Why Diets Make Us Fat – by Dr. Sandra Aamodt

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It’s well-known that crash-dieting doesn’t work. Restrictive diets will achieve short-term weight loss, but it’ll come back later. In the long term, weight creeps slowly upwards. Why?

Dr. Sandra Aamodt explores the science and sociology behind this phenomenon, and offers an evidence-based alternative.

A lot of the book is given over to explanations of what is typically going wrong—that is the title of the book, after all. From metabolic starvation responses to genetics to the negative feedback loop of poor body image, there’s a lot to address.

However, what alternative does she propose?

The book takes us on a shift away from focusing on the numbers on the scale, and more on building consistent healthy habits. It might not feel like it if you desperately want to lose weight, but it’s better to have healthy habits at any weight, than to have a wreck of physical and mental health for the sake of a lower body mass.

Dr. Aamodt lays out a plan for shifting perspectives, building health, and letting weight loss come by itself—as a side effect, not a goal.

In fact, as she argues (in agreement with the best current science, science that we’ve covered before at 10almonds, for that matter), that over a certain age, people in the “overweight” category of BMI have a reduced mortality risk compared to those in the “healthy weight” category. It really underlines how there’s no point in making oneself miserably unhealthy with the end goal of having a lighter coffin—and getting it sooner.

Bottom line: will this book make you hit those glossy-magazine weight goals by your next vacation? Quite possibly not, but it will set you up for actually healthier living, for life, at any weight.

Click here to check out Why Diets Make Us Fat, and live healthier and better!

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  • “Unfuck Your Body” In Under 10 Minutes A Day!

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    There’s a lot that can go wrong with mobility, but fortunately, a few compound exercises will take care of most parts of it:

    Full Body Mobility Routine

    Eleven exercises, 10 minutes, follow-along video if you want it!

    Kneeling side bend stretch:

    • Targets obliques, lats, hip flexors, and spinal mobility.
    • 10 reps total, focusing on stability and core engagement.

    Seated ankle stretch:

    • This one’s for ankle mobility and deep squat comfort.
    • 10 reps, adjust intensity by leaning forward or pressing on knees.

    Deep squat with prayer stretch:

    • Improves hip, ankle, and lower back flexibility.
    • 10 reps, maintain an upright chest and push knees outward.

    Deep squat with high reach:

    • Boosts thoracic mobility, hip, and ankle flexibility.
    • 5 reps per side, focus on spinal rotation and open chest.

    Deep shoulder stretch:

    • Improves overhead mobility and shoulder tension relief.
    • 10 reps in a child’s pose position with a forward reach.

    Frog rocks:

    • Opens hip abductors, groin, and inner thighs.
    • 10 reps, keep spine neutral and adjust knee position if needed.

    World’s greatest stretch” (with variations):

    • This is great for hip, spine, and shoulder mobility.
    • 5 reps per side, integrates a deep lunge and rotational movements.

    Hamstring stretch (from lunge position):

    • Focus on hamstring and calf flexibility.
    • 5 reps, maintain hands on the ground and shift hips back.

    Pigeon stretch with forward crawl:

    • Opens hips, glutes, and lower back.
    • 5 reps per side, adjust foot placement if knee discomfort occurs.

    Cat-cow stretch:

    • Mobilizes spine, improves posture, and relieves back tension.
    • 10 reps, synchronize movement with breath.

    Couch stretch:

    • Targets hip flexors and quadriceps mobility.
    • 5 reps per side, add a forward lunge for a deeper stretch.

    For more on each of these plus visual demonstrations, enjoy:

    Click Here If The Embedded Video Doesn’t Load Automatically!

    Want to learn more?

    You might also like:

    5 Exercises That Fix 95% Of Your Problems

    Take care!

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  • A Correction, And A New, Natural Way To Boost Daily Energy Levels

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    It’s Q&A Day at 10almonds!

    Have a question or a request? You can always hit “reply” to any of our emails, or use the feedback widget at the bottom!

