Sleep Tracking, For Five Million Nights

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5 Sleep Phenotypes, By Actual Science

You probably know people can be broadly divided into “early birds” and “night owls”:

Early Bird Or Night Owl? Genes vs Environment

…and then the term “hummingbird” gets used for a person who flits between the two.

That’s three animals so far. If you read a book we reviewed recently, specifically this one:

The Power of When – by Dr. Michael Breus

…then you may have used the guide within to self-diagnose your circadian rhythm type (chronotype) according to Dr. Breus’s system, which divides people into bears, lions, wolves, and dolphins.

That’s another four animals. If you have a FitBit, it can “diagnose” you with being those and/or a menagerie of others, such as giraffe, hedgehog, parrot, and tortoise:

How Fitbit Developed the Sleep Profile Experience (Part 2 – Sleep Animals)

Five million nights

A team of researchers recently took a step away from this veritable zoo of 11 different animals and counting, and used a sophisticated modelling system to create a spatial-temporal map of people’s sleep habits, and this map created five main “islands” that people’s sleep habits could settle on, or sometimes move from island to island.

Those “five million nights” by the way? It was actually 5,095,798 nights! You might notice that would take from the 2020s to the 15970s to complete, so this was rather a matter of monitoring 33,152 individuals between January and October of the same year. Between them, they got those 5,095,798 nights of sleep (or in some cases, nights of little or no sleep, but still, they were there for the nights).

The five main phenotypes that the researchers found were:

  1. What we think of as “normal” sleep. In this phenotype, people get about eight hours of uninterrupted sleep for at least six days in a row.
  2. As above for half the nights, but they only sleep for short periods of time in bouts of less than three hours the other half.
  3. As per normal sleep, but with one interrupted night per week, consisting of a 5 hour sleep period and then broken sleep for a few more hours.
  4. As per normal sleep generally, but with occasional nights in which long bouts of sleep are separated by a mid-sleep waking.
  5. Sleeping for very short periods of time every night. This phenotype was the rarest the researchers found, and represents extremely disrupted sleep.

As you might suspect, phenotype 1 is healthier than phenotype 5. But that’s not hugely informational, as the correlation between getting good sleep and having good health is well-established. So, what did the study teach us?

❝We found that little changes in sleep quality helped us identify health risks. Those little changes wouldn’t show up on an average night, or on a questionnaire, so it really shows how wearables help us detect risks that would otherwise be missed.❞

~ Dr. Benjamin Smarr

More specifically,

❝We found that the little differences in how sleep disruptions occur can tell us a lot. Even if these instances are rare, their frequency is also telling. So it’s not just whether you sleep well or not – it’s the patterns of sleep over time where the key info hides❞

~ Dr. Edward Wang

…and, which gets to the absolute point,

❝If you imagine there’s a landscape of sleep types, then it’s less about where you tend to live on that landscape, and more about how often you leave that area❞

~ Dr. Varun Viswanath

In other words: if your sleep pattern is not ideal, that’s one thing and it’d probably be good to address it, by improving your sleep. However, if your sleep pattern changes phenotype without an obvious known reason why, this may be considered an alarm bell warning of something else that needs addressing, which may be an underlying illness or condition—meaning it can be worthwhile being a little extra vigilant when it comes to regular health screenings, in case something new has appeared.

Want to read more?

You can read the paper in full here:

Five million nights: temporal dynamics in human sleep phenotypes

Take care!

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  • Do I have insomnia? 5 reasons why you might not

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    Even a single night of sleep trouble can feel distressing and lonely. You toss and turn, stare at the ceiling, and wonder how you’ll cope tomorrow. No wonder many people start to worry they’ve developed insomnia.

    Insomnia is one of the most talked-about sleep problems, but it’s also one of the most misunderstood.

    But just because you can’t sleep, it doesn’t mean you have insomnia. You might have another sleep disorder, or none at all.

    Oleg Breslavtsev/Getty

    What is insomnia?

    Let’s clear up some terms, and separate short-term or intermittent sleep problems from what health professionals call “insomnia disorder”.

