Prolonged Grief: A New Mental Disorder?
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The issue is not whether certain mental conditions are real—they are. It is how we conceptualize them and what we think treating them requires.
The latest edition of the Diagnostic and Statistical Manual of Mental Disorders (DSM-5) features a new diagnosis: prolonged grief disorder—used for those who, a year after a loss, still remain incapacitated by it. This addition follows more than a decade of debate. Supporters argued that the addition enables clinicians to provide much-needed help to those afflicted by what one might simply consider a too much of grief, whereas opponents insisted that one mustn’t unduly pathologize grief and reject an increasingly medicalized approach to a condition that they considered part of a normal process of dealing with loss—a process which in some simply takes longer than in others.
By including a condition in a professional classification system, we collectively recognize it as real. Recognizing hitherto unnamed conditions can help remove certain kinds of disadvantages. Miranda Fricker emphasizes this in her discussion of what she dubs hermeneutic injustice: a specific sort of epistemic injustice that affects persons in their capacity as knowers1. Creating terms like ‘post-natal depression’ and ‘sexual harassment’, Fricker argues, filled lacunae in the collectively available hermeneutic resources that existed where names for distinctive kinds of social experience should have been. The absence of such resources, Fricker holds, put those who suffered from such experiences at an epistemic disadvantage: they lacked the words to talk about them, understand them, and articulate how they were wronged. Simultaneously, such absences prevented wrong-doers from properly understanding and facing the harm they were inflicting—e.g. those who would ridicule or scold mothers of newborns for not being happier or those who would either actively engage in sexual harassment or (knowingly or not) support the societal structures that helped make it seem as if it was something women just had to put up with.
For Fricker, the hermeneutical disadvantage faced by those who suffer from an as-of-yet ill-understood and largely undiagnosed medical condition is not an epistemic injustice. Those so disadvantaged are not excluded from full participation in hermeneutic practices, or at least not through mechanisms of social coercion that arise due to some structural identity prejudice. They are not, in other words, hermeneutically marginalized, which for Fricker, is an essential characteristic of epistemic injustice. Instead, their situation is simply one of “circumstantial epistemic bad luck”2. Still, Fricker, too, can agree that providing labels for ill-understood conditions is valuable. Naming a condition helps raise awareness of it, makes it discursively available and, thus, a possible object of knowledge and understanding. This, in turn, can enable those afflicted by it to understand their experience and give those who care about them another way of nudging them into seeking help.
Surely, if adding prolonged grief disorder to the DSM-5 were merely a matter of recognizing the condition and of facilitating assistance, nobody should have any qualms with it. However, the addition also turns intense grief into a mental disorder—something for whose treatment insurance companies can be billed. With this, significant forces of interest enter the scene. The DSM-5, recall, is mainly consulted by psychiatrists. In contrast to talk-therapists like psychotherapists or psychoanalysts, psychiatrists constitute a highly medicalized profession, in which symptoms—clustered together as syndromes or disorders—are frequently taken to require drugs to treat them. Adding prolonged grief disorder thus heralds the advent of research into various drug-based grief therapies. Ellen Barry of the New York Times confirms this: “naltrexone, a drug used to help treat addiction,” she reports, “is currently in clinical trials as a form of grief therapy”, and we are likely to see a “competition for approval of medicines by the Food and Drug Administration.”3
Adding diagnoses to the DSM-5 creates financial incentives for players in the pharmaceutical industry to develop drugs advertised as providing relief to those so diagnosed. Surely, for various conditions, providing drug-induced relief from severe symptoms is useful, even necessary to enable patients to return to normal levels of functioning. But while drugs may help suppress feelings associated with intense grief, they cannot remove the grief. If all mental illnesses were brain diseases, they might be removed by adhering to some drug regimen or other. Note, however, that ‘mental illness’ is a metaphor that carries the implicit suggestion that just like physical illnesses, mental afflictions, too, are curable by providing the right kind of physical treatment. Unsurprisingly, this metaphor is embraced by those who stand to massively benefit from what profits they may reap from selling a plethora of drugs to those diagnosed with any of what seems like an ever-increasing number of mental disorders. But metaphors have limits. Lou Marinoff, a proponent of philosophical counselling, puts the point aptly:
Those who are dysfunctional by reason of physical illness entirely beyond their control—such as manic-depressives—are helped by medication. For handling that kind of problem, make your first stop a psychiatrist’s office. But if your problem is about identity or values or ethics, your worst bet is to let someone reify a mental illness and write a prescription. There is no pill that will make you find yourself, achieve your goals, or do the right thing.