    In cases where we’ve already covered something, we might link to what we wrote before, but will always be happy to revisit any of our topics again in the future too—there’s always more to say!

    As ever: if the question/request can be answered briefly, we’ll do it here in our Q&A Thursday edition. If not, we’ll make a main feature of it shortly afterwards!

    So, no question/request too big or small

    First: a correction and expansion!

    After yesterday’s issue of 10almonds covering breast cancer risks and checks, a subscriber wrote to say, with regard to our opening statement, which was:

    Anyone (who has not had a double mastectomy, anyway) can get breast cancer”

    ❝I have been enjoying your newsletter. This statement is misleading and should have a disclaimer that says even someone who has had a double mastectomy can get breast cancer, again. It is true and nothing…nothing is 100% including a mastectomy. I am a 12 year “thriver” (I don’t like to use the term survivor) who has had a double mastectomy. I work with a local hospital to help newly diagnosed patients deal with their cancer diagnosis and the many decisions that follow. A double mastectomy can help keep recurrence from happening but there are no guarantees. I tried to just delete this and let it go but it doesn’t feel right. Thank you!❞

    Thank you for writing in about this! We wouldn’t want to mislead, and we’re always glad to hear from people who have been living with conditions for a long time, as (assuming they are a person inclined to learning) they will generally know topics far more deeply than someone who has researched it for a short period of time.

    Regards a double mastectomy (we’re sure you know this already, but noting here for greater awareness, prompted by your message), a lot of circumstances can vary. For example, how far did a given cancer spread, and especially, did it spread to the lymph nodes at the armpits? And what tissue was (and wasn’t) removed?

    Sometimes a bilateral prophylactic mastectomy will leave the lymph nodes partially or entirely intact, and a cancer could indeed come back, if not every last cancerous cell was removed.

    A total double mastectomy, by definition, should have removed all tissue that could qualify as breast tissue for a breast cancer, including those lymph nodes. However, if the cancer spread unnoticed somewhere else in the body, then again, you’re quite correct, it could come back.

    Some people have a double mastectomy without having got cancer first. Either because of a fear of cancer due to a genetic risk (like Angelina Jolie), or for other reasons (like Elliot Page).

    This makes a difference, because doing it for reasons of cancer risk may mean surgeons remove the lymph nodes too, while if that wasn’t a factor, surgeons will tend to leave them in place.

    In principle, if there is no breast tissue, including lymph nodes, and there was no cancer to spread, then it can be argued that the risk of breast cancer should now be the same “zero” as the risk of getting prostate cancer when one does not have a prostate.

    But… Surgeries are not perfect, and everyone’s anatomy and physiology can differ enough from “textbook standard” that surprises can happen, and there’s almost always a non-zero chance of certain health outcomes.

    For any unfamiliar, here’s a good starting point for learning about the many types of mastectomy, that we didn’t go into in yesterday’s edition. It’s from the UK’s National Health Service:

    NHS: Mastectomy | Types of Mastectomy

    And for the more sciency-inclined, here’s a paper about the recurrence rate of cancer after a prophylactic double mastectomy, after a young cancer was found in one breast.

    The short version is that the measured incidence rate of breast cancer after prophylactic bilateral mastectomy was zero, but the discussion (including notes about the limitations of the study) is well worth reading:

    Breast Cancer after Prophylactic Bilateral Mastectomy in Women with a BRCA1 or BRCA2 Mutation

    ❝[Can you write about] the availability of geriatric doctors Sometimes I feel my primary isn’t really up on my 70 year old health issues. I would love to find a doctor that understands my issues and is able to explain them to me. Ie; my worsening arthritis in regards to food I eat; in regards to meds vs homeopathic solutions.! Thanks!❞

    That’s a great topic, worthy of a main feature! Because in many cases, it’s not just about specialization of skills, but also about empathy, and the gap between studying a condition and living with a condition.

    About arthritis, we’re going to do a main feature specifically on that quite soon, but meanwhile, you might like our previous article:

    Keep Inflammation At Bay (arthritis being an inflammatory condition)

    As for homeopathy, your question prompts our poll today!