    Sleep problems can involve being awake when you want to be asleep. This could be lying in bed for ages trying to fall asleep, waking in the middle of the night for hours, or waking up too early. Having a sleep problem is a subjective experience – you don’t need to tally up lost hours to prove it’s a problem.

    But insomnia disorder is the official term to describe a more problematic and persistent pattern of sleep difficulties. And this long-term or chronic sleep disorder has clear diagnostic criteria. These include at least three nights a week of poor sleep, lasting three months or more. These criteria help researchers and clinicians make sure they’re talking about the same thing, and not confusing it with another sleep problem.

    So, what are some reasons why a sleep problem might not be insomnia?

    1. It’s short term, or comes and goes

    About a third of adults will have a bout of “acute insomnia” in a given year. This short-term problem is typically triggered by stress, illness or big life changes.

    The good news is that about 72% of people with acute insomnia return to normal sleep after a few weeks.

    Insomnia disorder is a longer-term, persistent problem.

    2. It doesn’t affect you the next day

    Some people lie awake at night but still function well during the day. More fragmented and less refreshing sleep is also a near-universal part of ageing.

    So if your sleep problem doesn’t significantly affect you the next day, it usually isn’t considered to be insomnia.

    For people with insomnia, the struggle with sleep spills into the day and affects their mood, energy, concentration and wellbeing. Worry and distress about not sleeping can then make the problem worse, which creates a frustrating cycle of worrying and not sleeping.

    3. It’s more about work or caring

    If you feel tired during the day, an important question is whether you’re giving yourself enough time to sleep. Sometimes sleep problems reflect a “sleep opportunity” that is too short or too irregular.

    Work schedules, child care, or late-night commitments can cut sleep short, and sleep can slip down the priority list. In these cases, the problem is insufficient sleep, not insomnia.

    You might have noisy neighbours or an annoying cat. These can also affect your sleep, and reduce your “sleep opportunity”.

    The average healthy adult gets around seven hours sleep (though this varies widely). For someone who needs seven, it usually means setting aside about eight to allow for winding down, drifting off, and waking overnight.

    4. It’s another sleep disorder

    Other sleep disorders can look like insomnia, such as:

    • obstructive sleep apnoea (when your breathing stops multiple times during sleep) can cause frequent awakenings through the night and daytime sleepiness
    • restless legs syndrome creates an irresistible urge to move your legs in the evening that often interferes with falling asleep. It’s often described as jittery feelings or having “creepy crawlies”, and is often undiagnosed
    • circadian rhythm problems, such as being a natural night owl in an early-bird world, can also lead to trouble falling asleep.

    5. Medications and substances are interfering

    Caffeine, alcohol and nicotine all create insomnia symptoms and worsen the quality of sleep.

    Certain medications can also interfere with sleep, such as stimulants (for conditions such as attention-deficit hyperactivity disorder or ADHD) and beta-blockers (for various heart conditions).

    These issues need to be considered before labelling the problem as insomnia. However, it’s important to keep taking your medication as prescribed and discuss any concerns with your doctor.

    Getting the right help

    If your sleep is worrying you, the best first step is to see your GP. They can help rule out other causes, review your medications, or refer you for a sleep study if needed.

    However, once insomnia becomes frequent, chronic (long term) and distressing, you can worry too much about your sleep, constantly check or track your sleep, or try too hard to sleep, for instance by spending too much time in bed. These psychological and behavioural mechanisms can backfire, and make good sleep even less likely.

    That’s why “cognitive behavioural therapy for insomnia” (or CBT-I) is recommended as the first-line treatment.

    This is more effective, and longer-lasting than sleeping pills. This therapy is available via specially trained GPs, and sleep psychologists. You can take part in person or online.

    In the meantime

    If you’re in a rough patch of sleep:

    • remind yourself that short runs of poor sleep usually settle on their own
    • avoid lying in bed panicking if you wake at 3.30am. Instead, step out of bed or use the time in a way that feels restful
    • keep a consistent wake-up time, even after a poor night. Try to get some morning sunlight to reset your body clock
    • make sure you’re putting aside the right amount of time for sleep – not too little, not too much.