Much more could be said about the differences between psychotherapy, psychiatry, and the newcomer in the field: philosophical counselling. Interested readers may benefit from consulting Marinoff’s work. Written in a provocative, sometimes alarmist style, it is both entertaining and—if taken with a substantial grain of salt—frequently insightful. My own view is this: from Fricker’s work, we can extract reasons to side with the proponents of adding prolonged grief disorder to the DSM-5. Creating hermeneutic resources that allow us to help raise awareness, promote understanding, and facilitate assistance is commendable. If the addition achieves that, we should welcome it. And yet, one may indeed worry that practitioners are too eager to move from the recognition of a mental condition to the implementation of therapeutic interventions that are based on the assumption that such afflictions must be understood on the model of physical disease. The issue is not whether certain mental conditions are real—they are. It is how we conceptualize them and what we think treating them requires.
No doubt, grief manifests physically. It is, however, not primarily a physical condition—let alone a brain disease. Grief is a distinctive mental condition. Apart from bouts of sadness, its symptoms typically include the loss of orientation or a sense of meaning. To overcome grief, we must come to terms with who we are or can be without the loved one’s physical presence in our life. We may need to reinvent ourselves, figure out how to be better again and whence to derive a new purpose. What is at stake is our sense of identity, our self-worth, and, ultimately, our happiness. Thinking that such issues are best addressed by popping pills puts us on a dangerous path, leading perhaps towards the kind of dystopian society Aldous Huxley imagined in his 1932 novel Brave New World. It does little to help us understand, let alone address, the moral and broader philosophical issues that trouble the bereaved and that lie at the root not just of prolonged grief but, arguably, of many so-called mental illnesses.
Footnotes:
1 For this and the following, cf. Fricker 2007, chapter 7.
2 Fricker 2007: 152
3 Barry 2022
References:
Barry, E. (2022). “How Long Should It Take to Grieve? Psychiatry Has Come Up With an Answer.” The New York Times, 03/18/2022, URL = https://www.nytimes.com/2022/03/18/health/prolonged-grief-
disorder.html [last access: 04/05/2022])
Fricker, M. (2007). Epistemic Injustice. Power & the Ethics of knowing. Oxford/New York: Oxford University Press.
Huxley, A. (1932). Brave New World. New York: Harper Brothers.
Marinoff, L. (1999). Plato, not Prozac! New York: HarperCollins Publishers.
Professor Raja Rosenhagen is currently serving as Assistant Professor of Philosophy, Head of Department, and Associate Dean of Academic Affairs at Ashoka University. He earned his PhD in Philosophy from the University of Pittsburgh and has a broad range of philosophical interests (see here). He wrote this article a) because he was invited to do so and b) because he is currently nurturing a growing interest in philosophical counselling.
This article is republished from OpenAxis under a Creative Commons license. Read the original article.
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How Much Does A Vegan Diet Affect Biological Aging?
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Slow Your Aging, One Meal At A Time
This one’s a straightforward one today, and the ““life hack” can be summed up:
Enjoy a vegan diet to enjoy younger biological age.
First, what is biological age?
