Dr. Greger’s Daily Dozen

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Give Us This Day Our Daily Dozen

This is Dr. Michael Greger. He’s a physician-turned-author-educator, and we’ve featured him and his work occasionally over the past year or so:

But what we’ve not covered, astonishingly, is one of the things for which he’s most famous, which is…

Dr. Greger’s Daily Dozen

Based on the research in the very information-dense tome that his his magnum opus How Not To Die (while it doesn’t confer immortality, it does help avoid the most common causes of death), Dr. Greger recommends that we take care to enjoy each of the following things per day:

Beans

  • Servings: 3 per day
  • Examples: ½ cup cooked beans, ¼ cup hummus

Greens

  • Servings: 2 per day
  • Examples: 1 cup raw, ½ cup cooked

Cruciferous vegetables

  • Servings: 1 per day
  • Examples: ½ cup chopped, 1 tablespoon horseradish

Other vegetables

  • Servings: 2 per day
  • Examples: ½ cup non-leafy vegetables

Whole grains

  • Servings: 3 per day
  • Examples: ½ cup hot cereal, 1 slice of bread

Berries

  • Servings: 1 per day
  • Examples: ½ cup fresh or frozen, ¼ cup dried

Other fruits

  • Servings: 3 per day
  • Examples: 1 medium fruit, ¼ cup dried fruit

Flaxseed

  • Servings: 1 per day
  • Examples: 1 tablespoon ground

Nuts & (other) seeds

  • Servings: 1 per day
  • Examples: ¼ cup nuts, 2 tablespoons nut butter

Herbs & spices

  • Servings: 1 per day
  • Examples: ¼ teaspoon turmeric

Hydrating drinks

  • Servings: 60 oz per day
  • Examples: Water, green tea, hibiscus tea

Exercise

  • Servings: Once per day
  • Examples: 90 minutes moderate or 40 minutes vigorous

Superficially it seems an interesting choice to, after listing 11 foods and drinks, have the 12th item as exercise but not add a 13th one of sleep—but perhaps he quite reasonably expects that people get a dose of sleep with more consistency than people get a dose of exercise. After all, exercise is mostly optional, whereas if we try to skip sleep for too long, our body will force the matter for us.

Further 10almonds notes:

Enjoy!

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  • Krill Oil vs Fish Oil – Which is Healthier?

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    Our Verdict

    When comparing krill oil to fish oil, we picked the krill oil.

    Why?

    Both of these products are good sources of omega-3 fatty acids EPA and DHA, and for the specific brand depicted above, in both cases 2 softgels will give you the recommended daily amount (which is generally held to be 250–500mg combined omega-3s per day).

    This brand’s fish oil gives more (640mg combined omega-3s per 2 softgels, to the same brand’s krill oil’s 480mg per 2 softgels), but since the krill oil is already in the high end of RDA territory, the excess beyond the RDA is not helpful, and not a huge factor. More quantity is not always better, when the body can only process so much at a time.

    However, the krill oil gives some extra things that the fish oil doesn’t:

    • Astaxanthin, a “super-antioxidant”
    • and neuroprotectant, heart-healthy phospholipids

    Additional considerations

    We have declared “the winner” based on health considerations only. That’s a sticking point for us in all our writings; we’ll occasionally look at and mention other factors, but we know that health is what you’re here for, so that’s what we’ll always treat as most critical.

    However, in case these factors may interest you and/or influence you to one or the other:
    •⁠ ⁠The fish oil is about 30% cheaper financially
    •⁠ ⁠The krill oil is a lot more sustainable environmentally

    Back to the health science…

    Read more:
    •⁠ ⁠What Omega-3 Fatty Acids Really Do For Us
    •⁠ ⁠Astaxanthin: Super-Antioxidant & Neuroprotectant

    Want some? Here for your convenience are some example products on Amazon:

    Krill oil | Fish oil

    (brands available will vary per region, but now you know what to look out for on the labels!)

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  • 10,000 Steps, 30 Days, 4 Changes

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Ariel wasn’t the most active person, and took on a “30 day challenge” to do the commonly-prescribed 10,000 steps per day—without adjusting her diet or doing any other exercise. How much of a difference does it make, really?

    Stepping onwards

    The 4 main things that she found changed for her weren’t all what she expected:

    • Weight loss yes, but only marginally: she lost 3 lbs in a month, which did nevertheless make a visible difference. We might hypothesize that part of the reason for the small weight loss and yet visible difference is that she gained a little muscle, and the weight loss was specifically shifting away from a cortisol-based fat distribution, to a more healthy fat distribution.
    • Different eating habits: she felt less hungry and craved less sugar. This likely has less to do with calorie consumption, and more to do with better insulin signalling.
    • Increased energy and improved mood: these are going together in one item, because she said “4 things”, but really they are two related things. So, consider one of them a bonus item! In any case, she felt more energized and productive, and less reliant on caffeine.
    • Improved sleep: or rather, at first, disrupted sleep, and then slept better and stayed better. A good reminder that changes for the better don’t always feel better in the first instance!