    Amelia Scott, Honorary Affiliate and Clinical Psychologist at the Woolcock Institute of Medical Research, and Macquarie University Research Fellow, Macquarie University

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • Save Your Back With Strong Glutes

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    Your glutes really are the seat of your power, and without strong glutes, your back tries to pick up the slack. Unfortunately, without strong glutes, your back can only do that with questionable body mechanics, and guess who suffers for it?

    So instead…

    The brain-butt connection

    It might sound funny, but it’s true! Most squats don’t train the glutes through a proper neural connection; they over-recruit the quads and other nearby muscles, meaning the glutes have no reason to get stronger.

    This trainer (Elisi Wolf)’s own lower-back injury (sacroiliac joint ligament tear) forced a shift away from traditional squats. That injury led to years of experimenting, and she found that Bulgarian split squats are different in this regard.

    How they’re different: Bulgarian split squats reduce distractions to the brain, letting it build stronger neural pathways to the glutes, in contrast to how traditional squats divide focus between multiple large muscle groups and turn the movement into a calorie-burning, full-body effort rather than isolated glute training.

    Additionally, because Bulgarian split squats use lighter weight and one leg at a time, they allow better control, slower reps, and more focused muscle engagement—all key for glute hypertrophy (and thus: strengthening).

    For more on all of this, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like:

    Bulgarian Split Squats: How To Get The Best Glute Strength & Size ← this one details how, specifically, to do Bulgarian split squats correctly (and avoid common mistakes)

    Take care!

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  • Menopause can bring increased cholesterol levels and other heart risks. Here’s why and what to do about it

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Menopause is a natural biological process that marks the end of a woman’s reproductive years, typically between 45 and 55. As women approach or experience menopause, common “change of life” concerns include hot flushes, sweats and mood swings, brain fog and fatigue.

    But many women may not be aware of the long-term effects of menopause on the heart and blood vessels that make up the cardiovascular system. Heart disease accounts for 35% of deaths in women each year – more than all cancers combined.

    What should women – and their doctors – know about these risks?

    Hormones protect hearts – until they don’t

    As early as 1976, the Framingham Heart Study reported more than twice the rates of cardiovascular events in postmenopausal than pre-menopausal women of the same age. Early menopause (younger than age 40) also increases heart risk.

    Before menopause, women tend to be protected by their circulating hormones: oestrogen, to a lesser extent progesterone and low levels of testosterone.

    These sex hormones help to relax and dilate blood vessels, reduce inflammation and improve lipid (cholesterol) levels. From the mid-40s, a decline in these hormone levels can contribute to unfavourable changes in cholesterol levels, blood pressure and weight gain – all risk factors for heart disease.

    Speedkingz/Shutterstock

    4 ways hormone changes impact heart risk

    1. Dyslipidaemia– Menopause often involves atherogenic changes – an unhealthy imbalance of lipids in the blood, with higher levels of total cholesterol, triglycerides, and low-density lipoprotein (LDL-C), dubbed the “bad” cholesterol. There are also reduced levels of high-density lipoprotein (HDL-C) – the “good” cholesterol that helps remove LDL-C from blood. These changes are a major risk factor for heart attack or stroke.

    2. Hypertension – Declines in oestrogen and progesterone levels during menopause contribute to narrowing of the large blood vessels on the heart’s surface, arterial stiffness and raise blood pressure.

    3. Weight gain – Females are born with one to two million eggs, which develop in follicles. By the time they stop ovulating in midlife, fewer than 1,000 remain. This depletion progressively changes fat distribution and storage, from the hips to the waist and abdomen. Increased waist circumference (greater than 80–88 cm) has been reported to contribute to heart risk – though it is not the only factor to consider.

    4. Comorbidities – Changes in body composition, sex hormone decline, increased food consumption, weight gain and sedentary lifestyles impair the body’s ability to effectively use insulin. This increases the risk of developing metabolic syndromes such as type 2 diabetes.

    While risk factors apply to both genders, hypertension, smoking, obesity and type 2 diabetes confer a greater relative risk for heart disease in women.