Biological age is not one number, but a collection of numbers, as per different biomarkers of aging, including:
- Visual markers of aging (e.g. wrinkles, graying hair)
- Performative markers of aging (e.g. mobility tests)
- Internal functional markers of aging (e.g. tests for cognitive decline, eyesight, hearing, etc)
- Cellular markers of aging (e.g. telomere length)
We wrote more about this here:
Age & Aging: What Can (And Can’t) We Do About It?
A vegan diet may well impact multiple of those categories of aging, but today we’re highlighting a study (hot off the press; published only a few days ago!) that looks at its effect on that last category: cellular markers of aging.
There’s an interesting paradox here, because this category is:
- the most easily ignorable; because we all feel it if our knees are giving out or our skin is losing elasticity, but who notices if telomeres’ T/S ratio changed by 0.0407? ← the researchers, that’s who, as this difference is very significant
- the most far-reaching in its impact, because cellular aging in turn has an effect on all the other markers of aging
Second, how much difference does it make, and how do we know?
The study was an eight-week interventional identical twin study. This means several things, to start with:
- Eight weeks is a rather short period of time to accumulate cellular aging, let alone for an intervention to accumulate a significant difference in cellular aging—but it did. So, just imagine what difference it might make in a year or ten!
- Doing an interventional study with identical twin pairs already controlled for a lot of factors, that are usually confounding variables in population / cohort / longitudinal / observational studies.
Factors that weren’t controlled for by default by using identical twins, were controlled for in the experiment design. For example, twin pairs were rejected if one or more twin in a given pair already had medical conditions that could affect the outcome:
❝Inclusion criteria involved participants aged ≥18, part of a willing twin pair, with BMI <40, and LDL-C <190 mg/dL. Exclusions included uncontrolled hypertension, metabolic disease, diabetes, cancer, heart/renal/liver disease, pregnancy, lactation, and medication use affecting body weight or energy.
Eligibility was determined via online screening, followed by an orientation meeting and in-person clinic visit. Randomization occurred only after completing baseline visits, dietary recalls, and questionnaires for both twins❞
~ Dr. Varun Dwaraka et al. ← there’s a lot of “et al.” to this one; the paper had 16 collaborating authors!
As to the difference it made over the course of the 8 weeks…
❝Various measures of epigenetic age acceleration (PC GrimAge, PC PhenoAge, DunedinPACE) were assessed, along with system-specific effects (Inflammation, Heart, Hormone, Liver, and Metabolic).
Distinct responses were observed, with the vegan cohort exhibiting significant decreases in overall epigenetic age acceleration, aligning with anti-aging effects of plant-based diets. Diet-specific shifts were noted in the analysis of methylation surrogates, demonstrating the influence of diet on complex trait prediction through DNA methylation markers.❞
~ Ibid.
You can read the whole paper here (it goes into a lot more detail than we have room to here, and also gives infographics, charts, numbers, the works):
Were they just eating more healthily, though?
Well, arguably yes, as the results show, but to be clear:
The omnivorous diet compared to the vegan diet in this study was also controlled; both groups were given a healthy meal plan for their respective diet. So this wasn’t a case of “any omnivorous diet vs healthy vegan diet”, but rather “healthy omnivorous diet vs healthy vegan diet”.
Again, the paper itself has the full details—a short version is that it involved a healthy meal kit delivery service, followed by ongoing dietician involvement in an equal and carefully-controlled fashion.
So, aside from that one group had an omnivorous meal plan and the other vegan, both groups received the same level of “healthy eating” support, guidance, and oversight.
But isn’t [insert your preferred animal product here] healthy?
Quite possibly! For general health, general scientific consensus is that eating at least mostly plants is best, red meat is bad, poultry is neutral in moderation, fish is good in moderation, dairy is good in moderation if fermented, eggs are good in moderation if not fried.
This study looked at the various biomarkers of aging that we listed, and not every possible aspect of health—there’s more science yet to be done, and the researchers themselves are calling for it.