    To hear about it in her own words, and see the before and after pictures, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like to read:

    Take care!

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  • Antibiotics? Think Thrice

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Antibiotics: Useful Even Less Often Than Previously Believed (And Still Just As Dangerous)

    You probably already know that antibiotics shouldn’t be taken unless absolutely necessary. Not only does taking antibiotics frivolously increase antibiotic resistance (which is bad, and kills people), but also…

    It’s entirely possible for the antibiotics to not only not help, but instead wipe out your gut’s “good bacteria” that were keeping other things in check.

    Those “other things” can include fungi like Candida albicans.

    Candida, which we all have in us to some degree, feeds on sugar (including the sugar formed from breaking down alcohol, by the way) and refined carbs. Then it grows, and puts its roots through your intestinal walls, linking with your neural system. Then it makes you crave the very things that will feed it and allow it to put bigger holes in your intestinal walls.

    Don’t believe us? Read: Candida albicans-Induced Epithelial Damage Mediates Translocation through Intestinal Barriers

    (That’s scientist-speak for “Candida puts holes in your intestines, and stuff can then go through those holes”)

    And as for how that comes about, it’s like we said:

    See also: Candida albicans as a commensal and opportunistic pathogen in the intestine

    That’s not all…

    And that’s just C. albicans, never mind things like C. diff. that can just outright kill you easily.

    We don’t have room to go into everything here, but you might like to check out:

    Four Ways Antibiotics Can Kill You

    It gets worse (now comes the new news)

    So, what are antibiotics good for? Surely, for clearing up chesty coughs, lower respiratory tract infections, right? It’s certainly one of the two things that antibiotics are most well-known for being good at and often necessary for (the other being preventing/treating sepsis, for example in serious and messy wounds).

    But wait…

    A large, nationwide (US) observational study of people who sought treatment in primary or urgent care settings for lower respiratory tract infections found…

    (drumroll please)

    the use of antibiotics provided no measurable impact on the severity or duration of coughs even if a bacterial infection was present.

    Read for yourself:

    Antibiotics Not Associated with Shorter Duration or Reduced Severity of Acute Lower Respiratory Tract Infection

    And in the words of the lead author of that study,

    ❝Lower respiratory tract infections tend to have the potential to be more dangerous, since about 3% to 5% of these patients have pneumonia. But not everyone has easy access at an initial visit to an X-ray, which may be the reason clinicians still give antibiotics without any other evidence of a bacterial infection.❞

    ~ Dr. Daniel Merenstein

    So, what’s to be done about this? On a large scale, Dr. Merenstein recommends:

    ❝Serious cough symptoms and how to treat them properly needs to be studied more, perhaps in a randomized clinical trial as this study was observational and there haven’t been any randomized trials looking at this issue since about 2012.❞

    ~ Dr. Daniel Merenstein

    This does remind us that, while not a RCT, there is a good ongoing observational study that everyone with a smartphone can participate in:

    Dr. Peter Small’s medical AI: “The Cough Doctor”

    In the meantime, he advises that when COVID and SARS have been ruled out, then “basic symptom-relieving medications plus time brings a resolution to most people’s infections”.

    You can read a lot more detail here:

    Antibiotics aren’t effective for most lower tract respiratory infections

    In summary…

    Sometimes, antibiotics really are a necessary and life-saving medication. But most of the time they’re not, and given their great potential for harm, they may be best simultaneously viewed as the very dangerous threat they also are, and used only when those “heavy guns” are truly what’s required.

    Take care!

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  • Statins: Study Insights

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    It’s Q&A Day at 10almonds!

    Q: Can you let us know about more studies that have been done on statins? Are they really worth taking?

    That is a great question! We imagine it might have been our recent book recommendation that prompted it? It’s quite a broad question though, so we’ll do that as a main feature in the near future!

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  • This Is Your Brain on Music – by Dr. Daniel Levitin

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Music has sometimes been touted as having cognitive benefits, by its practice and even by the passive experience of it. But what’s the actual science of it?

    Dr. Levitin, an accomplished musician and neuroscientist, explores and explains.

    We learn about how music in all likelihood allowed our ancestors to develop speech, something that set us apart (and ahead!) as a species. How music was naturally-selected-for in accordance with its relationship with health. How processing music involves almost every part of the brain. How music pertains specifically to memory. And more.

    As a bonus, as well as explaining a lot about our brain, this book offers those of us with limited knowledge of music theory a valuable overview of the seven main dimensions of music, too.

    Bottom line: if you’d like to know more about the many-faceted relationship between music and cognitive function, this is a top-tier book about such.

    Click here to check out “This Is Your Brain On Music”, and learn more about yours!

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  • Too much or too little testosterone?

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    One Man’s Saw Palmetto Is Another Woman’s Serenoa Repens…

    Today we’re going to look at saw palmetto. So, first:

    What is it?

    Saw palmetto is a type of palm native to the southeastern United States. Its scientific name is “Serenoa repens”, so if that name appears in studies we cite, it’s the same thing. By whichever name, it’s widely enjoyed as a herbal supplement.