    So, what can women do?

    Every woman has a different level of baseline cardiovascular and metabolic risk pre-menopause. This is based on their genetics and family history, diet, and lifestyle. But all women can reduce their post-menopause heart risk with:

    • regular moderate intensity exercise such as brisk walking, pushing a lawn mower, riding a bike or water aerobics for 30 minutes, four or five times every week
    • a healthy heart diet with smaller portion sizes (try using a smaller plate or bowl) and more low-calorie, nutrient-rich foods such as vegetables, fruit and whole grains
    • plant sterols (unrefined vegetable oil spreads, nuts, seeds and grains) each day. A review of 14 clinical trials found plant sterols, at doses of at least 2 grams a day, produced an average reduction in serum LDL-C (bad cholesterol) of about 9–14%. This could reduce the risk of heart disease by 25% in two years
    • less unhealthy (saturated or trans) fats and more low-fat protein sources (lean meat, poultry, fish – especially oily fish high in omega-3 fatty acids), legumes and low-fat dairy
    • less high-calorie, high-sodium foods such as processed or fast foods
    • a reduction or cessation of smoking (nicotine or cannabis) and alcohol
    • weight-gain management or prevention.
    Women walking together outdoors with exercise clothes and equipment
    Exercise can reduce post-menopause heart disease risk. Monkey Business Images/Shutterstock

    What about hormone therapy medications?

    Hormone therapy remains the most effective means of managing hot flushes and night sweats and is beneficial for slowing the loss of bone mineral density.

    The decision to recommend oestrogen alone or a combination of oestrogen plus progesterone hormone therapy depends on whether a woman has had a hysterectomy or not. The choice also depends on whether the hormone therapy benefit outweighs the woman’s disease risks. Where symptoms are bothersome, hormone therapy has favourable or neutral effects on coronary heart disease risk and medication risks are low for healthy women younger than 60 or within ten years of menopause.

    Depending on the level of stroke or heart risk and the response to lifestyle strategies, some women may also require medication management to control high blood pressure or elevated cholesterol levels. Up until the early 2000s, women were underrepresented in most outcome trials with lipid-lowering medicines.

    The Cholesterol Treatment Trialists’ Collaboration analysed 27 clinical trials of statins (medications commonly prescribed to lower cholesterol) with a total of 174,000 participants, of whom 27% were women. Statins were about as effective in women and men who had similar risk of heart disease in preventing events such as stroke and heart attack.

    Every woman approaching menopause should ask their GP for a 20-minute Heart Health Check to help better understand their risk of a heart attack or stroke and get tailored strategies to reduce it.

    Treasure McGuire, Assistant Director of Pharmacy, Mater Health SEQ in conjoint appointment as Associate Professor of Pharmacology, Bond University and as Associate Professor (Clinical), The University of Queensland

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • The Hidden Danger Of Sorbitol

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Sorbitol, a common sweetener in many foods, is a sugar alcohol, which means it is neither a sugar nor an alcohol in the sense that most people understand those words, but chemists have their classification systems and sorbitol’s chemical structure is such that, with its hydroxyl groups each attached to one carbon atom, it’s a sugar alcohol.

    So, what’s the problem?

    We’ll cut right to the chase: it can be indirectly quite harmful to the liver.

    To understand why, first understand how fructose is so bad for the liver. It’s so bad, because while glucose and fructose (the monosaccharides found in equal parts in the disaccharide that is sucrose, i.e. table sugar) both ultimately get converted into glycogen (if not used immediately for energy), but for fructose, this happens mostly* in the liver, which a) taxes it b) goes very unregulated by the pancreas, causing potentially dangerous blood sugar spikes.

    This has several interesting effects:

    • Because fructose doesn’t directly affect insulin levels, it doesn’t cause insulin insensitivity (yay)
    • Because fructose doesn’t directly affect insulin levels, this leaves hyperglycemia untreated (oh dear)
    • Because fructose is metabolized by the liver and converted to glycogen which is stored there, it’s one of the main contributors to non-alcoholic fatty liver disease (at this point, we’re retracting our “yay”)

    Read more: Fructose and sugar: a major mediator of non-alcoholic fatty liver disease

    *”Mostly” in the liver being about 80% in the liver. The remaining 20%ish is processed by the kidneys, where it contributes to kidney stones instead. So, still not fabulous.