    (and then we’ll write about that tomorrow)

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  • Peach vs Strawberries – Which is Healthier?

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    Our Verdict

    When comparing peaches to strawberries, we picked the strawberries.

    Why?

    In terms of macros, peaches have more carbs while strawberries have more fiber. The differences aren’t huge, but are at least compelling enough to call this round a nominal win for strawberries.

    In the category of vitamins, peaches have more of vitamins A, B2, B3, and E, while strawberries have more of vitamins B6, B9, and C, making this round a marginal 4:3 win for peaches.

    When it comes to minerals, peaches have more copper, potassium, and zinc, while strawberries have more calcium, copper, iron, magnesium, manganese, phosphorus, and selenium. A clear win for strawberries.

    Looking at other properties, it’s worth noting that peaches have some anticancer properties that strawberries don’t (so far as we know), while strawberries have rather more polyphenols in general. We’re calling this round a tie.

    Adding up the sections makes for an overall win for strawberries, but it was very close, so by all means enjoy either or both!

    Want to learn more?

    You might like:

    Top 8 Fruits That Prevent & Kill Cancer

    Enjoy!

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  • Eat To Beat Cancer

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Controlling What We Can, To Avoid Cancer

    Every time a cell in our body is replaced, there’s a chance it will be cancerous. Exactly what that chance is depends on very many factors. Some of them we can’t control; others, we can.

    Diet is a critical, modifiable factor

    We can’t choose, for example, our genes. We can, for the most part, choose our diet. Why “for the most part”?

    • Some people live in a food desert (the Arctic Circle is a good example where food choices are limited by supply)
    • Some people have dietary restrictions (whether by health condition e.g. allergy, intolerance, etc or by personal-but-unwavering choice, e.g. vegetarian, vegan, kosher, halal, etc)

    But for most of us, most of the time, we have a good control over our diet, and so that’s an area we can and should focus on.

    Choose your animal protein wisely

    If you are vegan, you can skip this section. If you are not, then the short version is:

    • Fish: almost certainly fine
    • Poultry: the jury is out; data is leaning towards fine, though
    • Red meat: significantly increased cancer risk
    • Processed meat: significantly increased cancer risk

    For more details (and a run-down on the science behind the above super-summarized version):

    Skip The Ultra-Processed Foods

    Ok, so this one’s probably not a shocker in its simplest form:

    ❝Studies are showing us is that not only do the ultraprocessed foods increase the risk of cancer, but that after a cancer diagnosis such foods increase the risk of dying❞

    Source: Is there a connection between ultraprocessed food and cancer?

    There’s an unfortunate implication here! If you took the previous advice to heart and cut out [at least some] meat, and/but then replaced that with ultra-processed synthetic meat, then this was not a great improvement in cancer risk terms.

    Ultra-processed meat is worse than unprocessed, regardless of whether it was from an animal or was synthetic.

    In other words: if you buy textured soy pieces (a common synthetic meat), it pays to look at the ingredients, because there’s a difference between:

    • INGREDIENTS: SOY
    • INGREDIENTS: Rehydrated Textured SOY Protein (52%), Water, Rapeseed Oil, SOY Protein Concentrate, Seasoning (SULPHITES) (Dextrose, Flavourings, Salt, Onion Powder, Food Starch Modified, Yeast Extract, Colour: Red Iron Oxide), SOY Leghemoglobin, Fortified WHEAT Flour (WHEAT Flour, Calcium Carbonate, Iron, Niacin, Thiamin), Bamboo Fibre, Methylcellulose, Tomato Purée, Salt, Raising Agent: Ammonium Carbonates

    Now, most of those original base ingredients are/were harmless per se (as are/were the grapes in wine—before processing into alcohol), but it has clearly been processed to Hell and back to do all that.

    Choose the one that just says “soy”. Or eat soybeans. Or other beans. Or lentils. Really there are a lot of options.

