muscle mass loss than slow weight loss.
Slow weight loss better preserves muscle mass and often has the added benefit of greater fat mass loss.
One study compared people in the obese weight category who followed either a very low-calorie diet (500 calories per day) for five weeks or a low-calorie diet (1,250 calories per day) for 12 weeks. While both groups lost similar amounts of weight, participants following the very low-calorie diet (500 calories per day) for five weeks lost significantly more muscle mass.
2. You’re feeling tired and things feel more difficult
It sounds obvious, but feeling tired, sluggish and finding it hard to complete physical activities, such as working out or doing jobs around the house, is another strong signal you’re losing muscle.
Research shows a decrease in muscle mass may negatively impact your body’s physical performance.
3. You’re feeling moody
Mood swings and feeling anxious, stressed or depressed may also be signs you’re losing muscle mass.
Research on muscle loss due to ageing suggests low levels of muscle mass can negatively impact mental health and mood. This seems to stem from the relationship between low muscle mass and proteins called neurotrophins, which help regulate mood and feelings of wellbeing.
So how you can do to maintain muscle during weight loss?
Fortunately, there are also three actions you can take to maintain muscle mass when you’re following a calorie-restricted diet to lose weight.
1. Incorporate strength training into your exercise plan
While a broad exercise program is important to support overall weight loss, strength-building exercises are a surefire way to help prevent the loss of muscle mass. A meta-analysis of studies of older people with obesity found resistance training was able to prevent almost 100% of muscle loss from calorie restriction.
Relying on diet alone to lose weight will reduce muscle along with body fat, slowing your metabolism. So it’s essential to make sure you’ve incorporated sufficient and appropriate exercise into your weight-loss plan to hold onto your muscle mass stores.
Strength-building exercises help you retain muscle. BearFotos/Shutterstock But you don’t need to hit the gym. Exercises using body weight – such as push-ups, pull-ups, planks and air squats – are just as effective as lifting weights and using strength-building equipment.
Encouragingly, moderate-volume resistance training (three sets of ten repetitions for eight exercises) can be as effective as high-volume training (five sets of ten repetitions for eight exercises) for maintaining muscle when you’re following a calorie-restricted diet.
2. Eat more protein
Foods high in protein play an essential role in building and maintaining muscle mass, but research also shows these foods help prevent muscle loss when you’re following a calorie-restricted diet.
But this doesn’t mean just eating foods with protein. Meals need to be balanced and include a source of protein, wholegrain carb and healthy fat to meet our dietary needs. For example, eggs on wholegrain toast with avocado.
3. Slow your weight loss plan down
When we change our diet to lose weight, we take our body out of its comfort zone and trigger its survival response. It then counteracts weight loss, triggering several physiological responses to defend our body weight and “survive” starvation.
Our body’s survival mechanisms want us to regain lost weight to ensure we survive the next period of famine (dieting). Research shows that more than half of the weight lost by participants is regained within two years, and more than 80% of lost weight is regained within five years.
However, a slow and steady, stepped approach to weight loss, prevents our bodies from activating defence mechanisms to defend our weight when we try to lose weight.
Ultimately, losing weight long-term comes down to making gradual changes to your lifestyle to ensure you form habits that last a lifetime.
At the Boden Group, Charles Perkins Centre, we are studying the science of obesity and running clinical trials for weight loss. You can register here to express your interest.
Nick Fuller, Charles Perkins Centre Research Program Leader, University of Sydney
This article is republished from The Conversation under a Creative Commons license. Read the original article.
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The End of Food Allergy – by Dr. Kari Nadeau & Sloan Barnett
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We don’t usually mention author credentials beyond their occupation/title. However, in this case it bears acknowledging at least the first line of the author bio:
❝Kari Nadeau, MD, PhD, is the director of the Sean N. Parker Center for Allergy and Asthma Research at Stanford University and is one of the world’s leading experts on food allergy❞
We mention this, because there’s a lot of quack medicine out there [in general, but especially] when it comes to things such as food allergies. So let’s be clear up front that Dr. Nadeau is actually a world-class professional at the top of her field.
This book is, by the way, about true allergies—not intolerances or sensitivities. It does touch on those latter two, but it’s not the main meat of the book.
In particular, most of the research cited is around peanut allergies, though the usual other common allergens are all discussed too.
The authors’ writing style is that of a science educator (Dr. Nadeau’s co-author, Sloan Barnett, is lawyer and health journalist). We get a clear explanation of the science from real-world to clinic and back again, and are left with a strong understanding, not just a conclusion.
The titular “End of Food Allergy” is a bold implicit claim; does the book deliver? Yes, actually.
The book lays out guidelines for safely avoiding food allergies developing in infants, and yes, really, how to reverse them in adults. But…
Big caveat:
The solution for reversing severe food allergies (e.g. “someone nearby touched a peanut three hours ago and now I’m in anaphylactic shock”), drug-assisted oral immunotherapy, takes 6–24 months of weekly several-hour-long clinic visits, relies on having a nearby clinic offering the service, and absolutely 100% cannot be done at home (on pain of probable death).
Bottom line: it’s by no means a magic bullet, but yes, it does deliver.
Click here to check out The End of Food Allergy to learn more!
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