    Now know this: sorbitol can be converted very quickly and easily into fructose (oops!)

    Researchers (Dr. Madelyn Jackstadt et al.) found that if you have sufficient quantities of certain Aeromonas bacteria, they degrade sorbitol into harmless byproducts, but without them sorbitol passes to the liver, where it is converted into fructose and fructose derivatives.

    However, you cannot rely on “well, I’m pretty sure my gut is in good shape”, because excess sorbitol—whether eaten directly or generated from high glucose intake—can overwhelm even those beneficial bacteria.

    You can find the paper itself, here: Intestine-derived sorbitol drives steatotic liver disease in the absence of gut bacteria

    What should we use instead?

    Honestly, there are no sweeteners that we’re aware of that have no drawbacks.

    Simply sweetness itself can cause problems: we can build tolerance to sweetness. Many sugar substitutes are many times (in some cases, hundreds of times) sweeter than sugar. This leads to people craving increasingly sweeter foods for the same experiential sweetness level.

    Because of this, the World Health Organization has released a report offering guidance regards the use of sugar-free sweeteners.

    In a nutshell, the guidance is: don’t

    Nevertheless, if you really want to, we previously did a rundown on:

    • Sucrose (metabolic problems)
    • Sucralose (genotoxic)
    • Erythritol (ischemiagenic)
    • Xylitol (gut disruptor)
    • Acesulfame K (gut disruptor)
    • Stevia (strong risk of sweetness tolerance problem)
    • Glycine (beneficial in moderation, sweetness problem though)

    For more details than those one-or-few-word summaries, see: What’s The Healthiest Sweetener?

    We’ve also talked about: The Fascinating Truth About Aspartame, Cancer, & Neurotoxicity

    …which covers how the most popular beliefs about aspartame are myths, and in large part stemming from a single viral hoax chain letter in the 90s!

    Want to do more for your liver?

    Consider: N-Acetyl Cysteine For The Liver & More

    Or if you prefer a purely dietary approach, then: How To Unfatty A Fatty Liver

    Take care!

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  • Stiff After Sitting? Before You Stand Up, Do This For Easier First Steps

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Dr. Alyssa Kuhn, arthritis expert, shows us how:

    Are you sitting comfortably? Then we’ll begin…

    The answer is as simple as a leg extension warm-up: straighten your knee by kicking your leg out, or sliding your heel forwards, before standing.

    This need not be overly vigorous; keep your movement gentle and within a comfortable range, while aiming to get your knee as straight as possible without forcing it. Even a few repetitions before standing can noticeably improve how your first steps feel!

    The goal here is modest: just move your joints briefly before standing, rather than treating this like a full workout to make standing feel smoother and easier.

    That’s it; that’s the trick!

    For a visual demonstration though, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like:

    How To Get Out Of Any Low Chair Without Help ← for anyone wondering “but what if it’s a low chair?”

    Take care!

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  • Bamboo Shoots vs Onion – Which is Healthier?

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    Our Verdict

    When comparing bamboo shoots to onions, we picked the bamboo.

    Why?

    In terms of macros, bamboo has more protein and fiber, while onion has more carbs, meaning that so far as round 1 goes, bamboo shoots… and scores!

    In the category of vitamins, bamboo has more of vitamins A, B1, B2, B3, B5, B6, B7, and E, while onion has more of vitamins B9, C, and K, giving bamboo an 8:3 win here.

    Looking at minerals, bamboo has more copper, iron, manganese, phosphorus, potassium, selenium, and zinc, while onion has more calcium and magnesium—another win for bamboo, 7:2 this time.

    Adding up the sections makes for a clear overall win for bamboo shoots, but by all means enjoy either or both, as diversity is good!

    Want to learn more?

    You might like:

    What’s Your Plant Diversity Score?

    Enjoy!

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