It also bears mentioning that for some (relatively few, but not insignificantly few) people, extant health conditions may make a vegan diet unhealthy or otherwise untenable. Do speak with your own doctor and/or dietician if unsure.
See also: Do We Need Animal Products To Be Healthy?
We would hypothesize, by the way, that the anti-aging benefits of a vegan diet are probably proportional to abstention from animal products—meaning that even if you simply have some “vegan days”, while still consuming animal products other days, you’ll still get benefit for the days you abstained. That’s just our hypothesis though.
Take care!
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Tasty Tofu Scramble
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If you’re trying to eat more plant-based, this is a great way to enjoy a culinary experience that hits the same notes as scrambled egg, with many similar nutritional benefits too, and some of its own!
You will need
- 1 cup (10oz) silken tofu
- ¼ bulb garlic, crushed
- 1 tbsp nutritional yeast
- 2 tsp chia seeds
- 2 tsp dried thyme, or 1 tsp fresh thyme, stripped (i.e. pulled off the stalks)
- 2 tsp turmeric
- 2 tsp black pepper, coarse ground
- 1 tsp red chili flakes
- ½ tsp MSG, or 1 tsp low-sodium salt
- Extra virgin olive oil, for frying
Method
(we suggest you read everything at least once before doing anything)
1) Heat a skillet with olive oil in it; if you want a low-calorie option, you can use quite little oil here; the tofu is a lot more forgiving than egg in this regard and is almost impossible to burn unless you actively try. If you don’t want a low-calorie option, feel free to be generous with the oil if you prefer; it’ll go into the tofu and make it fattier, much like egg.
2) Add the tofu. You can just drop it (carefully) straight in; you don’t need to press it or anything.
3) Scramble it with a spatula, just the same as you would if it were egg.
4) Add the rest of the ingredients, mixing them in as you continue to scramble it, until it reaches the desired consistency.
5) Serve! Serving it on wholegrain toast is a great option—but this dish can also be enjoyed any other way you might use scrambled eggs (including for making
egg-friedtofu-fried rice; just stir it into our Tasty Versatile Rice recipe!)
Enjoy!
Want to learn more?
For those interested in some of the science of what we have going on today:
- Plant vs Animal Protein: Head to Head
- Black Pepper’s Impressive Anti-Cancer Arsenal
- Capsaicin For Weight Loss And Against Inflammation
- The Many Health Benefits Of Garlic
- Why Curcumin (Turmeric) Is Worth Its Weight In Gold
- If You’re Not Taking Chia, You’re Missing Out
Take care!
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Makkō-Hō – by Haruka Nagai
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We’ve all heard the claims, “Fluent in 3 Months!”, “Russian in Two Weeks!”, “Overnight Mandarin Chinese”, “15-Minute Arabic!”, “Instant Italian!”.
We see the same in the world of health and fitness too. So how does this one’s claim of “five minutes’ physical fitness” hold up?
Well, it is 5 minutes per day. And indeed, the author writes:
❝The total time [to do these exercises], then, is only one minute and thirty seconds. This series I call one round. When it has been completed, execute another complete round. You should find the exercises easier to do the second time. Executed this way, the exercsies will prove very effective, though they take only three minutes in all. After you have leaned back into the final position, you must remain in that posture for one minute. That brings the total time to four minutes. Even when [some small additions] are added, it takes only five minutes at most.❞
The exercises themselves are from makkō-hō, which is a kind of Japanese dynamic yoga. They involve repetitions of (mostly) moving stretches with good form, and are excellent for mobility and general health, keeping us supple and robust as we get older.
The text descriptions are clear, as are the diagrams and photos. The language is a little dated, as this book was written in the 1970s, but the techniques themselves are timeless.
Bottom line: consider it a 5-minute anti-aging regimen. And, as Nagai says, “the person who cannot find 5 minutes out of 24 hours, was never truly interested in their health”.
Click here to check out Makkō-Hō and schedule your five minutes!