    Why do people take it?

    Here’s where it gets interesting, because people take it for some completely opposite reasons…

    Indeed, searching for it on the Internet will cause Google to suggest “…for men” and “…for women” as the top suggestions.

    That’s because it works on testosterone, and testosterone can be a bit of a double-edged sword, so some people want to increase or decrease certain testosterone-related effects on their body.

    And it works for both! Here be science:

    • Testosterone (henceforth, “T”) is produced in the human body.
      • Yes, all human bodies, to some extent.
    • An enzyme called 5-alpha-reductase converts T in to DHT (dihydrogen testosterone)
    • DHT is a much more potent androgen (masculinizing agent) than T alone, such that its effects are often unwanted, including:
      • Enlarged prostate (if you have one)
      • Hair loss (especially in men)
      • New facial hair growth (usually unwanted by women)
        • Women are more likely to get this due to PCOS and/or the menopause

    To avoid those effects, you really want less of your T to be converted into DHT.

    Saw palmetto is a 5α-reductase inhibitor, so if you take it, you’ll have less DHT, and you’ll consequently lose less hair, have fewer prostate problems, etc.

    Read: Determination of the potency of a novel saw palmetto supercritical CO2 extract (SPSE) for 5α-reductase isoform II inhibition using a cell-free in vitro test system

    ^The above study showed that saw palmetto extract performed comparably to finasteride. Finasteride is the world’s main go-to prescription drug for treating enlarged prostate and/or hair loss.

    See also: Natural Hair Supplement: Friend or Foe? Saw Palmetto, a Systematic Review in Alopecia

    Hair today… Growing tomorrow!

    So, what was that about increasing T levels?

    Men usually suffer declining T levels as they get older, with a marked drop around the age of 45. With lower T comes lower energy, lower mood, lower libido, erectile dysfunction, etc.

    Guess what… It’s T that’s needed for those things, not DHT. So if you block the conversion of T to DHT, you’ll have higher blood serum T levels, higher energy, higher mood, higher libido, and all that.

    Read: Standardized Saw Palmetto Extract Directly and Indirectly Affects Testosterone Biosynthesis and Spermatogenesis

    (the above assumes you have testicles, without which, your T levels will certainly not increase)

    Saw Palmetto Against Enlarged Prostate?

    With higher DHT levels in mid-late life, prostate enlargement (benign prostatic hyperlasia) can become a problem for many men. The size of that problem ranges from urinary inconvenience (common, when the prostate presses against the bladder) to prostate cancer (less common, much more serious). Saw palmetto, like other 5α-reductase inhibitors such as finasteride, may be used to prevent or treat this.

    Wondering how safe/reliable it is? We found a very high-quality fifteen-year longitudinal observational study of the use of saw palmetto, and it found:

    ❝The 15 years’ study results suggest that taking S. repens plant extract continuously at a daily dose of 320 mg is an effective and safe way to prevent the progression of benign prostatic hyperplasia.❞

    Read: 15 years’ survey of safety and efficacy of Serenoa repens extract in benign prostatic hyperplasia patients with risk of progression

    Want a second opinion? We also found a 10-year study (by different researchers with different people taking it), which reached the same conclusion:

    ❝The results of study showed the absence of progression, both on subjective criteria (IPSS, and QoL scores), and objective criteria (prostate volume, the rate of urination, residual urine volume). Furthermore, patients had no undesirable effects directly related to the use of this drug.❞

    • IPSS = International Prostate Symptom Score
    • QoL = Quality of Life

    Read: The results of the 10-year study of efficacy and safety of Serenoa repens extract in patients at risk of progression of benign prostatic hyperplasia

    But wait a minute; I, a man over the age of 45 with potentially declining T levels but a fabulous beard, remember that you said just a minute ago that saw palmetto is used by women to avoid having facial hair; I don’t want to lose mine!

    You won’t. Once your facial hair follicles were fully developed and activated during puberty, they’ll carry on doing what they do for life. That’s no longer regulated by hormones once they’re up and running.

    The use of saw palmetto can only be used to limit facial hair if caught early—so it’s more useful at the onset of menopause, for those who have (or will have) such, or else upon the arrival of PCOS symptoms or hirsuitism from some other cause.

    Take The Test!

    Do you have a prostate, and would like to know your IPSS score, and what that means for your prostate health?

    Take The Test Here!

    (takes 1 minute, no need to pee or go probing for anything)

    Bottom Line on Saw Palmetto

    • It blocks the conversion of T into DHT
    • It will increase blood serum T levels, thus boosting mood, energy, libido, etc in men (who typically have more T, but whose T levels decline with age)
    • It will decrease DHT levels, thus limiting hair loss (especially in men) and later-life new facial hair growth (especially in women).
    • It can be used to prevent or treat prostate enlargement
    • Bonus: it’s a potent antioxidant and thus reduces general inflammation (in everyone)

    Want To Try Saw Palmetto?

    We don’t sell it (or anything else), but for your convenience…

    Click here to check out saw palmetto on Amazon!

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