    About soy, by the way…

    There is (mostly in the US, mostly funded by the animal agriculture industry) a lot of fearmongering about soy. Which is ironic, given the amount of soy that is fed to livestock to be fed to humans, but it does bear addressing:

    ❝Soy foods are safe for all cancer patients and are an excellent source of plant protein. Studies show soy may improve survival after breast cancer❞

    Source: Food risks and cancer: What to avoid

    (obviously, if you have a soy allergy then you should not consume soy—for most people, the above advice stands, though)

    Advanced Glycation End-Products

    These (which are Very Bad™ for very many things, including cancer) occur specifically as a result of processing animal proteins and fats.

    Note: not even necessarily ultra-processing, just processing can do it. But ultra-processing is worse. What’s the difference, you wonder?

    The difference between “ultra-processed” and just “processed”:
    • Your average hotdog has been ultra-processed. It’s not only usually been changed with many artificial additives, it’s also been through a series of processes (physical and chemical) and ends up bearing little relation to the creature it came from.
    • Your bacon (that you bought fresh from your local butcher, not a supermarket brand of unknown provenance, and definitely not the kind that might come on the top of frozen supermarket pizza) has been processed. It’s undergone a couple of simple processes on its journey “from farm to table”. Remember also that when you cook it, that too is one more process (and one that results in a lot of AGEs).

    Read more: What’s so bad about AGEs?

    Note if you really don’t want to cut out certain foods, changing the way you cook them (i.e., the last process your food undergoes before you eat it) can also reduce AGES:

    Advanced Glycation End Products in Foods and a Practical Guide to Their Reduction in the Diet

    Get More Fiber

    ❝The American Institute for Cancer Research shows that for every 10-gram increase in fiber in the diet, you improve survival after cancer diagnosis by 13%❞

    Source: Plant-based diet is encouraged for patients with cancer

    Yes, that’s post-diagnosis, but as a general rule of thumb, what is good/bad for cancer when you have it is good/bad for cancer beforehand, too.

    If you’re thinking that increasing your fiber intake means having to add bran to everything, happily there are better ways:

    Level-Up Your Fiber Intake! (Without Difficulty Or Discomfort)

    Enjoy!

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  • Medicinal cannabis is most often prescribed for pain, anxiety and sleep. Here’s what the evidence says

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Medicinal cannabis use has increased rapidly in recent years in Australia. Since access pathways were expanded in 2016, more than 700,000 prescription approvals have been issued.

    The vast majority of medicinal cannabis products on the market have not been registered on the Australian Register of Therapeutic Goods. But medical practitioners can apply to the Therapeutic Goods Administration (TGA) for approval to prescribe them to patients.

    Data shows the three most common conditions for which scripts are approved are chronic pain, anxiety and sleep disorders.

    Although many patients report benefits, professional bodies and regulators have raised concerns about whether prescribing is outpacing the evidence.

    So what does the evidence actually say? Does medicinal cannabis work for the conditions for which it’s most commonly prescribed?

    Vilin Visuals/Getty Images

    Medicinal cannabis for pain

    Medicinal cannabis refers to cannabis products that are legally prescribed to treat a medical condition. This can be the plant itself, or natural compounds extracted from the plant. Some compounds similar to or the same as those found in cannabis (for example, dronabinol and nabilone) are made in a lab.

    Two of the most common compounds in the plant are THC (tetrahydrocannabinol) and CBD (cannabidiol), known as cannabinoids.

    These are commonly found at various concentrations in medicinal cannabis products which come in forms including oils, capsules, dried flower (used in a vaporiser), sprays and gummies.

    Chronic pain is the most common reason for medicinal cannabis use. But as we’ve written in a previous article, research shows only modest benefits, with limited improvements in pain and physical functioning.

    The TGA says there’s limited evidence medicinal cannabis provides clinically significant pain relief for many conditions, and should only be tried if other standard therapies haven’t helped.

    Does medicinal cannabis work for anxiety?

    Beside chronic pain, a growing number of people are now turning to medicinal cannabis for anxiety.