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A Peek Behind The 10almonds Curtain
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At 10almonds we give a lot of health information, so you may wonder: how much do we (the 10almonds team) put into practice? Is it even possible to do all these things? Do we have an 80:20 rule going on?
So, here’s what someone who thinks, reads, and writes about health all day, does for her health—and how it ties in with what you read here at 10almonds.
Hi, it’s me, a member of the 10almonds team and regular writer here, and I’m going to do the rest of this article in the first-person, since it’s using me as an example!
(PS: yes, the thumbnail is a digital impression of my appearance, though I would correct it that my hair is much longer, and my eyes are more gray; I must admit though it captured my smile, not to mention my collarbones-that-you-could-sit-on)
Dietary habits
Before we get to foods, let’s talk intermittent fasting. I practise 16:8 intermittent fasting… Approximately. That is to say, I’m mostly not religious about it, but I will generally breakfast around 12 noon, and have finished eating dinner before 8pm, with no food outside of those hours.
See also: Intermittent Fasting: What’s the truth?
Importantly, while I feel free to be a little flexible around start and finish times, I do very consciously decide “I am now fasting” and “I will now break the fast”.
Note my imperfection: it would be ideal to have the eating period earlier in the day, and have a bigger breakfast and small dinner. However, that doesn’t really work for me (leisurely evening meal is an important daily event in this household), so this is how I do it instead.
Foods!
It gets an exclamation mark because a thing about me is that I do love foods.
Breakfast: a typical breakfast for me these past months is a couple of ounces of mixed nuts with about 1oz of goji berries.
The mixed nuts are in equal proportions: almonds, walnuts, hazelnuts, cashews. Why those four? Simple, it’s because that’s what Aldi sells as “mixed nuts” and they are the cheapest nuts around, as well as containing absolute nutritional heavyweights almonds and walnuts in generous portions.
Often, but not always, I’ll have some dates with it, or dried apricots, or prunes.
I go through phases; sometimes I’ll enjoy overnight oats as my breakfast for a month or two at a time. I really just follow my gut in this regard.
See for example: Spiced Pear & Pecan Polyphenol Porridge
Lunch: I don’t really lunch per se, but between breakfast and dinner I’ll usually snack on a 3–5 organic carrots (I literally just cut the tops off and otherwise eat them like Bugs Bunny—peeling them would be extra work just to lose fiber). Note my imperfection: I don’t buy all of my vegetables organic, but I do for the ones where it makes the biggest difference.
If I’m feeling like it, I may lunch on a selection of herbs sabzi khordan style (see: Invigorating Sabzi Khordan), though I’m vegan so for simplicity I just skip the cheeses that are also traditional with that dish. On the other hand, for protein and fat I’ll usually add a cup of beans (usually black beans or kidney beans), seasoned with garlic and black pepper in an olive oil and balsamic vinegar dressing (that I make myself, so it’s just those ingredients). See also: Kidney Beans vs Black Beans – Which Is Healthier?
Dinner: this is my main sit-down meal of the day, and it’s enjoyed in a leisurely fashion (say, 40 minutes average with a normal distribution bell curve running between 20 and 60 minutes) with my son who lives with me. I mention all of this, because of the importance of relaxed mindful eating. In the instances of it being nearer the 20 minutes end, it’s not because of rushing, but rather because of a lighter meal some days.
See also: How To Get More Nutrition From The Same Food
Regular recurrences in the menu include:
I’ll often snack on something probiotic (e.g. kimchi) while I’m cooking.
See also: Make Friends With Your Gut! (You Can Thank Us Later)
In terms of what’s not in my diet: as mentioned, I’m vegan, so animal products are out. I don’t drink alcohol or use other recreational drugs, and I mostly drink decaffeinated coffee, but I’ll have a caffeinated one if I’m out somewhere. I’m not a puritan when it comes to sugar, but also, I simply don’t like it and I know well its health effects, so it doesn’t really form part of my diet except insofar as it’s in some ready-made condiments I may sometimes use (e.g. sriracha, teriyaki sauce). I’m also not a puritan when it comes to wheat, but it’s not something I consume daily. Usually on a weekly basis I’ll have a wholegrain pasta dish, and a dish with some kind of wholegrain flatbread.