    Multiple reviews have examined whether it works for this purpose and have come to similar conclusions. For THC-based products the evidence is mixed, with some patients finding relief, while others report their symptoms are worse.

    There is emerging evidence for CBD, however it’s too soon to recommend medical cannabis as a first-line treatment for anxiety. So far, studies of CBD in anxiety have been small, only measured effects under experimental conditions designed to induce stress, had no comparison group, or only tested a one-off dose. Because of these limitations, the studies can’t tell us if CBD is effective for ongoing anxiety management.

    A recent review found CBD had positive effects on anxiety, but these effects were seen in studies deemed to have problems with their methods, and not in studies that were more rigorously designed and conducted.

    Similarly, a small Australian study (with no control group) demonstrated positive effects of CBD in young people with anxiety who had already tried other treatments. However, the authors stated more rigorous trials were still needed.

    What’s more, there are recent case reports of acute psychosis arising from medicinal cannabis use. Taken together with the ambiguous evidence, the role for cannabinoids for anxiety remains far from clear.

    How about sleep disorders?

    The evidence for cannabis in the treatment of sleep disorders and insomnia is perhaps even more limited, with neither CBD or THC having shown clear benefits reducing the number of awakenings or time spent awake during the night, or improved sleep quality. That said, some people do report they have fewer symptoms of insomnia when using medicinal cannabis.

    Similar to anxiety, many of the studies have major weaknesses in their study design which make it difficult to draw strong conclusions. There are also few studies that compare medicinal cannabis to proven treatments for sleep disorders and insomnia. This makes it hard to make recommendations for treatment based on the current research evidence.

    THC can make you drowsy, and in the short term, may help people fall asleep, or feel like they’re getting more sleep. But there are some important downsides to consider, too.

    For example, if you take medicinal cannabis regularly to fall asleep your body can get used to it, making it harder to fall asleep without it. In the long term, medicinal cannabis can also affect the amounts of light and deep sleep a person will have, which can result in poorer sleep quality.

    There is good evidence for some conditions

    Some of the strongest evidence for medicinal cannabis products are for rare forms of epilepsy that don’t respond to existing treatments, and for treating symptoms associated with multiple sclerosis.

    The only TGA-approved medicinal cannabis products are for these conditions.

    There’s also evidence medicinal cannabis can help with chemotherapy-induced nausea and vomiting. Though as newer medications with fewer side effects are now available, medicinal cannabis products are not considered first-line treatments.

    Risks and side effects

    Common side effects with THC in the short term include drowsiness, anxiety, dry mouth, nausea, vomiting and appetite changes. For some people, these effects reduce over time.

    Some people with preexisting health conditions such as schizophrenia, psychosis or heart conditions may be more prone to experiencing side effects.

    An estimated one in four people using medical cannabis meet the criteria for dependence (known as cannabis use disorder). In the longer term, dependence appears more common with medical use, particularly when combined with non-medical use.

    If you are suffering with anxiety, sleep problems or chronic pain, and are wondering what treatments might be most effective for you, speak to your regular GP.

    Suzanne Nielsen, Professor and Deputy Director, Monash Addiction Research Centre, Monash University and Myfanwy Graham, NHMRC Postgraduate Scholar and Fulbright Alumna in Public Health Policy, Monash University

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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  • Why eating disorder recovery is about more than what you eat or weigh

    10almonds is reader-supported. We may, at no cost to you, receive a portion of sales if you purchase a product through a link in this article.

    Recovering from an eating disorder can be long and complex.

    Treatment typically focuses on reducing the unhelpful behaviours and thoughts that characterise these disorders. These include extreme dieting, binge eating, purging, negative body image, and – in some (but not all) cases – having a very low body weight.

    But when recovery focuses on a clinical checklist of symptoms, such as reaching a healthy weight, it may ignore other important aspects of getting better.

    Eating disorders are not just physical. They are complex mental health conditions that severely disrupt people’s relationship with themselves, their bodies and other people. So the psychological aspects of recovery, and the way people feel about it, also plays an important role.