See also:
Exercise!
First, some things that are lifestyle factors:
- I do not own a car, and I dislike riding in cars, buses, etc. So, I walk everywhere, unless it’s far enough that I must take the train, and even then I usually stand between carriages rather than sitting down.
- I have a standing desk setup, that hasn’t been lowered even once since I got it. I highly recommend it, as someone who spends a lot of time at my desk.
- You may imagine that I spend a lot of time reading; if it’s books (as opposed to scientific papers etc, which I read at my desk), then I’ll most of the time read them while perched like a gargoyle in a sitting squat (Slav squat, Asian squat, resting squat, deep squat, etc) on a balance ball. Yes, it is comfortable once you’re used to it!
About that latter, see also: The Most Anti Aging Exercise
In terms of “actual” exercise, I get 150–300 minutes “moderate exercise” per week, which is mostly composed of:
- Most days I walk into town to get groceries; it’s a 40-minute round trip on foot
- On days I don’t do that, even if I do walk to a more local shop, I spend at least 20 minutes on my treadmill.
See also: The Doctor Who Wants Us To Exercise Less, & Move More
Strength and mobility training, for me, comes mostly in what has been called “exercise snacking”, that is to say, I intersperse my working day with brief breaks to do Pilates exercises. I have theme days (lower body, core, upper body) and on average one rest day (from Pilates exercises) per week, though honestly, that’s usually more likely because of time constraints than anything else, because a deadline is looming.
See also: Four Habits That Drastically Improve Mobility
You may be wondering about HIIT: when I’m feeling extra-serious about it, I use my exercise bike for this, but I’ll be honest, I don’t love the bike, so on a daily basis, I’m much more likely to do HIIT by blasting out a hundred or so Hindu squats, resting, and repeating.
See also: How To Do HIIT (Without Wrecking Your Body)
Supplements
First I’ll mention, I do have HRT, of which the hormones I have are bioidentical estradiol gel in the morning, and a progesterone pessary in the evening. They may not be for everybody, but they’ve made a world of difference to me.
See also: HRT: A Tale Of Two Approaches
In terms of what one usually means when one says supplements, many I use intermittently (which is good in some cases, as otherwise the body may stop using them so well, or other problems can arise), but regular features include:
- Magnesium glycinate, malate, & citrate (See: Which Magnesium? (And: When?))
- Active vitamers of vitamins B9 and B12 (See: Which B Vitamins? It Makes A Difference!)
- Liposomal vitamin C (I actually get enough for general purposes in my diet, but as a vegan I don’t get dietary collagen, so this helps collagen synthesis)
- L-theanine (bedtime only) (See: L-Theanine: What’s The Tea?)
- Lion’s mane mushroom (See: What does lion’s mane mushroom actually do, anyway?)
- Quercetin (See: Fight Inflammation & Protect Your Brain, With Quercetin)
- Fisetin (weekends only) (See: Fisetin: The Anti-Aging Assassin)
Why weekends only for Fisetin? See: The Drug & Supplement Combo That Reverses Aging ← the supplement is fisetin, which outperforms quercetin in this role, and/but it only needs be taken for two days every two weeks, as a sort of “clearing out” of senescent cells. There is no need to take it every day, because if you just cleared out your senescent cells, then guess what, they’re not there now. Also, while sensescent cells are a major cause of aging, on a lower level they do have some anti-tumor effects, so it can be good to let some live a least for a while now and again. In short, cellular sensescence can help prevent tumors on a daily level, but it doesn’t hurt that capacity to have a clearing-out every couple of weeks; so says the science (linked above). Note my imperfection: I take it at weekends instead of for two days every two weeks (as is standard in studies, like those linked above), because it is simpler than remembering to count the weeks.
Cognitive exercise
Lest we forget, exercising our mind is also important! In my case:
- I’m blessed to have work that’s quite cognitively stimulating; our topics here at 10almonds are interesting. If it weren’t for that, I’d still be reading and writing a lot.
- I play chess, though these days I don’t play competitively anymore, and play rather for the social aspect, but this too is important in avoiding cognitive decline.
- I am one of those people who compulsively learns languages, and uses them a lot. This is very beneficial, as language ability is maintained in a few small areas of the brain, and it’s very much “use it or lose it”. Now, while I may not need my French or Russian or Arabic to keep the lights on in this part of the brain or that, the fact that I am pushing my limits every day is the important part. It’s not about how much I know—it’s about how much I engage those parts of my brain on a daily basis. Thus, even if you speak only one language right now, learning even just one more, and learning even only a little bit, you will gain the brain benefits—because you’re engaging it regularly in a new way, and that forces the brain to wire new synapses and also to maintain volume in those parts.
See also: How To Reduce Your Alzheimer’s Risk
And about language-learning specifically: An Underrated Tool Against Alzheimer’s ←this also shows how you don’t have to be extreme about language learning like I admittedly am.
How’s all this working out for me?
I can say: it works! My general health is better now than it was decades ago. I’ve personally focused a lot on reducing inflammation, and that really pays dividends when it comes to the rest of health. I didn’t talk about it above, but focusing on my sleep regularity and quality has helped a lot too.
In terms of measurable results, I recently had a general wellness checkup done by means of a comprehensive panel of 14 blood tests, and various physical metrics (BMI, body fat %, blood pressure, etc), and per those, I could not be in better health; it was as though I had cheated and written in all the best answers. I say this not to brag (you don’t know me, after all), but rather to say: it can be done!
Even without extreme resources, and without an abundance of free time, etc, it can be done!
Caveat: if you have some currently incurable chronic disease, there may be some limits. For example, if you have Type 1 Diabetes, probably your HbA1c* is going to be a little off even if you do everything right.
*HbA1c = glycated hemoglobin, a very accurate measure of what your blood glucose has been on average for the past 2–3 months—why 2–3 months? Because that’s the approximate lifespan of a red blood cell, and we’re measuring how much hemoglobin (in the red blood cells) has been glycated (because of blood glucose).
In summary
The stuff we write about at 10almonds can be implemented, on a modest budget and while juggling responsibilities (work, family, classes, etc).
I’m not saying that my lifestyle should be everyone’s template, but it’s at least an example of one that can work.
- Maybe you hate walking and love swimming.
- Maybe you have no wish to give up fish and eggs, say (both of which are fine/good in moderation healthwise).
- Maybe you have different priorities with supplements.
- Maybe you find language-learning uninteresting but take singing lessons.
- And so on.
In the absolute fewest words, the real template is:
- Decide your health priorities (what matters most for you)
- Look them up on 10almonds
- Put the things into action in a way that works for you!
Take care!
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How To Stay A Step Ahead Of Peripheral Artery Disease
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Far less well-known than Coronary Artery Disease, it can still result in loss of life and limb (not in that order). Fortunately, there are ways to be on your guard:
What it is
Peripheral Artery Disease (PAD) is the same thing as Coronary Artery Disease (CAD), just, in the periphery—which by definition means “outside of the heart and brain”, but in practice, it starts with the extremities. And of the extremities, it tends to start with the feet and legs, for the simple reason that if someone’s circulation is sluggish, then because of gravity, that’s where’s going to get blocked first.
In both CAD and PAD, the usual root cause is atherosclerosis, that is to say, the build-up of fatty material inside the arteries, usually commensurate to LDL (“bad”) cholesterol, especially in men (high LDL is still a predictor of cardiovascular disease in women though, just more modestly so, at least pre-menopause or in cases of treated menopause whereby HRT has returned hormones to pre-menopause levels).