    Our new research shows when people’s broader wellbeing improves – such as developing a sense of self-acceptance or hope – they are more likely to report a “personal” recovery from an eating disorder, even if they still have some clinical symptoms.

    A.C./Unsplash

    How is recovery measured?

    There is no one definition of eating disorder recovery.

    But most research has focused on clinical symptoms. This means an absence of diagnostic criteria (for example, no binge eating or purging) over a specific timeframe, such as a 12-month period, to meet the definition of recovery.

    Emerging research points to the importance of “personal recovery” meaning that dimensions of psychological wellbeing are essential.

    For example, a 2020 review of studies focusing on perspectives of people with eating disorders showed supportive relationships, hope, identity, meaning and purpose, empowerment, and self-compassion were central to their recovery process.

    People with eating disorders also report that including these as goals (rather than just focusing on clinical symptoms) feels relevant and empowering, while emerging research shows this can improve long-term outcomes and improve quality of life, meaning people may be less likely to relapse.

    But there still hasn’t been much research on how both personal and clinical aspects can be incorporated into treatment and recovery.

    Understanding how to include these aspects in treatment is urgent, given eating disorders are among the most life-threatening psychiatric disorders, and recovery is often slow.

    What we did and what we found

    Our new study surveyed 234 adults who have lived through or are currently experiencing an eating disorder. Most identified as female (89%), and the average age was 28.

    Overall, we found less than a quarter of participants (22.6%) met the criteria for clinical improvement, meaning many were still dieting or preoccupied with food and body image.

    But more than half (52.1%) felt they had achieved personal recovery. This included experiencing self-acceptance, positive relationships, personal growth, reduced eating disorder behaviours, resilience and greater autonomy.

    Clinical improvement in symptoms did make personal recovery more likely. But nearly two-thirds (63.9%) of those who self-identified as personally recovered did not meet the clinical definition, meaning they still experienced some eating disorder symptoms.

    This points to a possible disconnect between definitions of recovery that focus on symptoms and what recovery actually means to the people living it.

    We also explored whether personal recovery looked different depending on someone’s eating disorder diagnosis.

    All participants had a past or current diagnosis of anorexia nervosa (68.4%), bulimia nervosa (8.5%) or binge eating disorder (8.1%).

    But, we found no meaningful differences in personal recovery rates across these diagnoses. This suggests the experience of personal recovery may be broadly similar regardless of the specific eating disorder a person has faced.

    Why does this matter?

    When treatment success is measured almost entirely through symptom checklists and clinical criteria, we risk missing – and failing to celebrate – the progress that may matter the most to the person in front of us.

    We suggest people seeking recovery from an eating disorder should be asked early on about what recovery looks like to them, not just what the clinical guidelines say it should look like. This might also improve the currently low rates of people seeking help for eating disorders. It may help clinicians set goals that are meaningful and better reflect the psychological nature of eating disorders, not just the physical aspects.

    If there’s something that feels important to your recovery, it’s worth raising with your treatment team. Recovery can look different for everyone, and your personal goals matter.

    For example, wellbeing goals could involve reconnecting with relationships, rebuilding a sense of identity, or simply feeling more in control of daily life, alongside improving clinical symptoms.

    This is also significant because funding for eating disorder services and policy decisions still often lean heavily on clinical benchmarks. If these don’t capture aspects of personal recovery, we are likely underestimating how many people are getting better, and potentially designing services around a narrower picture of recovery than the evidence actually supports.


    If you have a history of an eating disorder or suspect you may have one, you can contact the Butterfly Foundation’s national helpline on 1800 334 673, or via online chat.

    Catherine Houlihan, Senior Lecturer in Clinical Psychology, University of the Sunshine Coast; Andrew Allen, Senior Lecturer in Clinical Psychology, University of the Sunshine Coast; Dan Fassnacht, Associate Professor in Psychology, University of the Sunshine Coast, and Kathina Ali, Senior Lecturer in Clinical Psychology, University of the Sunshine Coast

    This article is republished from The Conversation under a Creative Commons license. Read the original article.

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