See also: Demystifying Cholesterol
And for that about sex differences: His & Hers: The Hidden Complexities of Statins and Cardiovascular Disease (CVD)
Why it is
This one’s straightforward, as it’s the same things as any kind of cardiovascular disease: high blood pressure, high cholesterol, older age, obesity, smoking, drinking, diabetes, and genetic factors (so, a risk factor is: family history of heart disease).
However, while those are the main causes and/or risk factors, it absolutely can still strike other people, so it’s as well to be watch out for…
What to look out for
Many people first notice signs and symptoms that turn out to be PAD when they experience pain or numbness in the foot or feet, and/or a discoloration of the feet (especially toes), and slow wound healing.
At that stage, chances are you will need to go urgently to a specialist, and surgery is a likely necessity. With a little luck, it’ll be a minimally-invasive surgery to unblock an artery; failing that, an amputation will be in order.
At that stage, under 50% will be alive 5 years from diagnosis:
You probably want to avoid those. Good news is, you can, by catching it earlier!
What to look out for before that
The most common test for PAD is one you can do at home, but enlisting a nurse to do it for you will help ensure accurate readings. It’s called the Ankle-Brachial Index (ABI) test, and it involves comparing the blood pressure in your ankle with the blood pressure in your arm, and expressing them as a ratio.
Here’s how to do it (instructions and a video demonstration if you want it):
Do Try This At Home: ABI Test For Clogged Arteries
If you need a blood pressure monitor, by the way, here’s an example product on Amazon.
- A healthy ABI score is between 1.0 and 1.4; anything outside this range may indicate arterial problems.
- Low ABI scores (below 0.8) suggest plaque is likely obstructing blood flow
- High ABI scores (above 1.4) may indicate artery hardening
Do note also that yes, if you have plaque obstructing blood flow and hardened arteries, your scores may cancel out and give you a “healthy” score, despite your arteries being very much not healthy.
For this reason, this test can be used to raise the alarm, but not to give the “all clear”.
There are other tests that clinicians can do for you, but you can’t do at home unless you have an MRI machine, a CT scanner, an x-ray machine, a doppler-and-ultrasound machine, etc. We’ll not go into those in detail here, but ask your doctor about them if you’re concerned.
What to do about it
In the mid-to-late stages of the disease, the options are medication and surgery, respectively, but your doctor will advise about those in that eventuality.
In the early stages of the disease, the first-line recommend treatment is exercise, of which, especially walking:
Lower Extremity Peripheral Artery Disease: Diagnosis and Treatment
Given that this more often happens when someone hasn’t been walking so much, it can be a walk-rest-walk approach at first (a treadmill on a low setting can be very useful for this):
See also: Exercise Comparison Head-to-Head: Treadmill vs Road
Take care!
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Rehab Science – by Dr. Tom Walters
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Many books of this kind deal with the injury but not the pain; some source talk about pain but not the injury; this one does both, and more.
Dr. Walters discusses in detail the nature of pain, various different kinds of pain, the factors that influence pain, and, of course, how to overcome pain.
He also takes us on a tour of various different categories of injury, because some require very different treatment than others, and while there are some catch-all “this is good/bad for healing” advices, sometimes what will help with one injury with hinder healing another. So, this information alone would make the book a worthwhile read already.
After this two-part theory-heavy introduction, the largest part of the book is given over to rehab itself, in a practical fashion.
We learn about how to make an appropriate rehab plan, get the material things we need for it (if indeed we need material things), and specific protocols to follow for various different body parts and injuries.
The style is very much that of a textbook, well-formatted and with plenty of illustrations throughout (color is sometimes relevant, so we recommend a print edition over Kindle for this one).
Bottom line: if you have an injury to heal, or even just believe in being prepared, this book is an excellent guide.
Click here to check out Rehab Science, to overcome pain and heal from injury!
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